reSee.it Video Transcript AI Summary
Speaker: A lot of the analysis techniques that were being employed now on the white clots. And about three years ago, there was one piece of analyses that I came across just by accident, which was a thing called an ICP analysis, which is an analysis that determines the elements present in white clots. It was done by a gentleman in The USA called Mike Adams, who presented the findings of his initial white clot analysis from samples that Richard had provided him some almost three years ago now. And it surprised me what the findings were, because I have used the ICP analysis. It’s called Inductively Coupled Plasma Mass Spectrometry.
Don't worry about the name. It's just a very well known piece of analytical equipment that you can rely upon to tell you what elements are there in the white clot and in what abundance. And what surprised me was in the analysis that Mike presented was a very the highest element they found was phosphorus, followed by sodium, tin, and later on sulphur, and carbon. And we also found that there were no normal blood marker elements present in the white clots. So, we've actually employed two laboratories in Europe, and there's a long reason behind that because I'm associated with people in Europe who do this kind of work.
Anyway, we sent clot samples, they all ran ICP analyses. They found exactly the same results that Mike Adams found. We found aberrantly high levels of phosphorus, we found aberrantly high levels of sulphur, tin, sodium, and carbon. And being an organic chemist, I know a little bit about tin chemistry, because I used to sell tin catalysts, believe it or not, for polymerisation reactions. So I had to study the chemistry of tin.
And I wondered why tin would be present in a white clot. So, obviously, we could not believe this first series of results. It's set about replicating the results in two separate labs, and they came back and said, you're right, there's high levels of phosphorus, tin, sulfon, carbon. So the next thing we did was to do an analysis called HPLC, High Performance Liquid Chromatography. And what this does is actually pulls apart the white clots and tells you what the protein components are.
And surprisingly, we found that the highest level of proteins we found was fibrinogen. But the HPLC analysis actually will determine what kinds of fibrinogen chains are there. In a normal red blood clot, there's normally one to one to one ratio of the alpha chain, beta chain and gamma chain. We found in our white clots that the beta chain far exceeded the alpha chain and the gamma chain. And in fact, it was beta gamma alpha.
So the alpha was the lowest proportion. Now, we're not biochemists, and we're not medically qualified in any way. But we can do simple research to find out why the fibrinogen content was so high. Fibrinogen forms fibrils. This is exactly why when John O'Learny first described the white clots as being calamari like, he is exactly correct.
That's exactly the texture that fibrin will bring into a white clot. Okay, so from then on, we then ran some amino analysis results. My colleagues ran an amino acid analysis, and they found a high level of Praline, Aspartic acid, Lysine, a whole range of about 18 amino acids, all that have a phosphorus affinity. And as we said in the white clot, the element that has the highest concentration that we could confirm is porpoise. If you take time then to research the aberrant pathways that what they call excess phosphorylation will cause, it causes a whole range of problems in the human body.
So we have determined, we think, the pathway to the formation of these white clots. We have three separate pieces of analysis, all confirming our findings. So we've now pieced together the formation of the white clots, and I'll come back to the very beginning. When we administer the injections, there is a lipid nanoparticle carrier, it's called a phospholipid. We found by our analysis that when the phospholipid releases the mRNA core of the lipid nanoparticle, at the very moment that it releases the core, it actually exposes a phosphorus head of the phosphorus lipid.
The phosphor lipid reacts within the bloodstream naturally formed fibrinogen, and that's what's nucleating the white clot formation. Now we can prove all this. We've actually got over 200 peer reviewed papers confirming the pathway that I'm describing. And more importantly, once that initiation of the DSPC now the actual phospholipid that is encapsulating the lipid nanoparticle is a phospholipid called DSPC. I won't go into the name of it, but what it means is that that particular phospholipid we found, again through research, that it will liberate 80 to 90% of raw phosphorus heads as it releases the mRNA core.
Those phosphorus heads all react with the fibrinogen in the bloodstream and they cause sandy blood. The reason you guys are seeing sandy blood and coffee grounds is that's the nucleation pathway to the final white clot formation. The other factor we found and proved the spike protein bonds to the phosphorylated fibrinogen. The body is generating methylcetiopridine generated spike, that spike in the bloodstream then starts to coagulate with the phosphorylated fibrinogen, and that feeds what we call a monomeric reaction that continues to grow. Those particles are free flowing in the blood and they find an anchor point.
The anchor points they find are in fact the damaged endothelial layers. When an endothelial layer of the vascular system is damaged via inflammation and the cytokine storm that the spike protein generates, That opens up the natural phospholipid layer of the endothelial layer, and that forms anchor points for these nuclei. From these anchor points, that's when the clots begin to grow. So, I'm trying to encapsulate this in a very simple term. It's quite a complex number.
Very