reSee.it - Related Video Feed

Out of every hundred cases of myocarditis, only one is likely to result in death due to vaccine side effects. However, it is important to note that myocarditis has a high mortality rate of 50% within five years and a significant number of deaths within ten years. This poses significant risks for individuals who develop myocarditis after vaccination. The question remains whether the vaccine is necessary for an epidemic that some argue is no more severe than the flu.

Myocarditis, or heart damage, is more common than previously thought. Studies in the US military and Thailand show that around 20% of people who receive the COVID vaccine develop myocarditis, as confirmed by echocardiograms and other tests. This means that out of every 1 million vaccinated individuals, 200,000 will experience heart damage. Unfortunately, 50% of those with myocarditis will die within 5 years. This alarming increase in myocarditis cases is due to the cardiotoxic nature of the vaccine. This information comes from Dr. Cressel and Shoemaker in Toronto, Canada.

We have administered this type of vaccine to over one billion people, demonstrating its safety. While there is a very low risk of myocarditis, particularly in young men, the risk of developing myocarditis from COVID-19 is actually higher than from the vaccine.

Myocarditis was found more in young men, with rates highest in those aged 16-17. The condition was less common in women and older age groups. Most cases were mild, but some were severe, impacting a person's life. Myocarditis was most common after the second vaccine dose and less frequent in younger children and with subsequent doses. Natural immunity from previous COVID infection was shown to be effective, even more so than two vaccine doses. Combining previous infection with vaccination provided even better protection. The speaker did not take a booster shot. Translation: Myocarditis was more common in young men, especially those aged 16-17. Most cases were mild, but some were severe. The condition was most frequent after the second vaccine dose. Natural immunity from prior COVID infection was found to be effective, even more so than two vaccine doses. Combining previous infection with vaccination provided even better protection. The speaker did not take a booster shot.

The discussion centers on new evidence regarding myocarditis and pericarditis in the context of mRNA vaccination. It cites earlier 2022 German research showing that heart damage seen in myocarditis cases after vaccination may be a vaccine-triggered autoimmune reaction. The study analyzed endomyocardial biopsy samples and found that the cardiac tissue’s spike protein detection and CD4+ T cell–dominated inflammation suggested an autoimmune mechanism linking the vaccine to heart damage. This was contrasted with an Israel-based population study of hundreds of thousands of unvaccinated individuals that reported no increase in myocarditis or pericarditis incidence, highlighting a discrepancy with the vaccine-triggered autoimmune hypothesis. The center of the new claim is a study published in Circulation by the American Heart Association. The speakers emphasize the credibility of Circulation as a top cardiovascular journal. The study used an experimental mouse model to induce cardiac damage and then examined humans with similar heart damage after vaccination to see if the same mechanism applied. They report that T cells from patients with acute myopericarditis recognize vaccine-encoded spike epitopes that are homologous to cardiac self-proteins. In other words, the immune cells targeting the spike protein may also attack cardiac proteins due to molecular similarity. Further details from the study indicate that, in patients with mild pericarditis after mRNA vaccination (but not in those with COVID-19), there was an expanded pattern of cytokine production similar to that observed in myopericarditis–affected mice and in autoimmune myocarditis. The takeaway provided in plain language is that post-mRNA vaccine myopericarditis is driven by molecular mimicry, causing the immune system to fail to distinguish self from non-self in susceptible patients. The susceptibility is described as being influenced by the widespread distribution of the vaccine, which purportedly leads to heart-homing imprinting and a heart-targeted autoimmune response. The speakers stress that this journal is not fringe and highlight its high impact in cardiovascular medicine. They conclude that the data collectively suggest a mechanism by which the vaccine could provoke cardiac autoimmunity, with implications for clinical communication and understanding of post-vaccination myocarditis.

Myocarditis is a rare side effect of the vaccine, with only 1% of cases resulting in death. However, it is important to note that myocarditis has a high mortality rate of 50% within five years and many deaths within ten years. This poses significant risks for individuals who develop myocarditis after vaccination. The question remains whether the vaccine is necessary for an epidemic that some argue is no more severe than the flu.

We are witnessing a significant increase in cases of myocarditis, with thousands reported in recent studies compared to only a few cases in the past. The potential long-term effects of vaccine-induced myocarditis are concerning, with some cases leading to cardiac arrests years after vaccination. This suggests that the current cases may just be the beginning, and regulatory concerns should extend for at least 5 to 15 years post-vaccination.

The speaker discusses a significant increase in myocarditis cases post-vaccination, with studies showing abnormal cardiac scans in vaccinated individuals. They suggest a potential link between mRNA vaccines and heart inflammation, emphasizing the need for long-term monitoring. Research indicates that mRNA and spike proteins can cause myocarditis, posing a concern for all mRNA products. The heart appears to be a vulnerable target due to various factors.

There is a causal link between vaccination and both myocarditis and pericarditis. The reason for this is still unclear. It may be that the SARS CoV-2 spike protein mimics a protein found on heart muscle cells. If that's the case, when you create an immune response to the SARS CoV-2 spike protein, you could also inadvertently create an immune response to your own heart muscle.

This vaccine has been widely used and is considered safe, with experience in over a billion people. While there is a very low risk of myocarditis, especially in young men, associated with the mRNA technology, the risk of getting myocarditis from COVID-19 itself is higher than the risk from the vaccine.

Before COVID-19, I only encountered two cases of myocarditis in my entire career as a cardiologist. It was a rare condition, usually caused by parvovirus or adenovirus. However, now I see two cases per day in the clinic. We have learned that COVID-19 can cause myocarditis. Various organizations, such as the Israeli and US military, as well as college leagues, conducted screening programs in 2020 and found a few cases, but none were serious or resulted in hospitalizations or deaths. These programs were later discontinued when vaccines were introduced. However, within six months, regulatory agencies confirmed that the COVID-19 vaccines can cause myocarditis, and it can be fatal. It's important for people to understand the risks associated with each vaccine dose they take.

We have experience with this vaccine in a billion people, showing it is safe. The mRNA vaccine carries a very low risk of myocarditis, especially in young men. However, the risk of myocarditis from COVID is higher than from the vaccine.

Out of every hundred cases of myocarditis, only one is likely to result in death due to vaccine side effects. Although the number of myocarditis cases is relatively low, it is crucial to note that myocarditis has a fifty percent mortality rate at five years and a significant number of deaths at ten years. This poses significant risks for individuals who develop myocarditis after vaccination. The ongoing question remains whether the vaccine is necessary for an epidemic that some argue is no more severe than the flu.

In the question, myocarditis is discussed in relation to COVID-19. It has been mentioned for some time that infection with the novel coronavirus can lead to myocarditis, and that in some cases myocarditis can be severe or progress to myocarditis with structural complications. It is noted that myocarditis can also occur after vaccination, but the incidence is small and the symptoms are mild, with most people recovering. The speaker emphasizes that even when myocarditis occurs after vaccination, the risk is small and the condition tends to be mild. The statement asserts that almost all individuals recover from vaccine-associated myocarditis. Therefore, even if people who have received a vaccine develop myocarditis, the situation is not something to be alarmed about. The speaker argues that the benefits of vaccination far outweigh the risks, and that the idea of significant changes or issues related to the vaccine is not supported. The overall conclusion presented is that the risks of myocarditis, whether from infection or vaccination, are outweighed by the benefits of vaccination. Key points reiterated include: - COVID-19 infection can cause myocarditis, sometimes with considerable severity or with structural heart complications. - Myocarditis can also occur after vaccination, but the occurrence is rare and the symptoms are generally mild. - The vast majority of people with vaccine-associated myocarditis recover. - The perceived risk of myocarditis following vaccination should not be a cause for alarm, given that the benefits of vaccination are greater. - There is no indication that anything about the vaccine itself changes in a way that would alter this risk-benefit balance. Overall, the message is that myocarditis is a potential outcome associated with both infection and vaccination, but the frequency is low, the illness is typically mild, recovery is common, and vaccination remains advantageous.

Studies have suggested myocarditis is a risk factor from COVID. However, it's claimed that the idea COVID is more of a cause of myocarditis and is more severe is untrue, based on literature reviews, assessments, and real-world experiences. Evidence supporting this claim was included in a written statement. An official information act request was made to determine the number of myocarditis cases due to COVID infection. The Ministry of Health couldn't provide a single case. Conversely, there were over 900 cases of myo- and pericarditis on the CALM safety report 46 by November 2022.

The COVID-19 vaccine can induce cardiac arrest. mRNA injections travel to all organ systems, including the heart, causing cardiomyocytes to produce spike proteins, which are also found circulating in the bloodstream and can reach the heart. The largest COVID-19 vaccine safety study, involving 99 million people, showed a 600% increased risk of myocarditis after mRNA injections. The trigger for cardiac arrest is usually in the waking morning hours of sleep, 3AM to 6AM, or during sports or exercise when there's a surge in catecholamines. Cardiac events are known to occur during sleep when catecholamines rise and during exercise when oxygen demands and catecholamines increase. This is not supposed to be seen in young healthy adults, but rather in those with intrinsic heart disease or the very elderly.

We have experience with this vaccine in a billion people, showing it is safe. While there is a low risk of myocarditis with mRNA vaccines, the risk from COVID is higher than the vaccine's risk, especially in young men.

This vaccine has been widely used and proven safe in billions of people. The risk of myocarditis, especially in young men, is very low.

Before COVID-19, I only encountered two cases of myocarditis in my entire career as a cardiologist. It was a rare condition, usually caused by parvovirus or adenovirus. However, now I see two cases per day in the clinic. We have learned that COVID-19 can cause myocarditis. Various organizations, such as the Israeli and US military, as well as college leagues, conducted extensive screening programs for COVID-induced myocarditis in 2020. They found a few cases that met the definition, but none were serious or resulted in hospitalizations or deaths. These screening programs were later discontinued when vaccines were introduced. However, within six months, regulatory agencies confirmed that the COVID-19 vaccines can cause myocarditis. It is important for people to understand that there is a risk of vaccine-induced myocarditis with every shot they take.

Myocarditis, or heart inflammation, is more common than previously believed. Recent studies show that around 20% of individuals who received the COVID vaccine experience myocarditis, as confirmed by echocardiograms and other tests. This means that out of every 1 million vaccinated people, around 200,000 will have evidence of heart damage. Unfortunately, those who develop myocarditis have a 50% chance of surviving only 5 years. This alarming increase in myocarditis cases is due to the cardiotoxic nature of the vaccine. These facts, shared by Dr. Cussell and Shoemaker from Toronto, highlight the concerning impact of the vaccine on heart health.

Before COVID-19, I only encountered two cases of myocarditis in my entire career as a cardiologist. However, now I see two cases per day in the clinic. We have learned that COVID-19 can cause myocarditis, and various organizations conducted screening programs in 2020. These programs found a few cases that met the definition of myocarditis, but none were serious or resulted in hospitalizations or deaths. After the introduction of vaccines, regulatory agencies acknowledged that the vaccines can cause COVID-19 vaccine-induced myocarditis, which can be fatal. It's important for people to understand that there is a risk associated with every vaccine shot they take.

Vaccination introduces mRNA into the bloodstream, which is taken up by major organs, including the heart. This process leads to the production of spike protein in heart muscle cells, resulting in inflammation and an increased risk of myocarditis. A large study indicated a 500% higher risk of myocarditis following COVID vaccination. Symptoms of myocarditis can be triggered during early morning hours (3 AM to 6 AM) when catecholamines like dopamine and epinephrine surge, as well as during exercise. These triggers can lead to serious heart issues, including ventricular tachycardia and sudden death.

We are witnessing a surge in myocarditis cases post-vaccination, with numbers far exceeding pre-pandemic levels. Previously rare, I now see hundreds of cases in my practice, some fatal. Studies show up to 18,000 cases reported. The Hoelscher paper suggests vaccine-induced myocarditis as a likely cause of sudden adult death syndrome, with cases emerging years after vaccination. FDA regulations indicate a potential 15-year window of concern post-injection. This issue may be more widespread and long-lasting than we realize.

In October 2020, the FDA mentioned that myocarditis could be a result of the COVID vaccines. In June 2021, the FDA confirmed that the vaccines can cause heart inflammation. Prior to COVID, patients with myocarditis were advised not to exercise due to the risk of cardiac arrest. Now, there are 800 peer-reviewed papers on COVID vaccine-induced myocarditis. Two studies showed a 2.5% rate of heart damage after receiving the second or third vaccine dose. When heart damage occurs, there can be variations in electrical conduction, leading to reentry and fast heart rhythms like ventricular tachycardia. This can progress to ventricular fibrillation, which is fatal. A recent study confirmed that vaccine-induced myocarditis is always fatal.

Myocarditis is a small percentage of vaccine deaths, with clotting and bleeding being more common causes. Younger people dying means more life years lost. Vaccines have taken more life years than COVID in the US.