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The speaker states that the NIH has a division devoted to studying long COVID and figuring out cures. They are also incorporating an agency within the CDC that will specialize in vaccine injuries. These issues, along with Lyme disease, are priorities because more and more people are suffering from these injuries. The speaker claims they are committed to having gold standard science to figure out what the treatments are and deliver the best treatments possible.

Speaker 0: Because there was such a mass vaccination campaign with a product that, you know, tens of thousands, hundreds of thousands of people were injured in this process, what work is the NIH doing in terms of research to somehow help these people? Because just from my own experience, my wife and I made a film about this, right? These people were, even though in some cases they were supported a bit by, but mostly just completely gaslit and just, no, your issue doesn't exist. Right? So how are you approaching this? Speaker 1: Well, you're absolutely right. There were absolutely like, lot patients of who were vaccine injured were gaslit, pretending as if they didn't get injured or that somehow their symptoms are all Speaker 1: in their head or something. Actually, this is part of a broader phenomenon, where, you have patients with conditions that are poorly understood, where the medical system will gaslight them leave. They can they're telling you it's a a psychological issue rather than a physical issue. It should make you think that you're crazy because you you you have symptoms that you just, you know you have, but you can't convince anyone else to do anything about it. Injury is one of them, long COVID, MECFS, Lyme chronic Lyme disease, a whole host of these conditions where it just fits a very similar pattern. Speaker 1: The key underlying thing is that there isn't excellent science to guide decision making for clinicians or anybody else, for patients. And I've made sure that people know at the NIH that I'm very interested in investing in answers for patients for all of those. Vaccine injury, long COVID, MECFS, chronic Lyme. We need to get better answers. The the gaslighting happens because the, if you're let's say you're a doctor and you see a patient and you have no idea what's causing their condition. Speaker 1: Right? Because the scientific literature doesn't have an answer. You're gonna be unless you're an amazing doctor who's really good at, you know, sort of being honest and compassionate, you're going to be wanting to, like, move on to the next patient. And, it's really, really unfortunate. The answer is to get good answers, right? Speaker 1: So invest in, research on treatments, on underlying physiology, physiological causes, you know, basic biological knowledge, so that those patients actually can can the doctors and the caregivers for those patients can will treat them correctly. Speaker 0: So but is is NIH doing this for people that are that have been COVID vaccine injures against a huge number of people relatively. Speaker 1: We have investments in that, and we're going have more investments in that at the start, you know, this year. For all of those conditions, I think patients deserve an answer, and I'm definitely, interested in finding I would love to know myself.

Some research suggests the coronavirus can infect brain cells, impacting memory and cognitive functions. Understanding this could help treat long COVID symptoms like brain fog and fatigue. Early treatment may prevent brain damage.

Having COVID may lead to a slight decline in cognitive function, with a 3-point IQ loss for those who recovered within 12 weeks and a 9-point loss for ICU patients. Long COVID patients experience more significant deficits. The study suggests these effects may improve over time. Doctor Adam Hampshire finds the findings promising, especially for those with persistent symptoms. Joanna, who has long COVID, highlights the need for better support and access to clinics for those affected. More research and funding are essential for effective care and management.

The speaker claims that chemotherapy first wipes out red blood cells, causing anemia, with Epogen; second, neutrophils that prevent infection, with Neupogen; and most importantly, NK and T cells—the lymphocytes. He says, "the only thing that protects your body against cancer is your lymphocytes, meaning the NK cells and T cells," and that, with chemotherapy or radiation, "within a day or two, you wipe out the only cells that matter, i.e. The cells that kill can." He notes, "for thirty five years, we've never had a treatment for that." He links this to long COVID: during viral infection, "the virus is smart, it wipes out the T cells and NK cells." He points out the irony that Epogen and Neupogen are needed because chemotherapy has wiped out the cells that matter. A South African pancreas transplant surgeon says, "everything we've done so far has been wrong. We've actually treated cancer wrongly."

On October 28, 2024, I listened in disbelief as some doctors explained why vaccinated individuals seem to get sick more often, claiming it's expected and that they recover fully. In reality, this reflects a deliberate weakening of their immune systems. I've seen this with cancer patients who, after treatment, would be in remission but then succumb to minor illnesses. This pattern mirrors the past with HIV, where people were told they could be asymptomatic yet seriously ill, leading to treatments that further compromised their health. It's shocking that people can't recognize this trend. Why would a medicine that’s supposed to help make you sicker? It's time to wake up and see the truth.

The blood brain barrier is a protective filter that allows essential substances into the brain while keeping out harmful ones. COVID-19 can disrupt this barrier, leading to brain damage and cognitive issues even in mild cases. Studies show brain shrinkage, cognitive impairment, and potential long-term effects on memory and thinking abilities. Research on dogs suggests that even asymptomatic cases of COVID-19 can cause brain damage similar to early signs of Alzheimer's. Preventing the spread of the virus through measures like wearing masks is crucial to avoid repeated infections and potential long-term brain damage.

The blood brain barrier is a protective membrane that controls what enters the brain. COVID-19 can disrupt this barrier, leading to brain damage and cognitive impairment even in mild cases. Studies on infected individuals show brain shrinkage, cognitive decline, and potential long-term consequences like dementia. Animal studies suggest that even asymptomatic cases may have brain damage that could lead to severe neurological disorders. To prevent further harm, it's crucial to reduce infections by wearing masks and improving air quality. The current trajectory of repeated infections with a brain-damaging virus is unsustainable, and we must prioritize stopping the spread of COVID-19.

It's a common misconception that doctors would already be implementing effective treatments. Medicine is often slow to adopt new discoveries. For example, people died of scurvy for centuries, despite repeated observations that citrus fruits could prevent it. Doctors dismissed these findings, causing recurring outbreaks until the discovery of vitamin C. Today, the major health challenges are complex chronic illnesses like Alzheimer's, dementia, ALS, cancers, and cardiovascular disease. These require a different approach than past diseases like pneumonia and TB. Early intervention is crucial, before symptoms manifest. Wearable devices like Oura Rings, Apple Watches, and Fitbits can be very helpful in monitoring changes in sleep, heart rate variability, and other metrics, enabling earlier detection and intervention.

The hardest part of being COVID aware is watching people unknowingly get infected and risk long COVID. The virus weakens the immune system, making people susceptible to other illnesses. Many are unaware of the risks and continue as if it's 2019. The lack of measures and education contributes to the spread. Long COVID can cause brain damage and heart issues. It's concerning to see so many unaware of the dangers they face.

Researchers at Macquarie University discovered that the COVID-19 virus causes brain cells to malfunction, leading to symptoms like loss of smell and brain fog. They used mini brains made from human stem cells to mimic brain activity. The mini brains were infected with the virus at the Queensland Brain Institute, showing fused cells where the virus hides. In some cases, neuronal activity stopped completely.

The speaker envisions a future in which everything will be linked to microbes, including cancer. They point to current examples such as HPV cervical cancer, Epstein-Barr virus with Burkitt’s lymphoma, and Helicobacter pylori with gastric cancer to illustrate how specific microbes are associated with particular cancers. They suggest it is only a matter of time before doctors begin saying that certain cancers, like colon cancer, are associated with specific bacteria, referring to a hypothetical “colon cancer with X bacteria.” This framing implies that cancer development could be driven or influenced by the presence of particular microbial communities. From there, the speaker raises the question of how to neutralize a particular microbe in order to prevent it from contributing to cancer alongside another microbe. They emphasize that microbes are constantly present and interacting, describing a ongoing “war in our guts” where microbes compete and influence disease outcomes. The idea is that some microbes are beneficial, or “good ones,” and that understanding these relationships is key to prevention and treatment strategies. A central claim the speaker highlights is what has been learned from the COVID experience: it reveals the ability of a microbe to survive inside a virus, but also the ability of a virus to cause death in a person. This observation reinforces the notion of a complex battle between microbes themselves and between microbes and viruses, where outcomes depend on how different organisms interact with one another. The speaker stresses that the crucial insight lies in identifying which microbe neutralizes which other microbe, suggesting that these inter-microbial dynamics could determine disease progression and outcomes. Ultimately, the speaker defines this understanding as “the key to the whole research that I’m doing.” The emphasis is on mapping out the interactions between microbes and viruses, recognizing the dual role of microbes as potential drivers of disease and as possible targets for interception, and using that knowledge to guide the research trajectory aimed at preventing cancer and other illnesses by modulating the microbiome.

The comparison to HIV is important because, like early HIV infections, mild or moderate COVID can cause unseen destruction. With HIV, people were infected for years before symptoms appeared, while the virus quietly destroyed the immune system. However, the HIV epidemic spurred brilliant science that changed how HIV is treated. We are now learning about mitochondria, viral impact, brain fog, changes in neurons, and cells that nourish neurons because of Long COVID. The goal is to reach a point where, through research, people with Long COVID can not only survive but thrive, just as HIV patients can live normal lifespans today.

The speaker reflects on the pandemic, describing it as a time of “miracles,” including not losing anyone and continuing to speak today, despite controversy surrounding her research. She emphasizes that the controversy has hindered the advancement of research and science, urging instead to ask questions as science is about questioning and pushing narratives. She asserts a specific finding: the spike protein reduces bifidobacteria. She explains that the vaccine caused bifidobacteria to die within a month, but the effect persisted, with data indicating zero bifidobacteria in long-COVID or vaccine-injured cases. She notes she has been dealing with this for five years and asserts that people with zero bifidobacteria experience ongoing loss of microbiome diversity and immunity, resulting in poor immunity. She highlights bifidobacteria’s role in absorbing sugar, and adds that another microbe responsible for calcium absorption is also destroyed, leading to impaired calcium uptake. From these observations, she links cellular-level consequences to mitochondrial function, describing how a lack of sugar and calcium results in energy shortfalls and a disrupted Krebs cycle, implying mitochondrial dysfunction. She concludes that long COVID is a spike protein injury and that in many cases these individuals have zero bifidobacteria whether due to the treatment, the virus, or the spike protein itself. She also notes that some patients still have residual COVID in their stools, underscoring the need to pay attention to this finding. Key points emphasized: - The pandemic featured perceived miracles and ongoing controversy around research and vaccines, which the speaker argues stifles scientific progress. - A claim that the spike protein reduces bifidobacteria; the vaccine allegedly kills bifidobacteria within a month, with long-COVID or vaccine-injured individuals showing zero bifidobacteria across the line. - Zero bifidobacteria is linked to loss of microbial diversity, compromised immunity, and poor immune function. - Bifidobacteria’s role in absorbing sugar and a related microbe’s role in calcium absorption are highlighted as critical, with their destruction affecting cellular energy and mitochondrial function. - Long COVID is described as a spike protein injury, with some cases having residual COVID in stools, suggesting the need for attention to these microbial findings.

According to the speaker, 95-99% of polio cases are asymptomatic because poliovirus is a commensal organism. Studies by the Indian Health Service of the Javonte Indians in South America found that 98-99% of those tested had immunity to all three strains of polio, yet the tribe had no instances of crippled children, short legs, or deaths from respiratory failure. The speaker claims that most viruses become less problematic as they spread through the human system. They cite COVID-19 as an example, where the initial strain was highly virulent but less contagious, while later variants like Omicron were more contagious but less pathological. The speaker suggests that significant problems with microbes typically arise when they are reverse attenuated, made more lethal in a lab, and then introduced into the population.

Comparison to HIV is important as both were asymptomatic initially. HIV taught us about immunology and revolutionized cancer therapy. Long COVID is shedding light on mitochondria, viral impact, brain fog, and neuron changes. Despite the unseen damage of mild/moderate COVID, like HIV, it can lead to scientific breakthroughs. Research on long COVID is crucial for people to not just survive but thrive.

The speaker discusses the inflammatory and amyloidogenic effects of small sequences called epitopes, which can cause memory dysfunction in mice. They also mention a study that found the introduction of gene transfection technologies containing the spike protein can induce amyloidogenic cascades. The speaker highlights a 200% increase in the diagnosis of CJD in France after the rollout of vaccination programs, suggesting a potential link. They discuss the loss of cognitive function associated with exposure to the spike protein and propose that amyloidogenic disease processes may underlie long-haul COVID-19 symptoms. The speaker mentions the role of viral infections in facilitating intercellular aggregate dissemination and shares examples of misfolding prion amyloidogenic diseases.

People are recovering from chronic pain, long COVID, and other chronic symptoms through a new understanding of pain. Individuals report overcoming chronic pain that previously limited their lives, now engaging in activities like hiking, cycling, running, and performing on Broadway. Neurologist Dario Zagar (Yale), Becca Kennedy (former lead of the Long COVID Clinic at Kaiser Permanente Northwest), and neuroscientist Yoni Ashar (University of Colorado) are among experts highlighting that all pain is generated in the brain. Pain is a protector signal, but the brain can get confused, perceiving threat even when there is none. Studies show that treating chronic pain by addressing the brain's role can lead to significant improvement; one study found that three-quarters of chronic back pain sufferers were virtually pain-free in one month. Brain scans reveal that chronic pain shifts to brain regions related to emotion, learning, and meaning-making, creating a feedback loop. This new science suggests that symptoms can be unlearned, offering hope for conditions like migraines, chronic fatigue syndrome, fibromyalgia, and more. Over 7,000 people have successfully improved, and thousands who were formerly hopeless are now completely well.

Inflammation in the brain from COVID can lead to long-term cognitive issues. The high levels of inflammation seen in even mild cases of COVID worried me about a potential neurological crisis. The rates of lasting cognitive symptoms in COVID survivors are concerning. Effective therapy is crucial to help the millions who may suffer from these symptoms.

Having COVID may lead to a small cognitive decline, with a 3 point IQ loss for those recovering within 12 weeks, and a 9 point loss for ICU patients. Long COVID can cause brain fog, fatigue, and heart issues. The study shows improvements over time for some. More support is needed for long COVID patients, including better access to clinics and research funding for treatments.

The comparison to HIV is important because both viruses can be asymptomatic. HIV taught us a lot about immunology and changed cancer therapy. Similarly, we are now learning about the impact of the virus on mitochondria, brain fog, and our neurons through long COVID. Mild and moderate COVID can cause destruction, just like HIV did to our immune system. However, the brilliant science that came out of HIV research transformed how we treat the virus, allowing people to live normal lives. We need to do the same for long COVID, so that those affected can not only survive but also thrive.

COVID-19 causes the accumulation of senescent cells, also known as zombie cells, in the brain, which speeds up the aging process. However, there is a way to combat and reverse these effects. According to Dr. Julio Aguado from the University of Queensland, senescent cells contribute to neurodegeneration and decline during aging. The SARS-CoV-2 virus can induce these senescent cells, but there are drugs called senolytics that effectively eliminate them from the brain. This discovery could potentially revolutionize the treatment of Alzheimer's disease by reducing inflammation in the brain. These findings offer promising hope for combating the effects of COVID-19 on the brain.

The symptoms of long COVID, such as joint pain, difficulty concentrating, brain fog, and chronic fatigue, can significantly affect patients' lives. The severity and occurrence of these symptoms vary among individuals and are not necessarily related to age, gender, or the severity of the initial COVID infection. Factors like the type of vaccine, individual constitution, age, and gender may contribute to the development of long COVID. Vaccinated individuals may experience a greater impact from long COVID compared to those who are unvaccinated. It is important to distinguish long COVID from other conditions, and while the vaccine may play a role, it is not clear if it can trigger long COVID.

During COVID, I traveled the country and saw many undiagnosed diseases that could have been treated early, but resulted in COVID deaths. I also witnessed the deterioration of our health system in rural areas, where access to healthcare is limited. The hub and spoke model, designed to get very sick people into regional medical centers, was overwhelmed. COVID highlighted issues with chronic disease management. Similar to early HIV treatment, we initially only treated symptomatic individuals, which was just the tip of the iceberg. When we started finding and treating asymptomatic individuals early, before they showed disease, they could thrive.