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Humanity was hit with two biological weapons: a manufactured SARS CoV two virus created in collaboration with UNC Chapel Hill and Wuhan, which exposed the entire population to the spike protein, and mRNA injections, which installed about 7x more spike into people per injection. It is believed that those who received the injections can shed spike protein onto others, particularly when freshly vaccinated and producing high levels of spike protein. This shedding is believed to occur via exosomes, either through breath or bodily fluids.

The Pfizer shot contains synthetic messenger RNA that stays in the body indefinitely, unable to be detoxed. It destroys toll-like receptors 3, 7, and 8, which are crucial for our immune system's defense against viruses and bacteria. This makes vaccinated individuals more susceptible to COVID-19. The spike protein from the shot enters the cell nucleus, binds to DNA, and blocks repair enzymes, potentially leading to cancer. There is evidence of an increase in cancer cases among vaccinated individuals. Multiple shots further weaken the immune system, with German data suggesting that by the end of 2022, fully vaccinated individuals over 30 may have immune suppression similar to AIDS.

Bonjour à tous, Anaïs Bloqué, docteur en biologie santé, explique les impacts de la protéine Spike du SARS-CoV-2 sur le système immunitaire inné, basés sur son article récent. La Spike seule n'active pas complètement le TLR 4, un récepteur immunitaire, et ne permet pas la production d'interférons de type 1, essentiels pour lutter contre le virus. Pour une activation complète, la Spike doit s'associer au LPS (des bactéries gram négatifs). L'activation des interférons 1 augmente l'expression d'ACE2, le récepteur du virus, sensibilisant l'organisme à l'infection. Les interférons 1 peuvent aussi être activés par les RLR, notamment MDA5, qui détecte l'ARN messager modifié des vaccins anti-COVID. De plus, la Spike, protéine amyloïde, peut déclencher le TLR 4 en s'associant aux fibres amyloïdes A bêta 42, créant un "double effet amyloïde". L'augmentation de NF-κB par les interférons 1 peut bloquer la p53, un suppresseur de tumeur, et induire l'expression du MIR-29b, qui bloque ACE2. Chez les personnes avec comorbidités, ayant déjà de faibles niveaux d'ACE2, cette interaction Spike-LPS devient dangereuse, créant une boucle d'amplification inflammatoire. La Spike persiste longtemps dans l'organisme, et avec ses propriétés amyloïdes, pourrait entraîner des pathologies dégénératives à long terme. --- Hello everyone, Anaïs Bloqué, Doctor of Philosophy in Health Biology, explains the impacts of the SARS-CoV-2 Spike protein on the innate immune system, based on her recent article. The Spike alone does not fully activate TLR 4, an immune receptor, and does not allow the production of type 1 interferons, which are essential for fighting the virus. For complete activation, the Spike must associate with LPS (from gram-negative bacteria). Activation of interferon 1 increases the expression of ACE2, the virus's receptor, sensitizing the body to infection. Interferons 1 can also be activated by RLRs, in particular MDA5, which detects the modified messenger RNA of anti-COVID vaccines. In addition, Spike, an amyloid protein, can trigger TLR 4 by associating with amyloid A beta 42 fibers, creating a "double amyloid effect". The increase in NF-κB by interferons 1 can block p53, a tumor suppressor, and induce the expression of MIR-29b, which blocks ACE2. In people with comorbidities, who already have low levels of ACE2, this Spike-LPS interaction becomes dangerous, creating an inflammatory amplification loop. Spike persists in the body for a long time, and with its amyloid properties, could lead to long-term degenerative pathologies.

The speaker discusses a hypothesis regarding the connection between COVID-19 and the vaccines. They mention that SARS-CoV-2 was a product of dual-use research and that the spike protein in the vaccines is also from SARS-CoV-2. They explain that some people who received multiple vaccine shots experienced an interesting effect called IgG 4, which turns down the immune response. The speaker suggests that if the vaccines induce this attenuation signal, it could potentially make a population less reactive to a pathogen. They note that the Chinese did not use mRNA vaccines like other countries, which could mean that populations are now different in terms of their immune response. The speaker acknowledges that this is only a hypothesis and lacks evidence. They also express concerns about the widespread vaccination efforts and the unknown long-term impacts.

The speaker describes a nationwide study conducted in South Korea, stating that every resident was included in the research. The study compared individuals who received the vaccine to those who did not, and the analysis was stratified by dose number (one dose, two doses, three doses, and four or more doses). A central claim of the speaker is that this study provides the strongest signal to date supporting vaccine acquired immunodeficiency syndrome, referred to as VADES. According to the speaker, as each dose was administered, the immune function of individuals declined. By the time of the fourth dose, the speaker asserts there was a significant increase in the risk of other infections, quantified as about a 550% increase, including infections such as the common cold, tuberculosis, and upper respiratory tract infections. The speaker notes that the effect was most pronounced in young people, specifically ages zero to nineteen, who reportedly had the highest risks of these other infections. The implication presented is that the injections are causing immune collapse and exhausting T cells, leading to immune dysregulation described as IgG4 class switching. The immune system is said to become dysfunctional as a result. Additionally, the speaker mentions that, consistent with other studies they reference, genes related to immune function are claimed to become shut down. The overall assertion is that these findings point to a troubling pattern of immune impairment associated with multiple vaccine doses, culminating in the claimed immune dysfunction and increased susceptibility to other infections. The speaker emphasizes the magnitude and reliability of the sample size, stating that having an entire country’s population as the study cohort constitutes the strongest possible sample size. The summary of the presented claims centers on dose-dependent immune decline, a marked increase in non-target infections after the fourth dose, greater impact on children, evidence of immune system exhaustion and dysregulation, and purported genetic downregulation of immune pathways, all described as arising from the vaccination regimen in this nationwide South Korean study.

Professor Jean-Marc Sabatier's recent study sheds light on the toxicity of the Spike protein produced by mRNA vaccines. This protein disrupts the renin-angiotensin system in our organs, leading to numerous side effects. The Spike protein also affects the anti-oncogenic protein p53, which repairs damaged DNA. By inhibiting DNA repair, the Spike protein can contribute to the development of autoimmune diseases and cancers, even years after vaccination. The proposed remedy, the vaccine, is deemed more dangerous than the potential harm it aims to prevent, especially since it does not protect against reinfection or transmission. Despite personal convictions, the speaker remains in their position as a doctor and parent to protect their children and patients from complications and allow them to continue their education and work.

Je suis Anaïs Bloqué, docteur en biologie, expliquant les impacts de la protéine Spike du SARS-CoV-2 sur le système immunitaire. La Spike seule ne déclenche pas la production d'interférons de type 1, nécessitant une association avec le LPS pour activer le TLR4. Cette interaction peut augmenter l'expression d'ACE2, favorisant l'infection virale. Des boucles inflammatoires complexes peuvent se former, menant à des conséquences potentiellement dangereuses, surtout chez les individus avec des comorbidités. La persistance de la protéine Spike dans l'organisme peut entraîner des problèmes dégénératifs à long terme.

Speaker: Humanity was hit with dual biological weapons. When I say dual, I mean by the manufactured SARS CoV two virus that was manufactured in collaboration with UNC Chapel Hill and Wuhan. So we have that that has exposed basically the entire population to the spike protein. But then we also had the second biological weapon, was the mRNA injections, which installed about 7x more spike into people and those who received it per injection. And now we do understand that this can indeed shed onto others, particularly when you're freshly vaccinated and you're producing high levels of spike protein. We do believe this can shed via what's called exosomes, either through your breath or through your bodily fluids. So, yes, humanity has been hit by these weapons.

Cette vidéo résume: selon Nakaoota et al. (3 avril 2025), « l’expression de la protéine Spike chez 43.8 pour 100 des personnes vaccinées anti Covid » persiste « au niveau des artères coronaires » jusqu’à 17 mois après l’injection, avec « l’ARN messager, du vaccin, mais également du virus » détecté. Il y a « persistance de la protéine Spike » et « persistance du SARS-CoV-2 » possiblement malgré les traitements précoces. L’auteure mentionne aussi « Crüssfeld Jacob, 14 mois après infection » et une étude sur des « AVC 17 mois après injections ». L’interaction de la nucléocapside (protéine N) avec TRIM28 pourrait retarder la réponse immunitaire innée, renforçant la tolérance immunitaire via TLR2/RAGE et IgG4. Conséquences: infection potentiellement asymptomatique et dégâts cumulatifs; Spike persistante pourrait entraîner « spike viral plus spike vaccinal ». Des spycopathies neurologiques sont évoquées; dépistage de Spike et traque du virus recommandés; traitements personnalisés et soutien par curcumine, quercétine, vitamine D; approche individuelle. This video summarizes: according to Nakaoota et al. (April 3, 2025), « the expression of the spike protein in 43.8 per 100 of vaccinated people » persists « at the level of the coronary arteries » for up to 17 months after injection, with « mRNA from the vaccine, but also the virus » detected. There is « persistence of the Spike protein » and « persistence of SARS-CoV-2 » possibly despite early treatments. The author also cites « Crüssfeld Jacob, 14 months after infection » and a study on « strokes 17 months after injections ». The interaction of the nucleocapsid protein (N) with TRIM28 could delay the innate antiviral response, reinforcing immune tolerance via TLR2/RAGE and IgG4. Consequences: potentially asymptomatic infection and cumulative damage; persistent Spike could lead to « spike viral plus spike vaccinal ». Neurological spycopathies are discussed; diagnostics to detect Spike and tracking the virus are recommended; therapies to block/remove Spike and personalized approaches, with supports like curcumin, quercetin, vitamin D.

**French Summary:** Anaïs Bloqué explique l'impact de la protéine Spike du SARS-CoV-2 sur le système immunitaire inné, en se concentrant sur le TLR 4. La Spike seule n'active pas complètement le TLR 4 pour induire une réponse antivirale complète (interférons de type 1). L'association Spike-LPS (lipopolysaccharide bactérien) est nécessaire. L'activation des interférons 1 augmente l'expression d'ACE2, récepteur du virus, via les ISG, sensibilisant l'organisme à l'infection. Les ARN messagers des vaccins peuvent aussi activer les interférons 1 via MDA5. La Spike, protéine amyloïde, peut induire la production de fibres A bêta 42, aggravant l'inflammation. L'augmentation de NF-κB par les ISG peut bloquer p53 (suppresseur de tumeur) et induire le micro-ARN MIR-200c, diminuant l'expression d'ACE2. Chez les personnes avec comorbidités (diabète, obésité), une boucle d'amplification inflammatoire Spike-LPS-TLR4 réduit l'ACE2 disponible, menant à une suractivation de l'angiotensine 2. La Spike persistante pourrait causer des pathologies dégénératives à long terme. **English Translation:** Anaïs Bloqué explains the impact of the SARS-CoV-2 Spike protein on the innate immune system, focusing on TLR 4. Spike alone does not fully activate TLR 4 to induce a complete antiviral response (type 1 interferons). The Spike-LPS (bacterial lipopolysaccharide) association is necessary. Activation of interferon 1 increases ACE2 expression, the virus receptor, via ISGs, sensitizing the body to infection. Vaccine mRNAs can also activate interferon 1 via MDA5. Spike, an amyloid protein, can induce the production of A beta 42 fibers, worsening inflammation. Increased NF-κB by ISGs can block p53 (tumor suppressor) and induce microRNA MIR-200c, decreasing ACE2 expression. In people with comorbidities (diabetes, obesity), a Spike-LPS-TLR4 inflammatory amplification loop reduces available ACE2, leading to overactivation of angiotensin 2. Persistent Spike could cause long-term degenerative pathologies.

The speaker explains that the SARS CoV-2 virus, which causes COVID-19, has been successfully isolated and observed under a microscope. They show how the virus affects the lungs, causing scar tissue and disrupting blood flow. The virus enters the body through the ACE2 receptors and triggers an inflammatory pathway, leading to complications. The speaker highlights that the virus affects blood vessels throughout the body, including the brain, heart, lungs, kidneys, and GI tract. This widespread damage occurs due to the activation of the AT1R pathway, which triggers various inflammatory cells and molecules. Overall, COVID-19 damages multiple organs by instigating an internal battle within the blood vessels.

The speaker reflects on the pandemic, describing it as a time of “miracles,” including not losing anyone and continuing to speak today, despite controversy surrounding her research. She emphasizes that the controversy has hindered the advancement of research and science, urging instead to ask questions as science is about questioning and pushing narratives. She asserts a specific finding: the spike protein reduces bifidobacteria. She explains that the vaccine caused bifidobacteria to die within a month, but the effect persisted, with data indicating zero bifidobacteria in long-COVID or vaccine-injured cases. She notes she has been dealing with this for five years and asserts that people with zero bifidobacteria experience ongoing loss of microbiome diversity and immunity, resulting in poor immunity. She highlights bifidobacteria’s role in absorbing sugar, and adds that another microbe responsible for calcium absorption is also destroyed, leading to impaired calcium uptake. From these observations, she links cellular-level consequences to mitochondrial function, describing how a lack of sugar and calcium results in energy shortfalls and a disrupted Krebs cycle, implying mitochondrial dysfunction. She concludes that long COVID is a spike protein injury and that in many cases these individuals have zero bifidobacteria whether due to the treatment, the virus, or the spike protein itself. She also notes that some patients still have residual COVID in their stools, underscoring the need to pay attention to this finding. Key points emphasized: - The pandemic featured perceived miracles and ongoing controversy around research and vaccines, which the speaker argues stifles scientific progress. - A claim that the spike protein reduces bifidobacteria; the vaccine allegedly kills bifidobacteria within a month, with long-COVID or vaccine-injured individuals showing zero bifidobacteria across the line. - Zero bifidobacteria is linked to loss of microbial diversity, compromised immunity, and poor immune function. - Bifidobacteria’s role in absorbing sugar and a related microbe’s role in calcium absorption are highlighted as critical, with their destruction affecting cellular energy and mitochondrial function. - Long COVID is described as a spike protein injury, with some cases having residual COVID in stools, suggesting the need for attention to these microbial findings.

The Pfizer vaccine uses synthetic messenger RNA that replicates indefinitely in cells, making it impossible to detoxify from it. This mRNA damages toll-like receptors 3, 7, and 8, which are crucial for the innate immune response, increasing susceptibility to infections like COVID. Consequently, vaccinated individuals are more likely to become ill and face higher hospitalization rates. The spike protein can bind to DNA, potentially leading to abnormal cell growth and cancer, which explains the rise in various cancers among vaccinated individuals. Recent data indicates that vaccinated people are significantly more likely to contract omicron, and ongoing vaccinations may lead to severe immune suppression, comparable to AIDS, particularly in those over 30.

Il y a eu une manipulation du virus, avec des séquences ajoutées, notamment du VIH. Ce n'est pas un processus naturel, mais le résultat d'un travail minutieux de biologistes moléculaires. Les raisons de cette manipulation ne sont pas claires, mais il est possible qu'il s'agisse d'une tentative de créer un vaccin contre le sida en intégrant des séquences du VIH dans le coronavirus. Le matériel génétique du virus contient des segments de VIH qui pourraient modifier des sites antigéniques, permettant ainsi de modifier la protéine pour un vaccin. Bien que des rumeurs sur une origine humaine aient circulé, elles ont été largement réfutées, mais il existe une volonté d'étouffer ces recherches, comme l'a montré un groupe de chercheurs indiens qui a été contraint de se rétracter. --- There has been manipulation of the virus, with sequences added, including from HIV. This is not a natural process but the result of meticulous work by molecular biologists. The reasons for this manipulation are unclear, but it may involve an attempt to create an AIDS vaccine by integrating HIV sequences into the coronavirus. The virus's genetic material contains segments of HIV that could modify antigenic sites, allowing for changes to the protein for a vaccine. Although rumors of a human origin circulated, they have been largely refuted, yet there is a desire to suppress this research, as evidenced by a group of Indian researchers who were forced to retract their findings.

Speaker introduces herself as 'the girl that brought vaccines to market' and cites data that 'the messenger RNA killed the bifidobacteria,' claiming this controversy has halted research and science. She asserts, 'What we discovered with the vaccine is that it did kill the bifida bacteria within a month, persisted in killing the bifida bacteria.' She notes daily reports of 'long COVID or vax' and outlines her interview approach: asking patients if they had COVID, were vaccinated, and whether they contracted COVID after vaccination, concluding 'did the vaccine kill their bifida bacteria.' She describes the immune concept as thinking of the body as 'a group of communities, group of gangs or communities in your gut,' warning that a 'foreigner' prompts autoimmunity. She argues vaccination may 'kill your bifidobacteria,' leading to timing effects on long COVID or vaccine injury, with 'zero bifidobacteria across the line.'

The spike protein of the coronavirus plays a crucial role in triggering a strong antibody response. To study it in the lab, Keith uses a small fragment of HIV as a clamp to lock the spike protein into its original shape. This helps maintain the structure of the virus on its surface.

The speaker explains that the spike protein on the coronavirus is crucial for its structure and interaction with our cells. To trigger a protective antibody response, Keith replicates the spike protein in the lab and locks it into the same shape using a clamp-like protein. Surprisingly, this clamp-like protein is a small fragment of HIV.

The spike protein from the COVID-19 virus circulates in the body and can land in multiple organs, causing various diseases. Lab studies have shown that even without the virus, just injecting the spike protein can induce the same lung, vascular, heart, and brain diseases as COVID-19. The spike protein is considered the toxin responsible for causing the disease. This raises questions about why we are injecting something that is essentially a toxin into the human body, as it is not a traditional vaccine.

Dr. Yeason presents three principal assertions about the vaccines. 1) The first principle: we were told these molecules were gene sequences that encoded something called the spike protein. The spike protein is described as on the outside of the virus, and vaccines were said to encode the protein to train the immune system. Dr. Yeason explains that the immune system treats anything foreign as a threat and will attack cells that manufacture a foreign protein, leading to tissue damage. He notes that this principle of “self, non self” and tissue targeting is fundamental to organ transplantation and autoimmune diseases, and says this was designed into every company’s molecule (Moderna, Pfizer, Johnson & Johnson, AstraZeneca). He asserts that by 2020 he knew these were designed to cause injury. 2) The second principle: what was encoded is the spike protein. He states he did not know what spike protein was at first, but describes spikes on the outside of the virus and claims they are known toxins (neurotoxins, cardiotoxins) that prompt blood coagulation. He questions why a medicinal product would encode something that would harm the body when expressed. 3) The third principle: lipid nanoparticles (LNPs) used to formulate two of the Pfizer and Moderna products. He explains that lipid nanoparticles are toxic in general and are known to promote uptake of their payload into visceral organs, especially the liver and ovaries. He asserts that when injected into women and girls, these materials would travel through the body, concentrate in reproductive organs, be expressed, be recognized as foreign, and kill those cells. He asks what possible motivation there could be for using that formulation when other options exist. This, he says, confirms that the first two observations were not mere risks but intentional design. Dr. Yeason concludes that these three points together indicate that someone in a room designed injections to injure, kill, and reduce fertility in the people given them, aiming to lower fertility and reduce the population over time. He states he has observed this “all around me for five years since that moment.”

The spike and pseudouridine in the shots are changing receptor patterns on cells, suppressing the immune system's ability to fight off viruses like herpes, HPV, and RSV. Toll-like receptors, which train cells to fight cancer, are also being suppressed. These receptors are like the marines of our immune system, constantly circulating and identifying friend or foe. However, the shots are causing these marines, along with dendritic cells and macrophages, to become inactive, leaving the body defenseless against cancer cells and pathogens. It is unclear when this suppression stops or how to reverse it. While not affecting everyone, the degree to which it is happening is concerning.

Bonjour, je suis Anaïs Bocquet, docteur en biologie santé, spécialisée en immunologie. Je vais vous parler du virus SARS-CoV-2 et de sa protéine Spike. Cette protéine est un GOF, clé d'entrée du virus dans l'organisme. Elle présente des propriétés persistantes, bactériophages et cancérogènes. Elle est également amyloïde, fusogène et impacte le système immunitaire. Les injections anti-Covid produisent une protéine Spike similaire à celle du virus, avec les mêmes propriétés. Cela peut causer des problèmes à long terme. Il est important de comprendre comment le virus et la protéine Spike interagissent avec d'autres pathogènes, comme les bactéries. Les questions scientifiques sont nombreuses et nécessitent l'intervention d'experts dans divers domaines. Merci de votre attention. Translation: Hello, I am Anaïs Bocquet, a doctor in health biology, specialized in immunology. I will talk to you about the SARS-CoV-2 virus and its Spike protein. This protein is a GOF, the key to the virus entering the body. It has persistent, bacteriophage, and carcinogenic properties. It is also amyloid, fusogenic, and impacts the immune system. Anti-Covid injections produce a Spike protein similar to that of the virus, with the same properties. This can cause long-term problems. It is important to understand how the virus and the Spike protein interact with other pathogens, such as bacteria. There are many scientific questions that require the intervention of experts in various fields. Thank you for your attention.

Bonjour à tous, Anaïs Bloqué, docteur en biologie santé, explique les impacts de la protéine Spike du SARS-CoV-2 sur le système immunitaire inné, basés sur son article récent. La Spike seule n'active pas complètement le TLR 4, un récepteur immunitaire, et ne produit pas d'interférons de type 1, essentiels pour la réponse antivirale. Pour une activation complète, la Spike doit s'associer au LPS (des bactéries Gram négatif). L'activation des interférons 1 augmente l'expression d'ACE2, le récepteur du virus, via les ISG, sensibilisant l'organisme à l'infection. Les ARN messagers des vaccins peuvent aussi lancer la production d'interférons 1 via MDA5. La Spike, protéine amyloïde, peut aussi déclencher le TLR 4 avec des fibres amyloïdes, entraînant un "double effet amyloïde". L'augmentation de NF-κB par les ISG peut bloquer la p53, potentiellement cancérigène. De plus, NF-κB induit le MIR-200c, qui bloque l'ACE2. Chez les individus avec comorbidités, une boucle d'amplification inflammatoire se crée : Spike-LPS-TLR4 induit interférons 1, ISG, surexpression d'ACE2, augmentation de NF-κB, MIR-200c, diminution d'ACE2 et augmentation d'angiotensine 2. La Spike persiste longtemps, et ses propriétés amyloïdes font craindre des pathologies dégénératives à long terme. --- Hello everyone, Anaïs Bloqué, PhD in health biology, explains the impacts of the SARS-CoV-2 Spike protein on the innate immune system, based on her recent article. Spike alone does not fully activate TLR 4, an immune receptor, and does not produce type 1 interferons, which are essential for the antiviral response. For complete activation, Spike must associate with LPS (from Gram-negative bacteria). Activation of interferon 1 increases the expression of ACE2, the virus's receptor, via ISGs, sensitizing the body to infection. Vaccine mRNAs can also trigger the production of interferon 1 via MDA5. Spike, an amyloid protein, can also trigger TLR 4 with amyloid fibers, leading to a "double amyloid effect." The increase in NF-κB by ISGs can block p53, which is potentially carcinogenic. In addition, NF-κB induces MIR-200c, which blocks ACE2. In individuals with comorbidities, an inflammatory amplification loop is created: Spike-LPS-TLR4 induces interferon 1, ISG, ACE2 overexpression, increased NF-κB, MIR-200c, decreased ACE2 and increased angiotensin 2. Spike persists for a long time, and its amyloid properties raise concerns about long-term degenerative pathologies.

Speaker 1 explains cancer is usually a mutation, and the immune system surveils with 30,000,000 T cells. After vaccination there is suppression of many cell lines. The mRNA shot uses pseudouridine in a lipid nanoparticle to evade the immune system, and it can "hijack your cells, and makes your cells the spike factory." The spike "breaks off of the surface of your cell and goes into circulation." Pseudouridine can cause decrease in certain protein kinase pathways and certain retinoic acid receptor pathways, many things that are responsible for normal cell function and then can lead to mutagenesis as well. Stanford study by Doctor Woltkin et al shows "up to sixty days later, the synthetic mRNA is still in lymph nodes and spike is still circulating." Toll-like receptors downregulated: seven, eight, three, four; Dr. Fossa notes downregulation linked to multiple cancers. Micro RNA array disruption and G protein disruptions are mentioned.

In the lab, it's easy to manipulate spike proteins, which play a significant role in the zoonotic risk of coronaviruses. By obtaining the sequence and constructing the protein, we collaborated with Ralph Barrick at UNC to insert it into another virus. This allows us to conduct experiments and enhance our ability to predict outcomes based on specific sequences.

Dr. Michael Palmer, an MD and professor of biochemistry, discusses the mechanisms by which COVID mRNA vaccines can cause toxicity. He identifies four major mechanisms: the spike protein triggering an immune response, toxic activity of the lipid nanoparticles used to deliver the mRNA, toxicity of the spike protein itself, and contamination of the vaccines with DNA. He explains how these mechanisms can lead to inflammation, blood clotting, and other adverse events. Dr. Palmer emphasizes that the immune attack on spike-expressing cells is the main driver of vaccine damage. He concludes that mRNA vaccines are a bad idea and urges support for scientist Sutra At Bakhti, who has faced persecution for speaking out against the vaccines.