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A cardiologist provides an update on the Pfizer and Moderna vaccines, highlighting concerning findings. Studies on heart muscle cells from rats showed abnormal contractions and electrical activity within 48 hours of exposure to the vaccines. Messenger RNA from the vaccines was found in the human heart and circulating in the blood for up to 28 days. Circulating spike protein, produced by the messenger RNA, was detected in half of vaccinated individuals for up to 6 months. The spike protein is known to be harmful to cells and organs. The messenger RNA used in the vaccines has been modified and has numerous patents. Autopsy studies suggest that a significant number of deaths may be attributed to the vaccines. A case of a basketball player who suffered cardiac arrest after vaccination is highlighted as a cause for concern.

A recent study by Akahara and colleagues examined the effects of COVID-19 vaccines on the heart. They found that nearly every person who received the vaccine showed a preference for glucose over free fatty acids in their heart muscle cells. This metabolic dysfunction was evident in cardiac positron emission tomography scans, which lit up like a Christmas tree with fluorodeoxyglucose. Even those with a sore arm after vaccination showed more intense changes. In contrast, those who did not receive the vaccine had normal PET scans. These findings suggest that the vaccine may have an impact on heart function, although the long-term implications are still unknown.

First, the mRNA is injected within lipid nanoparticles in your arm. It travels through every organ system, including the heart. There are two papers, one by Baumeyer and colleagues, one by Crosson and colleagues. Crosson found mRNA directly in the heart of deceased mRNA recipients. So we know it reaches the heart. Baumeyer found the spike protein directly in the heart in biopsies of patients with vaccine induced myocarditis. So we know the vaccine mRNA and lipid nanoparticles get into the heart, translate into spike proteins, so your cardiomyocytes begin to produce a toxic non human protein and your own body attacks the heart resulting in inflammation and cardiac scarring including micro scars which are undetectable with imaging. You can only detect it with a microscope, which is very disturbing. And so once you have this scarring, you're going to have cardiac electrical abnormalities, electrical conduction abnormalities, and your heart's not going to beat properly. Then when you go exercise, we found there's two triggers either during exercise or sports when there's exertion or during the morning waking hours of sleep. In these two periods of time, there's a surge in catecholamines including dopamine, norepinephrine, and epinephrine. And so during these times when you have this cardiac damage, you have this scarring, that's the trigger you do that leads to this vaccine induced cardiac arrest. And that's why we saw a lot of sudden deaths among athletes back in 2021.

Myocarditis, or heart damage, is more common than previously thought. Studies in the US military and Thailand show that around 20% of people who receive the COVID vaccine develop myocarditis, as confirmed by echocardiograms and other tests. This means that out of every 1 million vaccinated individuals, 200,000 will experience heart damage. Unfortunately, 50% of those with myocarditis will die within 5 years. This alarming increase in myocarditis cases is due to the cardiotoxic nature of the vaccine. This information comes from Dr. Cressel and Shoemaker in Toronto, Canada.

Speaker 1 discusses the progression of understanding around heart damage associated with this product, emphasizing key studies and their implications. He recalls that in 2022 German scientists tested and reported that heart damage seen in myocarditis patients corresponds to vaccine-triggered autoimmune reactions. They examined endomyocardial biopsies and observed that the cardiac detection of the spike protein and CD4+ T cell–dominated inflammation suggested a vaccine-triggered autoimmune process. He notes headlines that infections could cause more myocarditis than vaccination and cites a large Israeli population study from that year finding no increase in myocarditis or pericarditis among unvaccinated individuals, challenging the notion that vaccination is the sole driver. Speaker 1 then highlights a new study published in the American Heart Association journal Circulation, framing it as a major development not from fringe sources but from a prestigious, mainstream journal. He asserts that this study goes beyond epidemiology by demonstrating a mechanism. In an experimental model, mice were used to induce myocarditis-like cardiac damage; researchers then compared these findings to humans who had similar heart damage post-vaccination. They found that T cells from patients with acute myopericarditis recognize vaccine-encoded spike epitopes that are homologous to cardiac self proteins, indicating cross-reactivity where the immune system targets both the spike protein and cardiac proteins. Speaker 1 explains that the study measured functional responses to potassium channels (KV) and observed an expanded pattern of cytokine production in patients with mild pericarditis after mRNA vaccination, but not in patients with COVID-19 without vaccination. This expanded cytokine response mirrored what was seen in AMP (myopericarditis) mice and in autoimmune myocarditis, linking the clinical data with the animal model. He paraphrases the study’s plain-language takeaway: post-mRNA vaccine myopericarditis is driven by molecular mimicry, with the immune system failing to distinguish self from non-self in susceptible patients. The study further notes that vaccine distribution contributes to susceptibility; the widespread distribution of the vaccine allows the heart to be targeted, leading to cardiac-selective autoimmunity by “heart-homing imprinting.” Speaker 1 emphasizes the significance of the source, stating that the journal is not fringe: it is the Circulation journal, ranked third in its field with a cardiovascular-focused impact in the 99th percentile. The overall conclusion presented is that these findings provide a clear mechanism for post-vaccination myopericarditis and establish a direct link between vaccine-encoded spike epitopes and cardiac autoimmunity.

The discussion centers on new evidence regarding myocarditis and pericarditis in the context of mRNA vaccination. It cites earlier 2022 German research showing that heart damage seen in myocarditis cases after vaccination may be a vaccine-triggered autoimmune reaction. The study analyzed endomyocardial biopsy samples and found that the cardiac tissue’s spike protein detection and CD4+ T cell–dominated inflammation suggested an autoimmune mechanism linking the vaccine to heart damage. This was contrasted with an Israel-based population study of hundreds of thousands of unvaccinated individuals that reported no increase in myocarditis or pericarditis incidence, highlighting a discrepancy with the vaccine-triggered autoimmune hypothesis. The center of the new claim is a study published in Circulation by the American Heart Association. The speakers emphasize the credibility of Circulation as a top cardiovascular journal. The study used an experimental mouse model to induce cardiac damage and then examined humans with similar heart damage after vaccination to see if the same mechanism applied. They report that T cells from patients with acute myopericarditis recognize vaccine-encoded spike epitopes that are homologous to cardiac self-proteins. In other words, the immune cells targeting the spike protein may also attack cardiac proteins due to molecular similarity. Further details from the study indicate that, in patients with mild pericarditis after mRNA vaccination (but not in those with COVID-19), there was an expanded pattern of cytokine production similar to that observed in myopericarditis–affected mice and in autoimmune myocarditis. The takeaway provided in plain language is that post-mRNA vaccine myopericarditis is driven by molecular mimicry, causing the immune system to fail to distinguish self from non-self in susceptible patients. The susceptibility is described as being influenced by the widespread distribution of the vaccine, which purportedly leads to heart-homing imprinting and a heart-targeted autoimmune response. The speakers stress that this journal is not fringe and highlight its high impact in cardiovascular medicine. They conclude that the data collectively suggest a mechanism by which the vaccine could provoke cardiac autoimmunity, with implications for clinical communication and understanding of post-vaccination myocarditis.

A recent study from Switzerland found that 1 in 35 people who received the Moderna booster shot experienced heart injury. Researchers hypothesized that many cases went unnoticed due to lack of symptoms. They tested for heart injury by measuring levels of cardiac troponin t in the blood, which indicates heart damage. This study aimed to uncover the true prevalence of vaccine-associated heart injury post mRNA booster vaccination.

A recent study suggests a potential link between mRNA COVID-19 vaccines and sudden cardiac deaths. Researchers found that when Pfizer and Moderna vaccines were applied directly to heart muscle cells, abnormal function and electrical currents occurred within 48 hours, indicating cardiac toxicity. Another study showed that mRNA was physically stuck in the hearts of individuals who died after vaccination. Additionally, a large study found abnormal heart scans in those who received the vaccine, with virtually everyone showing abnormal results. Even individuals with inflammation or higher mRNA doses had worse cardiac findings. This suggests the possibility of cardiac dysfunction or arrest without myocarditis. The information challenges the official narrative and raises concerns.

We are witnessing a significant increase in cases of myocarditis, with thousands reported in recent studies compared to only a few cases in the past. The potential long-term effects of vaccine-induced myocarditis are concerning, with some cases leading to cardiac arrests years after vaccination. This suggests that the current cases may just be the beginning, and regulatory concerns should extend for at least 5 to 15 years post-vaccination.

Myocarditis was most common in young men, with rates as high as 1 in 5000 vaccine recipients. The condition was mostly mild but could have lasting effects. Natural immunity from prior COVID infection was shown to be more protective than two vaccine doses. Combining prior infection with vaccination provided even better protection. The speaker did not take a booster shot.

The discussion centers on evidence linking myocarditis and pericarditis to mRNA vaccination and the proposed mechanism behind it. It references a 2022 German study reporting that endomyocardial biopsy data from people with myocarditis showed cardiac detection of the spike protein and CD4+ T cell–dominated inflammation, suggesting a vaccine-triggered autoimmune reaction. The presenters note headlines at the time comparing myocarditis risk to infection, with claims that infection causes more myocarditis, and remind that vaccines were said not to stop transmission. They then cite a large Israeli population study from the same year involving subjects not vaccinated against SARS-CoV-2, which found no increase in the incidence of myocarditis or pericarditis, implying no observed vaccine-related signal in that cohort. Attention shifts to a more recent study published in Circulation by the American Heart Association, described as a high-impact, non-fringe journal, indicating a clearer mechanism has been demonstrated. The study described used an experimental mouse model to induce cardiac damage and then compared it to human cases with heart damage following vaccination. It states that T cells from patients with acute myocarditis or myopericarditis recognize vaccine-encoded spike epitopes that are homologous to cardiac self proteins, meaning the immune response to the spike protein can cross-react with heart tissues. The researchers further report that functional responses to potassium channels in patients with mild pericarditis after mRNA vaccination, but not in patients with COVID-19, showed an expanded pattern of cytokine production similar to that observed in myopericarditis mice and in autoimmune myocarditis. In plain terms, the summary of their takeaway is that post-mRNA vaccine myopericarditis is driven by molecular mimicry: the immune system cannot distinguish self from non-self, leading to an autoimmune attack on heart tissue in susceptible patients. The distribution of the vaccine (its widespread dissemination) is cited as a factor that makes patients susceptible by promoting heart-homing imprinting, effectively creating an anti-heart autoimmune response. The speakers emphasize that this Circulation article is a top-tier source, underscoring that the mechanism has been demonstrated with both animal models and human pathology, supporting the claim that the phenomenon has a defined immunological basis.

The speaker discusses a significant increase in myocarditis cases post-vaccination, with studies showing abnormal cardiac scans in vaccinated individuals. They suggest a potential link between mRNA vaccines and heart inflammation, emphasizing the need for long-term monitoring. Research indicates that mRNA and spike proteins can cause myocarditis, posing a concern for all mRNA products. The heart appears to be a vulnerable target due to various factors.

Before COVID-19, I only encountered two cases of myocarditis in my entire career as a cardiologist. It was a rare condition, usually caused by parvovirus or adenovirus. However, now I see two cases per day in the clinic. We have learned that COVID-19 can cause myocarditis. Various organizations, such as the Israeli and US military, as well as college leagues, conducted screening programs in 2020 and found a few cases, but none were serious or resulted in hospitalizations or deaths. These programs were later discontinued when vaccines were introduced. However, within six months, regulatory agencies confirmed that the COVID-19 vaccines can cause myocarditis, and it can be fatal. It's important for people to understand the risks associated with each vaccine dose they take.

The speaker explains that myocarditis from natural infection is usually seen in ICU patients and is mild, with only troponin elevation. However, vaccine-induced myocarditis is different and more severe. Preclinical studies suggest that the vaccine's lipid nanoparticles directly affect the heart, causing the body to attack it. This leads to dramatic EKG changes and significantly higher troponin levels compared to natural infection. When children experience myocarditis after vaccination, 90% require hospitalization and show symptoms like chest pain and early heart failure. They may need echocardiograms and medications to prevent heart failure. Vaccine-induced myocarditis is a significant concern, especially in children.

A cardiologist provides an update on the Pfizer and Moderna vaccines. Studies have shown that the vaccines can cause direct harm to heart muscle cells, abnormal heart contractions, and abnormal electrical activity. Messenger RNA from the vaccines has been found in the human heart and circulating in the blood for up to 28 days. The spike protein produced by the messenger RNA has also been detected in the blood for up to 6 months. The spike protein is dangerous to cells, tissues, and organs in the body. The messenger RNA used in the vaccines has been modified and is synthetic. Autopsy studies have shown that the vaccine can cause myocarditis and cardiac damage. A basketball player who received the vaccine suffered a cardiac arrest and died two years later.

A recent study from Switzerland found that 1 in 35 individuals who received the Moderna booster shot experienced vaccine-associated heart injury. The researchers hypothesized that this type of injury was more common than previously believed due to the lack of symptoms or mild symptoms. To investigate this, the researchers tested the blood of participants instead of relying solely on self-reported symptoms. They defined heart injury as a significant increase in high sensitivity cardiac troponin levels on the third day after vaccination, without any other apparent cause. The use of the troponin test was chosen because it is a reliable indicator of heart damage.

Before COVID-19, I only encountered two cases of myocarditis in my entire career as a cardiologist. It was a rare condition, usually caused by parvovirus or adenovirus. However, now I see two cases per day in the clinic. We have learned that COVID-19 can cause myocarditis. Various organizations, such as the Israeli and US military, as well as college leagues, conducted extensive screening programs for COVID-induced myocarditis in 2020. They found a few cases that met the definition, but none were serious or resulted in hospitalizations or deaths. These screening programs were later discontinued when vaccines were introduced. However, within six months, regulatory agencies confirmed that the COVID-19 vaccines can cause myocarditis. It is important for people to understand that there is a risk of vaccine-induced myocarditis with every shot they take.

Myocarditis, or heart inflammation, is more common than previously believed. Recent studies show that around 20% of individuals who received the COVID vaccine experience myocarditis, as confirmed by echocardiograms and other tests. This means that out of every 1 million vaccinated people, around 200,000 will have evidence of heart damage. Unfortunately, those who develop myocarditis have a 50% chance of surviving only 5 years. This alarming increase in myocarditis cases is due to the cardiotoxic nature of the vaccine. These facts, shared by Dr. Cussell and Shoemaker from Toronto, highlight the concerning impact of the vaccine on heart health.

Before COVID-19, I only encountered two cases of myocarditis in my entire career as a cardiologist. However, now I see two cases per day in the clinic. We have learned that COVID-19 can cause myocarditis, and various organizations conducted screening programs in 2020. These programs found a few cases that met the definition of myocarditis, but none were serious or resulted in hospitalizations or deaths. After the introduction of vaccines, regulatory agencies acknowledged that the vaccines can cause COVID-19 vaccine-induced myocarditis, which can be fatal. It's important for people to understand that there is a risk associated with every vaccine shot they take.

As a cardiologist, the speaker states their role is to fight disease, preserve life, and do no harm. The topic is myocarditis or heart damage from the COVID-19 vaccines. The speaker claims to have examined thousands of patients with this problem, whereas before the pandemic, they state they only had two patients ever with this condition. The speaker references a New England Journal of Medicine paper from Washington University in St. Louis, August 18, 2021, where a 42-year-old man died three days after taking Moderna. They also cite a case from Korea by Choi and colleagues, where a younger man died within eight hours of being in the hospital after Pfizer. The speaker examined images from the Korean case and states the heart appeared "fried with inflammation" and "destroyed." The speaker concludes these cases should have gotten everyone's attention.

Doctors can be fooled and pressured, and many are afraid to speak out against the government's COVID measures. The speaker mentions truckers who were allowed to work without vaccines or passports, but were criticized for questioning the safety of the vaccine. They discuss a study by Nakahara that found vaccinated individuals had hearts that worked 46% harder for 6-7 months after receiving the vaccine, while non-vaccinated individuals showed no increase in heart effort. The speaker concludes that this study proves the vaccine is harmful to the heart and criticizes the idea of giving booster shots every 6 months, which further strains the heart.

There is concerning evidence of cardiac arrests in people who have received the vaccine. A study by Nakahara found that positron emission tomography scans showed changes in heart metabolism in almost everyone who took the shot for at least 6 months. This is worrisome, as we don't fully understand the implications. Harvard researchers discovered messenger RNA stuck in the hearts of deceased individuals. Additionally, Schreckener from Germany revealed that Pfizer and Moderna vaccines may be directly toxic to heart muscle cells. Based on this information, there is a strong suggestion that these vaccines should be removed from the market.

Vaccination introduces mRNA into the bloodstream, which is taken up by major organs, including the heart. This process leads to the production of spike protein in heart muscle cells, resulting in inflammation and an increased risk of myocarditis. A large study indicated a 500% higher risk of myocarditis following COVID vaccination. Symptoms of myocarditis can be triggered during early morning hours (3 AM to 6 AM) when catecholamines like dopamine and epinephrine surge, as well as during exercise. These triggers can lead to serious heart issues, including ventricular tachycardia and sudden death.

The speaker discusses the increasing awareness and concern surrounding myocarditis as a result of COVID-19 vaccines. They mention that there are now 800 peer-reviewed papers on COVID vaccine-induced myocarditis, with a rate of heart damage at 2.5% in two studies. They explain the pathogenesis of vaccine-induced cardiac arrest and highlight the fatality of this condition. The speaker also mentions cases of athletes and public figures who have experienced myocarditis after vaccination. They express concern about the lingering effects of myocarditis and the recurrence of symptoms. The speaker concludes by discussing the case of a European athlete who experienced a cardiac arrest two years after vaccination, emphasizing the ongoing risk associated with myocarditis.

In October 2020, the FDA mentioned that myocarditis could be a result of the COVID vaccines. In June 2021, the FDA confirmed that the vaccines can cause heart inflammation. Prior to COVID, patients with myocarditis were advised not to exercise due to the risk of cardiac arrest. Now, there are 800 peer-reviewed papers on COVID vaccine-induced myocarditis. Two studies showed a 2.5% rate of heart damage after receiving the second or third vaccine dose. When heart damage occurs, there can be variations in electrical conduction, leading to reentry and fast heart rhythms like ventricular tachycardia. This can progress to ventricular fibrillation, which is fatal. A recent study confirmed that vaccine-induced myocarditis is always fatal.