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Both COVID-19 and exosomes have similar sizes and use the ACE2 receptor to invade cells. ACE2 is an enzyme that regulates blood pressure. Both COVID-19 and exosomes contain RNA, not DNA, and are found in lung fluid. The comparison between the two shows that they are essentially the same in important ways. Some researchers in virology even suggest that exosomes and viruses could be the same thing. Dr. James Hildreth, a prominent virology researcher and physician, supports this idea.

Bonjour à tous, Anaïs Bloqué, docteur en biologie santé, explique les impacts de la protéine Spike du SARS-CoV-2 sur le système immunitaire inné, basés sur son article récent. La Spike seule n'active pas complètement le TLR 4, un récepteur immunitaire, et ne permet pas la production d'interférons de type 1, essentiels pour lutter contre le virus. Pour une activation complète, la Spike doit s'associer au LPS (des bactéries gram négatifs). L'activation des interférons 1 augmente l'expression d'ACE2, le récepteur du virus, sensibilisant l'organisme à l'infection. Les interférons 1 peuvent aussi être activés par les RLR, notamment MDA5, qui détecte l'ARN messager modifié des vaccins anti-COVID. De plus, la Spike, protéine amyloïde, peut déclencher le TLR 4 en s'associant aux fibres amyloïdes A bêta 42, créant un "double effet amyloïde". L'augmentation de NF-κB par les interférons 1 peut bloquer la p53, un suppresseur de tumeur, et induire l'expression du MIR-29b, qui bloque ACE2. Chez les personnes avec comorbidités, ayant déjà de faibles niveaux d'ACE2, cette interaction Spike-LPS devient dangereuse, créant une boucle d'amplification inflammatoire. La Spike persiste longtemps dans l'organisme, et avec ses propriétés amyloïdes, pourrait entraîner des pathologies dégénératives à long terme. --- Hello everyone, Anaïs Bloqué, Doctor of Philosophy in Health Biology, explains the impacts of the SARS-CoV-2 Spike protein on the innate immune system, based on her recent article. The Spike alone does not fully activate TLR 4, an immune receptor, and does not allow the production of type 1 interferons, which are essential for fighting the virus. For complete activation, the Spike must associate with LPS (from gram-negative bacteria). Activation of interferon 1 increases the expression of ACE2, the virus's receptor, sensitizing the body to infection. Interferons 1 can also be activated by RLRs, in particular MDA5, which detects the modified messenger RNA of anti-COVID vaccines. In addition, Spike, an amyloid protein, can trigger TLR 4 by associating with amyloid A beta 42 fibers, creating a "double amyloid effect". The increase in NF-κB by interferons 1 can block p53, a tumor suppressor, and induce the expression of MIR-29b, which blocks ACE2. In people with comorbidities, who already have low levels of ACE2, this Spike-LPS interaction becomes dangerous, creating an inflammatory amplification loop. Spike persists in the body for a long time, and with its amyloid properties, could lead to long-term degenerative pathologies.

Chronic diseases like Alzheimer's, Parkinson's, cardiovascular diseases, cancers, diabetes, obesity, and autoimmune diseases are linked to inflammation. Glutathione, a powerful antioxidant, can detoxify heavy metals, reduce inflammation by targeting the main inflammatory switch in the body called nuclear factor kappa beta. This switch is challenging to turn off once activated.

The speaker describes a nationwide study conducted in South Korea, stating that every resident was included in the research. The study compared individuals who received the vaccine to those who did not, and the analysis was stratified by dose number (one dose, two doses, three doses, and four or more doses). A central claim of the speaker is that this study provides the strongest signal to date supporting vaccine acquired immunodeficiency syndrome, referred to as VADES. According to the speaker, as each dose was administered, the immune function of individuals declined. By the time of the fourth dose, the speaker asserts there was a significant increase in the risk of other infections, quantified as about a 550% increase, including infections such as the common cold, tuberculosis, and upper respiratory tract infections. The speaker notes that the effect was most pronounced in young people, specifically ages zero to nineteen, who reportedly had the highest risks of these other infections. The implication presented is that the injections are causing immune collapse and exhausting T cells, leading to immune dysregulation described as IgG4 class switching. The immune system is said to become dysfunctional as a result. Additionally, the speaker mentions that, consistent with other studies they reference, genes related to immune function are claimed to become shut down. The overall assertion is that these findings point to a troubling pattern of immune impairment associated with multiple vaccine doses, culminating in the claimed immune dysfunction and increased susceptibility to other infections. The speaker emphasizes the magnitude and reliability of the sample size, stating that having an entire country’s population as the study cohort constitutes the strongest possible sample size. The summary of the presented claims centers on dose-dependent immune decline, a marked increase in non-target infections after the fourth dose, greater impact on children, evidence of immune system exhaustion and dysregulation, and purported genetic downregulation of immune pathways, all described as arising from the vaccination regimen in this nationwide South Korean study.

The speaker claims that chemotherapy first wipes out red blood cells, causing anemia, with Epogen; second, neutrophils that prevent infection, with Neupogen; and most importantly, NK and T cells—the lymphocytes. He says, "the only thing that protects your body against cancer is your lymphocytes, meaning the NK cells and T cells," and that, with chemotherapy or radiation, "within a day or two, you wipe out the only cells that matter, i.e. The cells that kill can." He notes, "for thirty five years, we've never had a treatment for that." He links this to long COVID: during viral infection, "the virus is smart, it wipes out the T cells and NK cells." He points out the irony that Epogen and Neupogen are needed because chemotherapy has wiped out the cells that matter. A South African pancreas transplant surgeon says, "everything we've done so far has been wrong. We've actually treated cancer wrongly."

Professor David Moranz discusses the concept of viral-bacterial co-pathogenesis and its relevance to sepsis, septic shock, and death from bacterial disease, using early 20th-century pandemics as a lens. Key points: - The idea that sepsis, septic shock, and death can be downstream events prompts examination of upstream events and progression to prevent outcomes. - Co-pathogenesis, involving simultaneous infection with a virus and a bacterium, was recognized in the early 1900s. Ellis Island health officers noted higher mortality in children with viral infections like measles when co-infected with bacteria such as streptococci or diphtheria. - During World War I, crowded army camps experienced massive measles outbreaks and secondary bacterial pneumonia deaths. A notable study tracked soldiers with measles: those who were colonized with group A beta-hemolytic streptococcus (Strep pyogenes) had all complications and deaths among colonized individuals, while non-colonized soldiers fared better. - With influenza in 1918, pathology from the Armed Forces Institute of Pathology showed that death was associated with severe bacterial pneumonia in all 58 autopsies studied. Across 173 autopsy studies from 15 countries (over 8,000 individuals), 95% had pneumopathogens cultured from the lungs; 80.4% of pleural effusions contained pneumopathogens; 70% of those with pre-death blood cultures had one or more positive cultures. - The principal pneumopathogens identified were Streptococcus pneumoniae (pneumococcus), Streptococcus pyogenes (Group A beta-hemolytic strep), and Staphylococcus, though other pathogens occurred as well, including meningococcus in some outbreaks of fatal influenza-associated pneumonia. - Pathology commonly showed bronchopneumonia, with viral lesions characterized by infection of apical cells of the bronchiolar and bronchial epithelium. The virus disrupts the protective epithelial layer, enabling bacteria to colonize the basal layer and cause pneumonia. This is why bronchopneumonia was prevalent in both measles- and influenza-associated deaths. - The proximate cause of death often involved hypoxia from damaged pulmonary tissue or alveolar edema; sepsis and multi-organ failure were also cited in some cases, alongside heart or renal failure in others. - Notable interpretation by contemporaries: French physician quote that influenza condemns secondary infections; William McCallum remarked he never autopsied a flu death without finding bacteria. He viewed these events as epidemics of severe bacterial pneumonia precipitated by the two viruses. - In modern research, experimental models (mostly mice, sometimes primates) show that adding influenza (or similar viruses) to a bacterial infection like pneumococcus results in markedly worse pathology and faster death, illustrating the continuing relevance of viral-bacterial co-pathogenesis. The talk links historical observations to current inquiry, describing how colonization by pneumopathogens in crowded settings, followed by a cytolytic viral infection that damages the respiratory epithelium, creates conditions for severe bacterial pneumonia and respiratory compromise.

Vitamin D can enter a cell's nucleus where DNA is stored. It recognizes DNA sequences, binds to a receptor, and activates or deactivates genes in a coordinated manner. These genes are important for brain function, such as serotonin synthesis, and immune function. This is why vitamin D plays a critical role in helping prevent respiratory diseases.

When mRNA vaccines are injected, the amount of protein produced is not directly controlled by the dose of RNA given. Factors like metabolism and cell activity influence protein output. Doses vary between Moderna and Pfizer vaccines, but the key is the spike protein produced in the body. Studies show some individuals may not produce enough spike protein for immunity, while others may have excess leading to side effects or toxicity. In vitro experiments may not accurately predict in vivo outcomes due to individual differences.

Mitochondria, you know, the energy producers, the powerhouses within every cell. Brain cells may have as many as a thousand mitochondria in each neuron. Mitochondria are seen diffusely throughout the body in virtually all of our cells, interestingly, not in our red blood cells, but certainly in our white blood cells. And having good mitochondrial function and numbers within our white blood cells, is an important player as it relates to a proper effective immune function and keeping, inflammation in balance. They recognize the importance of dysfunction or problems with the mitochondria, as being a major risk factor in things like obesity, diabetes and hypertension.

We all carry dormant cancers and tumors to varying degrees. The innate immune system's destruction allows these dormant tumors to become active. Once the immune system is compromised, existing weak cancer cells can proliferate unchecked, similar to antibiotic-resistant bacteria. Each cancer cell is unique, and in a weakened immune environment, those that can survive will thrive rapidly. The immune system's complexity is immense, and its overall functionality is being diminished. Notably, IgG4 levels have surged significantly in those exposed to certain injections, which may suppress the immune response to cancer cells. While the details are complex, the general principles indicate that these changes could hinder the immune system's ability to combat cancer effectively.

A study involving immune profiling showed that fermented foods can decrease inflammatory markers. Measurements taken at multiple points revealed a stepwise reduction in inflammatory mediators like interleukin-6 and interleukin-12. Analysis of immune cell signaling cascades indicated less activation at the end of the study compared to the beginning. This suggests that fermented foods may lead to an attenuation of inflammation, potentially reducing the likelihood of inflammatory disease over time.

Cortisol, a hormone released during stress, suppresses the immune system, allowing pathogens and viruses to cause problems. Stress essentially puts the immune system to sleep, potentially causing viruses to emerge from remission, especially after major losses. In these situations, vitamins and good nutrition may be ineffective due to the overriding impact of stress.

In integrative medicine, proactive measures like intravenous vitamin C are utilized. Administering high doses, such as 20 to 50 grams, can significantly elevate blood levels and has a direct antiviral effect. The immune system produces hydrogen peroxide to combat pathogens, and high-dose vitamin C can enhance this response. It's commonly used by integrative doctors for both viral and bacterial infections, either alone or alongside other treatments. Additionally, intravenous vitamin C helps reduce inflammation, alleviating pain and symptoms associated with infections like the flu, which are often caused by inflammatory compounds released by the body.

**French Summary:** Anaïs Bloqué explique l'impact de la protéine Spike du SARS-CoV-2 sur le système immunitaire inné, en se concentrant sur le TLR 4. La Spike seule n'active pas complètement le TLR 4 pour induire une réponse antivirale complète (interférons de type 1). L'association Spike-LPS (lipopolysaccharide bactérien) est nécessaire. L'activation des interférons 1 augmente l'expression d'ACE2, récepteur du virus, via les ISG, sensibilisant l'organisme à l'infection. Les ARN messagers des vaccins peuvent aussi activer les interférons 1 via MDA5. La Spike, protéine amyloïde, peut induire la production de fibres A bêta 42, aggravant l'inflammation. L'augmentation de NF-κB par les ISG peut bloquer p53 (suppresseur de tumeur) et induire le micro-ARN MIR-200c, diminuant l'expression d'ACE2. Chez les personnes avec comorbidités (diabète, obésité), une boucle d'amplification inflammatoire Spike-LPS-TLR4 réduit l'ACE2 disponible, menant à une suractivation de l'angiotensine 2. La Spike persistante pourrait causer des pathologies dégénératives à long terme. **English Translation:** Anaïs Bloqué explains the impact of the SARS-CoV-2 Spike protein on the innate immune system, focusing on TLR 4. Spike alone does not fully activate TLR 4 to induce a complete antiviral response (type 1 interferons). The Spike-LPS (bacterial lipopolysaccharide) association is necessary. Activation of interferon 1 increases ACE2 expression, the virus receptor, via ISGs, sensitizing the body to infection. Vaccine mRNAs can also activate interferon 1 via MDA5. Spike, an amyloid protein, can induce the production of A beta 42 fibers, worsening inflammation. Increased NF-κB by ISGs can block p53 (tumor suppressor) and induce microRNA MIR-200c, decreasing ACE2 expression. In people with comorbidities (diabetes, obesity), a Spike-LPS-TLR4 inflammatory amplification loop reduces available ACE2, leading to overactivation of angiotensin 2. Persistent Spike could cause long-term degenerative pathologies.

Arrhythmias are common in COVID-19, and there are auto antibodies in this pathway that may be causing them. Additionally, there is a connection between t1r receptors and depression and dementia, which can lead to brain fog and fatigue. The challenge is to find something in COVID-19 that cannot be explained using these four substances.

The comparison to HIV is important because both viruses can be asymptomatic. HIV taught us a lot about immunology and changed cancer therapy. Similarly, we are now learning about the impact of the virus on mitochondria, brain fog, and our neurons through long COVID. Mild and moderate COVID can cause destruction, just like HIV did to our immune system. However, the brilliant science that came out of HIV research transformed how we treat the virus, allowing people to live normal lives. We need to do the same for long COVID, so that those affected can not only survive but also thrive.

The spike and pseudouridine in the shots are changing receptor patterns on cells, suppressing the immune system's ability to fight off viruses like herpes, HPV, and RSV. Toll-like receptors, which train cells to fight cancer, are also being suppressed. These receptors are like the marines of our immune system, constantly circulating and identifying friend or foe. However, the shots are causing these marines, along with dendritic cells and macrophages, to become inactive, leaving the body defenseless against cancer cells and pathogens. It is unclear when this suppression stops or how to reverse it. While not affecting everyone, the degree to which it is happening is concerning.

Vitamin C boosts the immune system by increasing white blood cell mobility and activity; without it, they become sluggish. Vitamin C also enhances phagocytosis, which is the ability of phagocytes to consume pathogens. Additionally, vitamin C aids white blood cells in producing defenses against infections. A lack of vitamin C can prolong the duration of illness. White blood cells contain 100 times more vitamin C than blood because the immune system relies heavily on it.

Bonjour à tous, Anaïs Bloqué, docteur en biologie santé, explique les impacts de la protéine Spike du SARS-CoV-2 sur le système immunitaire inné, basés sur son article récent. La Spike seule n'active pas complètement le TLR 4, un récepteur immunitaire, et ne produit pas d'interférons de type 1, essentiels pour la réponse antivirale. Pour une activation complète, la Spike doit s'associer au LPS (des bactéries Gram négatif). L'activation des interférons 1 augmente l'expression d'ACE2, le récepteur du virus, via les ISG, sensibilisant l'organisme à l'infection. Les ARN messagers des vaccins peuvent aussi lancer la production d'interférons 1 via MDA5. La Spike, protéine amyloïde, peut aussi déclencher le TLR 4 avec des fibres amyloïdes, entraînant un "double effet amyloïde". L'augmentation de NF-κB par les ISG peut bloquer la p53, potentiellement cancérigène. De plus, NF-κB induit le MIR-200c, qui bloque l'ACE2. Chez les individus avec comorbidités, une boucle d'amplification inflammatoire se crée : Spike-LPS-TLR4 induit interférons 1, ISG, surexpression d'ACE2, augmentation de NF-κB, MIR-200c, diminution d'ACE2 et augmentation d'angiotensine 2. La Spike persiste longtemps, et ses propriétés amyloïdes font craindre des pathologies dégénératives à long terme. --- Hello everyone, Anaïs Bloqué, PhD in health biology, explains the impacts of the SARS-CoV-2 Spike protein on the innate immune system, based on her recent article. Spike alone does not fully activate TLR 4, an immune receptor, and does not produce type 1 interferons, which are essential for the antiviral response. For complete activation, Spike must associate with LPS (from Gram-negative bacteria). Activation of interferon 1 increases the expression of ACE2, the virus's receptor, via ISGs, sensitizing the body to infection. Vaccine mRNAs can also trigger the production of interferon 1 via MDA5. Spike, an amyloid protein, can also trigger TLR 4 with amyloid fibers, leading to a "double amyloid effect." The increase in NF-κB by ISGs can block p53, which is potentially carcinogenic. In addition, NF-κB induces MIR-200c, which blocks ACE2. In individuals with comorbidities, an inflammatory amplification loop is created: Spike-LPS-TLR4 induces interferon 1, ISG, ACE2 overexpression, increased NF-κB, MIR-200c, decreased ACE2 and increased angiotensin 2. Spike persists for a long time, and its amyloid properties raise concerns about long-term degenerative pathologies.

Chronic inappropriate inflammation can stem from autoimmune conditions or exposure to toxins/chemicals in diet or environment. This overworks and confuses the immune system, raising the risk of autoimmune conditions where the immune system attacks the body. Chronic inflammation not only causes pain, stiffness, and disability, but can also lead to new diseases. Chronic inflammation is detrimental, so it's important to identify and address the root cause.

The first vaccine decreases white blood cell production by 50%. The second dose, given eight weeks later, decreases saline while increasing harmful ingredients, further attacking white blood cell production by another 25%, leaving only 25% functionality. The booster contains 81 strands of foreign bacteria that the body can't fight effectively due to the reduced white blood cell production, leading to chronic inflammation in areas of predisposition, such as gut health, respiratory issues, or pre-existing conditions. This puts the body in a constant state of fight or flight with low immunity. The second booster contains eight strands of HIV, which shuts off the ability to make white blood cells, mirroring the effects of the disease itself. People are left with no immune system, foreign bacteria, HIV strains, and other harmful ingredients.