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If a genetic sequence is injected that causes the body to manufacture a foreign protein, the body recognizes it as an invasion and launches an attack on cells. This autoimmune reaction can occur anywhere the injection lands, potentially causing myocarditis or a heart attack if it lands in the heart, stroke or neurological conditions if in the brain, blindness if in the eyes, or sterilization if in the ovaries. The body is being made to manufacture something that does not belong in it. The speaker believes the so-called vaccines encode spike proteins, which are acutely toxic to blood cells, prompting blood clots, and to nerve cells, causing them to malfunction. The body is forced to make something directly toxic, intentionally. The injectables are wrapped in lipid nanoparticles.

Bonjour à tous, Anaïs Bloqué, docteur en biologie santé, explique les impacts de la protéine Spike du SARS-CoV-2 sur le système immunitaire inné, basés sur son article récent. La Spike seule n'active pas complètement le TLR 4, un récepteur immunitaire, et ne permet pas la production d'interférons de type 1, essentiels pour lutter contre le virus. Pour une activation complète, la Spike doit s'associer au LPS (des bactéries gram négatifs). L'activation des interférons 1 augmente l'expression d'ACE2, le récepteur du virus, sensibilisant l'organisme à l'infection. Les interférons 1 peuvent aussi être activés par les RLR, notamment MDA5, qui détecte l'ARN messager modifié des vaccins anti-COVID. De plus, la Spike, protéine amyloïde, peut déclencher le TLR 4 en s'associant aux fibres amyloïdes A bêta 42, créant un "double effet amyloïde". L'augmentation de NF-κB par les interférons 1 peut bloquer la p53, un suppresseur de tumeur, et induire l'expression du MIR-29b, qui bloque ACE2. Chez les personnes avec comorbidités, ayant déjà de faibles niveaux d'ACE2, cette interaction Spike-LPS devient dangereuse, créant une boucle d'amplification inflammatoire. La Spike persiste longtemps dans l'organisme, et avec ses propriétés amyloïdes, pourrait entraîner des pathologies dégénératives à long terme. --- Hello everyone, Anaïs Bloqué, Doctor of Philosophy in Health Biology, explains the impacts of the SARS-CoV-2 Spike protein on the innate immune system, based on her recent article. The Spike alone does not fully activate TLR 4, an immune receptor, and does not allow the production of type 1 interferons, which are essential for fighting the virus. For complete activation, the Spike must associate with LPS (from gram-negative bacteria). Activation of interferon 1 increases the expression of ACE2, the virus's receptor, sensitizing the body to infection. Interferons 1 can also be activated by RLRs, in particular MDA5, which detects the modified messenger RNA of anti-COVID vaccines. In addition, Spike, an amyloid protein, can trigger TLR 4 by associating with amyloid A beta 42 fibers, creating a "double amyloid effect". The increase in NF-κB by interferons 1 can block p53, a tumor suppressor, and induce the expression of MIR-29b, which blocks ACE2. In people with comorbidities, who already have low levels of ACE2, this Spike-LPS interaction becomes dangerous, creating an inflammatory amplification loop. Spike persists in the body for a long time, and with its amyloid properties, could lead to long-term degenerative pathologies.

The speaker discusses a hypothesis regarding the connection between COVID-19 and the vaccines. They mention that SARS-CoV-2 was a product of dual-use research and that the spike protein in the vaccines is also from SARS-CoV-2. They explain that some people who received multiple vaccine shots experienced an interesting effect called IgG 4, which turns down the immune response. The speaker suggests that if the vaccines induce this attenuation signal, it could potentially make a population less reactive to a pathogen. They note that the Chinese did not use mRNA vaccines like other countries, which could mean that populations are now different in terms of their immune response. The speaker acknowledges that this is only a hypothesis and lacks evidence. They also express concerns about the widespread vaccination efforts and the unknown long-term impacts.

Professor Jean-Marc Sabatier's recent study sheds light on the toxicity of the Spike protein produced by mRNA vaccines. This protein disrupts the renin-angiotensin system in our organs, leading to numerous side effects. The Spike protein also affects the anti-oncogenic protein p53, which repairs damaged DNA. By inhibiting DNA repair, the Spike protein can contribute to the development of autoimmune diseases and cancers, even years after vaccination. The proposed remedy, the vaccine, is deemed more dangerous than the potential harm it aims to prevent, especially since it does not protect against reinfection or transmission. Despite personal convictions, the speaker remains in their position as a doctor and parent to protect their children and patients from complications and allow them to continue their education and work.

The speaker discusses how the spike protein in vaccines can lead to clotting issues, immune suppression, and reactivation of latent viruses like mono. This can also weaken the body's ability to fight off other viruses and cancers. An increase in cancer cases post-vaccination is noted anecdotally. The speaker attributes these effects to the spike protein in the vaccines.

Speaker: Humanity was hit with dual biological weapons. When I say dual, I mean by the manufactured SARS CoV two virus that was manufactured in collaboration with UNC Chapel Hill and Wuhan. So we have that that has exposed basically the entire population to the spike protein. But then we also had the second biological weapon, was the mRNA injections, which installed about 7x more spike into people and those who received it per injection. And now we do understand that this can indeed shed onto others, particularly when you're freshly vaccinated and you're producing high levels of spike protein. We do believe this can shed via what's called exosomes, either through your breath or through your bodily fluids. So, yes, humanity has been hit by these weapons.

Je suis Anaïs Bocquet, biologiste et enseignante. La protéine Spike active les récepteurs TLR 4 et 2, déclenchant des réponses inflammatoires. L'IL-10 régule l'immunité, tandis que les alarmines activent les cellules mdsc pour inhiber la réponse immunitaire. L'IL-10 favorise la surexpression de ACE2 et la production d'IgG4 anti-inflammatoires. L'activation de TLR 2 inhibe les TLR 7 et 9, stoppant la réponse aux interférons de type 1. La persistance de Spike favorise la tolérance immunitaire, augmentant la sensibilité aux infections virales. Le TLR 4 est crucial dans la bascule entre inflammation et tolérance. En résumé, Spike manipule l'immunité pour favoriser la pathogénèse virale.

"Now, when these genes, packages, enter the cells, then the cells will start making this damn virus protein which is called the spike." "This is going to happen to any mRNA or gene based vaccine." "Those packages are going to cause your cells in the blood vessels to create this protein and this protein is going to be a foreign non self protein that is going to be recognised by any antibodies that you have and these antibodies are going to be there after the first injection." "If any of these vessels is clogged because of a thrombus or because it's injured, the cells that are being supplied by oxygen are going to die." "So if these tiny vessels in the brain or the heart are damaged, you are damaged for life. You will never be the same again."

Cette vidéo résume: selon Nakaoota et al. (3 avril 2025), « l’expression de la protéine Spike chez 43.8 pour 100 des personnes vaccinées anti Covid » persiste « au niveau des artères coronaires » jusqu’à 17 mois après l’injection, avec « l’ARN messager, du vaccin, mais également du virus » détecté. Il y a « persistance de la protéine Spike » et « persistance du SARS-CoV-2 » possiblement malgré les traitements précoces. L’auteure mentionne aussi « Crüssfeld Jacob, 14 mois après infection » et une étude sur des « AVC 17 mois après injections ». L’interaction de la nucléocapside (protéine N) avec TRIM28 pourrait retarder la réponse immunitaire innée, renforçant la tolérance immunitaire via TLR2/RAGE et IgG4. Conséquences: infection potentiellement asymptomatique et dégâts cumulatifs; Spike persistante pourrait entraîner « spike viral plus spike vaccinal ». Des spycopathies neurologiques sont évoquées; dépistage de Spike et traque du virus recommandés; traitements personnalisés et soutien par curcumine, quercétine, vitamine D; approche individuelle. This video summarizes: according to Nakaoota et al. (April 3, 2025), « the expression of the spike protein in 43.8 per 100 of vaccinated people » persists « at the level of the coronary arteries » for up to 17 months after injection, with « mRNA from the vaccine, but also the virus » detected. There is « persistence of the Spike protein » and « persistence of SARS-CoV-2 » possibly despite early treatments. The author also cites « Crüssfeld Jacob, 14 months after infection » and a study on « strokes 17 months after injections ». The interaction of the nucleocapsid protein (N) with TRIM28 could delay the innate antiviral response, reinforcing immune tolerance via TLR2/RAGE and IgG4. Consequences: potentially asymptomatic infection and cumulative damage; persistent Spike could lead to « spike viral plus spike vaccinal ». Neurological spycopathies are discussed; diagnostics to detect Spike and tracking the virus are recommended; therapies to block/remove Spike and personalized approaches, with supports like curcumin, quercetin, vitamin D.

**French Summary:** Anaïs Bloqué explique l'impact de la protéine Spike du SARS-CoV-2 sur le système immunitaire inné, en se concentrant sur le TLR 4. La Spike seule n'active pas complètement le TLR 4 pour induire une réponse antivirale complète (interférons de type 1). L'association Spike-LPS (lipopolysaccharide bactérien) est nécessaire. L'activation des interférons 1 augmente l'expression d'ACE2, récepteur du virus, via les ISG, sensibilisant l'organisme à l'infection. Les ARN messagers des vaccins peuvent aussi activer les interférons 1 via MDA5. La Spike, protéine amyloïde, peut induire la production de fibres A bêta 42, aggravant l'inflammation. L'augmentation de NF-κB par les ISG peut bloquer p53 (suppresseur de tumeur) et induire le micro-ARN MIR-200c, diminuant l'expression d'ACE2. Chez les personnes avec comorbidités (diabète, obésité), une boucle d'amplification inflammatoire Spike-LPS-TLR4 réduit l'ACE2 disponible, menant à une suractivation de l'angiotensine 2. La Spike persistante pourrait causer des pathologies dégénératives à long terme. **English Translation:** Anaïs Bloqué explains the impact of the SARS-CoV-2 Spike protein on the innate immune system, focusing on TLR 4. Spike alone does not fully activate TLR 4 to induce a complete antiviral response (type 1 interferons). The Spike-LPS (bacterial lipopolysaccharide) association is necessary. Activation of interferon 1 increases ACE2 expression, the virus receptor, via ISGs, sensitizing the body to infection. Vaccine mRNAs can also activate interferon 1 via MDA5. Spike, an amyloid protein, can induce the production of A beta 42 fibers, worsening inflammation. Increased NF-κB by ISGs can block p53 (tumor suppressor) and induce microRNA MIR-200c, decreasing ACE2 expression. In people with comorbidities (diabetes, obesity), a Spike-LPS-TLR4 inflammatory amplification loop reduces available ACE2, leading to overactivation of angiotensin 2. Persistent Spike could cause long-term degenerative pathologies.

The speaker explains that the SARS CoV-2 virus, which causes COVID-19, has been successfully isolated and observed under a microscope. They show how the virus affects the lungs, causing scar tissue and disrupting blood flow. The virus enters the body through the ACE2 receptors and triggers an inflammatory pathway, leading to complications. The speaker highlights that the virus affects blood vessels throughout the body, including the brain, heart, lungs, kidneys, and GI tract. This widespread damage occurs due to the activation of the AT1R pathway, which triggers various inflammatory cells and molecules. Overall, COVID-19 damages multiple organs by instigating an internal battle within the blood vessels.

The speaker reflects on the pandemic, describing it as a time of “miracles,” including not losing anyone and continuing to speak today, despite controversy surrounding her research. She emphasizes that the controversy has hindered the advancement of research and science, urging instead to ask questions as science is about questioning and pushing narratives. She asserts a specific finding: the spike protein reduces bifidobacteria. She explains that the vaccine caused bifidobacteria to die within a month, but the effect persisted, with data indicating zero bifidobacteria in long-COVID or vaccine-injured cases. She notes she has been dealing with this for five years and asserts that people with zero bifidobacteria experience ongoing loss of microbiome diversity and immunity, resulting in poor immunity. She highlights bifidobacteria’s role in absorbing sugar, and adds that another microbe responsible for calcium absorption is also destroyed, leading to impaired calcium uptake. From these observations, she links cellular-level consequences to mitochondrial function, describing how a lack of sugar and calcium results in energy shortfalls and a disrupted Krebs cycle, implying mitochondrial dysfunction. She concludes that long COVID is a spike protein injury and that in many cases these individuals have zero bifidobacteria whether due to the treatment, the virus, or the spike protein itself. She also notes that some patients still have residual COVID in their stools, underscoring the need to pay attention to this finding. Key points emphasized: - The pandemic featured perceived miracles and ongoing controversy around research and vaccines, which the speaker argues stifles scientific progress. - A claim that the spike protein reduces bifidobacteria; the vaccine allegedly kills bifidobacteria within a month, with long-COVID or vaccine-injured individuals showing zero bifidobacteria across the line. - Zero bifidobacteria is linked to loss of microbial diversity, compromised immunity, and poor immune function. - Bifidobacteria’s role in absorbing sugar and a related microbe’s role in calcium absorption are highlighted as critical, with their destruction affecting cellular energy and mitochondrial function. - Long COVID is described as a spike protein injury, with some cases having residual COVID in stools, suggesting the need for attention to these microbial findings.

The spike protein, according to research in South Africa, induces fibrin from fibrinogen, forming the backbone of clotting in a way not previously seen. Unlike normal fibrin clots that are easily broken down, clots formed from COVID or the spike protein from the vaccine are difficult to break down, causing issues for many people. A cardiologist stated that in their decades of practice, they have never treated as many blood clots as in the last five years. These blood clots occur after the virus infection and the vaccine because the spike protein causes blood clots. Therefore, it is reckless to continue vaccinating people and loading the body with spike protein, causing more blood clots. According to a paper in Cell (July 2021), the nucleoprotein, not the spike protein, supplied broad and durable immunity for the prevention of infection. The speaker questions why the vaccine wasn't changed to target the nucleoprotein once this information came to light.

The speaker discusses the inflammatory and amyloidogenic effects of small sequences called epitopes, which can cause memory dysfunction in mice. They also mention a study that found the introduction of gene transfection technologies containing the spike protein can induce amyloidogenic cascades. The speaker highlights a 200% increase in the diagnosis of CJD in France after the rollout of vaccination programs, suggesting a potential link. They discuss the loss of cognitive function associated with exposure to the spike protein and propose that amyloidogenic disease processes may underlie long-haul COVID-19 symptoms. The speaker mentions the role of viral infections in facilitating intercellular aggregate dissemination and shares examples of misfolding prion amyloidogenic diseases.

I'm Philippe Boucalt, a cancer genomics researcher at the University of South Carolina. I've sequenced the DNA in the Pfizer vaccine and found that it contains fragments of DNA. This DNA could potentially cause rare but serious side effects, such as cardiac arrest and future cancer risks. The regulatory process that allowed this contamination is concerning. The DNA could integrate into long-lived somatic cells and potentially cause autoimmune attacks or disrupt tumor suppressors. To produce the vaccine, they cloned the PCR product into a plasmid vector, which led to the contamination. We can easily measure the amount of this substance in the vaccine and should conduct further studies to understand its implications. The FDA should require Pfizer to remove the DNA from the vaccine.

The speaker, a viral immunologist, discusses the presence of bacterial plasma DNA in the Pfizer and Moderna COVID-19 vaccines. They explain that the DNA is not supposed to be there and that its presence indicates improper manufacturing. The speaker highlights the potential dangers of bacterial DNA, including its ability to activate the immune system and promote inflammation. They also suggest that the DNA could lead to prolonged expression of the spike protein and raise concerns about legal immunity for the manufacturers. The speaker calls for a worldwide moratorium on the technology until further research is conducted.

The ACE2 receptor is well-known and plays a crucial role in our bodies. The left side of the chart shows cells lining our blood vessels, which have ACE2 receptors. On the right side, the spike protein from the vaccine affects the mitochondria, the cell's energy source. The spike protein causes fragmentation and damage to the mitochondria. This highlights the contrast between the smooth, intact cells on the left and the disrupted cells on the right, which is a result of the vaccine.

The speaker explains that the spike protein on the coronavirus is crucial for its structure and interaction with our cells. To trigger a protective antibody response, Keith replicates the spike protein in the lab and locks it into the same shape using a clamp-like protein. Surprisingly, this clamp-like protein is a small fragment of HIV.

The video discusses the potential adverse effects of the spike protein in SARS-CoV-2 on human prion protein and amyloid fibril formation. The speaker highlights a study showing that spike amyloid fibrils can accelerate the formation of amyloid fibrils associated with prion diseases and Alzheimer's. They also mention recent research suggesting that other viruses, like H5N1 influenza, may impact and misfold prion protein. The speaker emphasizes the importance of understanding these interactions and their potential implications. They briefly mention the symptoms and diagnostic challenges of prion diseases like Creutzfeldt-Jakob disease. Overall, the video explores the role of the spike protein in amyloidosis and its potential impact on neurological tissues.

The spike protein from the COVID-19 virus circulates in the body and can land in multiple organs, causing various diseases. Lab studies have shown that even without the virus, just injecting the spike protein can induce the same lung, vascular, heart, and brain diseases as COVID-19. The spike protein is considered the toxin responsible for causing the disease. This raises questions about why we are injecting something that is essentially a toxin into the human body, as it is not a traditional vaccine.

Dr. Yeason presents three principal assertions about the vaccines. 1) The first principle: we were told these molecules were gene sequences that encoded something called the spike protein. The spike protein is described as on the outside of the virus, and vaccines were said to encode the protein to train the immune system. Dr. Yeason explains that the immune system treats anything foreign as a threat and will attack cells that manufacture a foreign protein, leading to tissue damage. He notes that this principle of “self, non self” and tissue targeting is fundamental to organ transplantation and autoimmune diseases, and says this was designed into every company’s molecule (Moderna, Pfizer, Johnson & Johnson, AstraZeneca). He asserts that by 2020 he knew these were designed to cause injury. 2) The second principle: what was encoded is the spike protein. He states he did not know what spike protein was at first, but describes spikes on the outside of the virus and claims they are known toxins (neurotoxins, cardiotoxins) that prompt blood coagulation. He questions why a medicinal product would encode something that would harm the body when expressed. 3) The third principle: lipid nanoparticles (LNPs) used to formulate two of the Pfizer and Moderna products. He explains that lipid nanoparticles are toxic in general and are known to promote uptake of their payload into visceral organs, especially the liver and ovaries. He asserts that when injected into women and girls, these materials would travel through the body, concentrate in reproductive organs, be expressed, be recognized as foreign, and kill those cells. He asks what possible motivation there could be for using that formulation when other options exist. This, he says, confirms that the first two observations were not mere risks but intentional design. Dr. Yeason concludes that these three points together indicate that someone in a room designed injections to injure, kill, and reduce fertility in the people given them, aiming to lower fertility and reduce the population over time. He states he has observed this “all around me for five years since that moment.”

Bonjour, je suis Anaïs Bocquet, docteur en biologie santé, spécialisée en immunologie. Je vais vous parler du virus SARS-CoV-2 et de sa protéine Spike. Cette protéine est un GOF, clé d'entrée du virus dans l'organisme. Elle présente des propriétés persistantes, bactériophages et cancérogènes. Elle est également amyloïde, fusogène et impacte le système immunitaire. Les injections anti-Covid produisent une protéine Spike similaire à celle du virus, avec les mêmes propriétés. Cela peut causer des problèmes à long terme. Il est important de comprendre comment le virus et la protéine Spike interagissent avec d'autres pathogènes, comme les bactéries. Les questions scientifiques sont nombreuses et nécessitent l'intervention d'experts dans divers domaines. Merci de votre attention. Translation: Hello, I am Anaïs Bocquet, a doctor in health biology, specialized in immunology. I will talk to you about the SARS-CoV-2 virus and its Spike protein. This protein is a GOF, the key to the virus entering the body. It has persistent, bacteriophage, and carcinogenic properties. It is also amyloid, fusogenic, and impacts the immune system. Anti-Covid injections produce a Spike protein similar to that of the virus, with the same properties. This can cause long-term problems. It is important to understand how the virus and the Spike protein interact with other pathogens, such as bacteria. There are many scientific questions that require the intervention of experts in various fields. Thank you for your attention.

Bonjour à tous, Anaïs Bloqué, docteur en biologie santé, explique les impacts de la protéine Spike du SARS-CoV-2 sur le système immunitaire inné, basés sur son article récent. La Spike seule n'active pas complètement le TLR 4, un récepteur immunitaire, et ne produit pas d'interférons de type 1, essentiels pour la réponse antivirale. Pour une activation complète, la Spike doit s'associer au LPS (des bactéries Gram négatif). L'activation des interférons 1 augmente l'expression d'ACE2, le récepteur du virus, via les ISG, sensibilisant l'organisme à l'infection. Les ARN messagers des vaccins peuvent aussi lancer la production d'interférons 1 via MDA5. La Spike, protéine amyloïde, peut aussi déclencher le TLR 4 avec des fibres amyloïdes, entraînant un "double effet amyloïde". L'augmentation de NF-κB par les ISG peut bloquer la p53, potentiellement cancérigène. De plus, NF-κB induit le MIR-200c, qui bloque l'ACE2. Chez les individus avec comorbidités, une boucle d'amplification inflammatoire se crée : Spike-LPS-TLR4 induit interférons 1, ISG, surexpression d'ACE2, augmentation de NF-κB, MIR-200c, diminution d'ACE2 et augmentation d'angiotensine 2. La Spike persiste longtemps, et ses propriétés amyloïdes font craindre des pathologies dégénératives à long terme. --- Hello everyone, Anaïs Bloqué, PhD in health biology, explains the impacts of the SARS-CoV-2 Spike protein on the innate immune system, based on her recent article. Spike alone does not fully activate TLR 4, an immune receptor, and does not produce type 1 interferons, which are essential for the antiviral response. For complete activation, Spike must associate with LPS (from Gram-negative bacteria). Activation of interferon 1 increases the expression of ACE2, the virus's receptor, via ISGs, sensitizing the body to infection. Vaccine mRNAs can also trigger the production of interferon 1 via MDA5. Spike, an amyloid protein, can also trigger TLR 4 with amyloid fibers, leading to a "double amyloid effect." The increase in NF-κB by ISGs can block p53, which is potentially carcinogenic. In addition, NF-κB induces MIR-200c, which blocks ACE2. In individuals with comorbidities, an inflammatory amplification loop is created: Spike-LPS-TLR4 induces interferon 1, ISG, ACE2 overexpression, increased NF-κB, MIR-200c, decreased ACE2 and increased angiotensin 2. Spike persists for a long time, and its amyloid properties raise concerns about long-term degenerative pathologies.

Dr. Michael Palmer, an MD and professor of biochemistry, discusses the mechanisms by which COVID mRNA vaccines can cause toxicity. He identifies four major mechanisms: the spike protein triggering an immune response, toxic activity of the lipid nanoparticles used to deliver the mRNA, toxicity of the spike protein itself, and contamination of the vaccines with DNA. He explains how these mechanisms can lead to inflammation, blood clotting, and other adverse events. Dr. Palmer emphasizes that the immune attack on spike-expressing cells is the main driver of vaccine damage. He concludes that mRNA vaccines are a bad idea and urges support for scientist Sutra At Bakhti, who has faced persecution for speaking out against the vaccines.

The first vaccine decreases white blood cell production by 50%. The second dose, given eight weeks later, decreases saline while increasing harmful ingredients, further attacking white blood cell production by another 25%, leaving only 25% functionality. The booster contains 81 strands of foreign bacteria that the body can't fight effectively due to the reduced white blood cell production, leading to chronic inflammation in areas of predisposition, such as gut health, respiratory issues, or pre-existing conditions. This puts the body in a constant state of fight or flight with low immunity. The second booster contains eight strands of HIV, which shuts off the ability to make white blood cells, mirroring the effects of the disease itself. People are left with no immune system, foreign bacteria, HIV strains, and other harmful ingredients.