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Speaker 0: When you put the patient into nutritional ketosis, it opens up a whole new array of drugs that may have been considered ineffective. This is the field of repurposing drugs: once in this new physiological state, things thought to be ineffective can become super effective. This is a whole new field that will emerge as the results get shared more. You mentioned mebendazole in your talk as well. Does that have a glutamine-blocking aspect to it? Yes, that's in our paper too. Parasite medications. I don’t know about ivermectin because there was a lot of political stuff around that, so I told the lab we’re not going to do that to avoid wrath from somebody.
Albendazole, fenbendazole, I said, why the hell do people have taken that? You hear people say, well, it got rid of my cancer with this and that. So I did a dive on it and asked why parasite medications work against cancer cells. It turns out that the parasites use mitochondrial substrate level phosphorylation in the tissue. Albendazole and fenbendazole kill these parasites, so I tried them on cancer cells, and sure as hell, they target the mitochondrial substrate and glycolysis. So we have a mechanism now why parasite medications are working.
But cancer is not a parasite. All these people say cancer is a parasite – it’s not. Parasites and tumors use a common metabolic pathway, and a drug that works against parasites can be very effective against cancer, and that’s what we begin to see, especially under nutritional ketosis. Right?
Speaker 1: Ivermectin actually works on the mitochondrial cell death pathway, the BCL2 Bax BAD pathway, so it actually helps push the cell death pathway.