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A recent paper suggests that COVID vaccines may meet the multi-hit hypothesis for cancer. According to the paper, the vaccines affect the p53 and BRCA tumor suppressor systems, impair DNA repair, and contain DNA fragments with cancer-promoting properties. The presence of SV40, a known cancer promoter, in the vaccines further supports this hypothesis. While it is important to note that this applies to some individuals, it suggests a potential link between the vaccines and cancer development.

Speaker 0 asserts that packaged DNA fragments have been found en masse as vaccine contaminants. Once they reach the nucleus, short DNA sequences have an increased propensity to insert into chromosomal DNA. The possible consequences are unending, including disruption of the exquisitely tuned network that controls cell division and differentiation, which can lead to cancer and developmental defects. Mutations in sperm and fertilized egg cells could render altered traits inheritable. Speaker 0 further states that cost effective procedures to reliably separate mass produced RNA from plasmids do not exist, and therefore contamination of RNA vaccines with plasmid DNA must be expected to be the rule and not the exception. Whoever propagates RNA vaccines as being safe and effective, whoever claims that nothing can happen to your genome is either incredibly ignorant or endlessly evil. That person is turning his back on the horror scenario that is unfolding in front of our very eyes. Fellow citizens and physicians of the world are urged to turn away from the perpetrators of this monstrous crime against humanity. Speaker 0 concludes with admonitions to do this to save yourself, your descendants, and to rescue the name of your family or go down in history as one of the greatest criminals of all time. Speaker 1 responds: Thank you very much, professor Bhakti. You continue to be an inspiration both scientifically and ethically for all of us.

The spike protein from various vaccines can bind to cancer-related genes, induce cancer pathways, and inhibit DNA repair. It can turn cells into spike factories, leading to immune system attacks and potential cancer development. Reports of increased cancers and harmful effects on organs have been observed. Military data was allegedly suppressed. This situation is seen as a crime against humanity.

But every mutation is the beginning of a microscopic cancer. Take a guess of how many mistakes in DNA of copying and pasting your own body are made every twenty four hours. Take a guess. This has been calculated randomly. Well, there's so many cells in my body, so it's gonna be a big number, a million? Okay. Every day, every twenty four hours, there are 10,000 mistakes that are made in your body that your body doesn't catch, that propagate in the document of our body as it goes on. 10,000. Each of those is a microscopic cancer. A microscopic cancer is just said. It's microscopic. It's too small to be seen with the naked eye, but it's abnormal. And that thing could turn into a big tumor that could eventually kill you. So why don't we die from cancer all the time?

Kevin McKernan recently discovered that there is contamination in the mRNA shots with cDNA, including a cancer-promoting segment called SV40. SV40 turns on cancer genes in the human body and impairs tumor suppressor systems. This means that the shots not only promote cancer through SV40 but also inhibit our ability to fight cancer. The increase in cancer rates is undeniable, but the question remains: how much of this is due to the vaccines?

The SV40 component, highlighted by David Dean and others, interacts with p53, known as the "guardian of the genome," crucial for maintaining DNA integrity. The introduction of billions of these molecules raises concerns about their effects, especially since they bind to p53. Research from the Brown Cancer Institute suggests that the spike protein may alter p53 transcription, potentially leading to cancer if p53 is compromised. Damaged DNA fragments can trigger the cGAS-STING pathway, signaling danger within cells and potentially leading to oncogenesis. There is skepticism about whether this DNA enters the nucleus, but even its presence in the cytosol can be harmful. Observations of rare cancers in vaccinated children, particularly blood cancers like lymphoma, raise alarms about these potential risks.

The speaker claims SV40 in literature turns on cancer genes. They further claim the spike protein impairs tumor suppressor systems P53 and BRCA, promoting cancer and inhibiting the ability to fight it. The speaker suggests cancer rates are up, and the question is how much is due to vaccines. They state that repeated shots every six months increase the chances of getting loaded with synthetic genetic material that will cause harm, including heart disease, neurologic disease, blood clotting, immunologic problems, and cancer.

I've been in cancer research for decades and have never seen anything like turbocancer before. It's a phenomenon where cancer develops rapidly in young people within months, leading to late-stage cancer and death. This acceleration is likely caused by genetic injections weakening the immune system, allowing tumors to grow unchecked. This process mirrors what we've observed in animals when their immune systems are compromised. Turbocancer is essentially the unchecked proliferation of cancer cells due to a compromised immune system.

The spike protein may inhibit tumor suppressor genes like MHS 3p53 and BRCA2, potentially leading to cancer. The mRNA vaccine contains a base that allows the spike protein to be produced for longer, possibly further inhibiting tumor suppressor genes. Concerns are raised about the long-term effects of these vaccines, with a call for them to be banned for general use and reserved for gene therapy in advanced cancer cases.

COVID-19 vaccines, especially mRNA ones, may promote cancer through impairing DNA repair, inhibiting tumor suppressor systems like p53 and BRCA, and containing DNA impurities. These impurities include fragments from circular DNA used in the manufacturing process, such as SV40, a known proto-oncogene activator. The vaccines could potentially initiate or accelerate cancer growth by weakening natural tumor surveillance systems. This phenomenon is referred to as "turbo cancer."

Cancer cannot occur if mitochondria in cells remain healthy, and healthy people are in metabolic homeostasis. Our bodies are falling out of this homeostasis due to environmental, dietary, and lifestyle factors. Mitochondria within cells are responsible for maintaining metabolic homeostasis. When this organelle becomes dysfunctional, it can manifest as cardiovascular disease, type two diabetes, cancer, or Alzheimer's disease, depending on the tissue and cells. In cancer, every major cancer studied has defects in the number, structure, and function of mitochondria. This causes cells to rely on fermentation, leading to dysregulated cell growth. There is a clear understanding of the origin of cancer and how to manage it.

We compared gene expression profiles across several groups: a healthy control group pre COVID, individuals described as mRNA injured, a group with cancer, and a few individuals who had neurological and cardiovascular adverse events. The analysis showed that in the mRNA injured group, thousands of gene expressions become dysfunctional in several key cellular pathways. Specifically, dysfunction was noted in mitochondrial function, immune function, and protein production. The changes included the production of abnormal proteins as a result of these altered gene expressions. In addition, the analysis reported that cancer surveillance genes were literally turned off in the mRNA injured group. The genes mentioned as turned off include p53, KRAS, and BRCA. This observation is presented as part of the overall pattern of molecular disruption associated with the mRNA injury state. Overall, the findings are described as indicating that flooding the body with synthetic messenger RNA unleashes biochemical havoc. The speaker emphasizes that this biochemical havoc has severe consequences, as evidenced by the observed widespread gene expression dysfunction and the suppression of critical cancer surveillance genes, alongside the production of abnormal proteins and impairments in mitochondrial, immune, and protein production pathways.

The mitochondria, not the nucleus, is the center of cancer. Cancer is a mitochondrial metabolic disease. Realizing this will massively drop death rates in just a few years. We may never completely get rid of cancer, but we can learn to live with it and keep it at bay. If we restrict the fuels that cancer needs through diet and lifestyle, and keep our mitochondria healthy, we can manage it. If we don't focus on the mitochondria, then almost 50% of people will continue to get cancer.

Inherited mutations like BRCA1 and p53 damage mitochondria and disrupt oxidative phosphorylation. BRCA1 isn't 100% penetrant, meaning not every woman with the mutation develops breast cancer. Since these mutations aren't fully penetrant, risk factors exist, similar to how not everyone exposed to chemical carcinogens develops cancer. Lifestyle can influence whether these genes are activated or deactivated. Diet and lifestyle could either provoke or diminish the impact of chemical carcinogens or genetic risk factors. Prophylactic organ removal may not be the best approach for BRCA1. Because it's not 100% penetrant, diet and lifestyle could reduce the risk associated with it.

Cancer is cell division out of control, caused by damage to oxidative phosphorylation, which leads to dysregulation of the cells. The organelle that controls the cell cycle is corrupted and not producing energy correctly. The primary cause of cancer is damage to the cell's ability to generate energy through oxygen. Radiation, chemical carcinogens, intermittent hypoxia, chronic inflammation, oncogenic viruses, and rare inherited mutations are secondary risk factors. The primary risk factor is damage to oxidative phosphorylation with a compensatory fermentation process, as Warburg stated. We live in an environment that makes this possible, coupled with a diet of highly processed carbohydrates, minimal exercise, and secondary risk factors. This leads to an epidemic of almost seventeen hundred people a day in The United States dying from cancer.

The speakers describe a study in which gene expression profiles are compared across several groups to assess the impact of mRNA injury. The comparison set includes a healthy control group that was observed before the COVID-19 era, individuals classified as mRNA injured, a subset of three individuals with cancer, and a smaller number of participants who exhibited neurological and cardiovascular adverse events. The central finding reported is that, in the mRNA-injured group, thousands of gene expressions become dysfunctional. The dysfunction spans several critical cellular and biological processes, notably mitochondrial function, immune function, and protein production. The speakers indicate that these alterations include the production of abnormal proteins as a consequence of the disrupted gene expression patterns. In addition to widespread dysfunction in metabolic and cellular pathways, the speakers note that genes involved in cancer surveillance are turned off in the mRNA-injured group. Specific genes named are p53, KRAS, and BRCA, with their expression or regulatory activity described as being suppressed or deactivated. The implication conveyed is that the disruption of cancer surveillance mechanisms accompanies the broader profile of gene expression changes observed in response to the mRNA injury. The overall conclusion presented by the speakers is that flooding the body with synthetic messenger RNA is associated with unleashing biochemical havoc, which they characterize as having severe consequences. The framing suggests a causal or strongly associative link between exposure to synthetic mRNA and the observed downstream effects on gene expression, including mitochondrial and immune dysfunction, abnormal protein production, and the suppression of key cancer-related surveillance genes. The narrative emphasizes the magnitude of the molecular disturbances, noting that thousands of gene expressions become dysfunctional and that critical safeguards against cancer may be compromised in the mRNA-injured group.

The DNA sequence in gene therapy plasmids contains the SV40 promoter and enhancer region, an SV40 origin of replication, and an SV40 poly A signal. The SV40 enhancers are nuclear targeting sequences, ensuring the DNA enters the cell nucleus, especially during cell division when the nuclear envelope dissolves. Claims that it doesn't reach the nucleus are misleading. This plasmid was sourced from Pfizer's gene therapy department. The SV40 promoter and enhancer bind to the p53 gene, a tumor suppressor, which is concerning given that the spike protein also inhibits p53 expression. Literature indicates this sequence is a hypermutability element, inducing mutations in nearby DNA, suggesting potential tumorigenic activity. These findings contradict claims that this DNA has no function.

Research has shown that COVID vaccines are contaminated with DNA, including a cancer virus promoter sequence. The DNA can enter the human genome, potentially activating carcinogenic genes and increasing cancer risk. Vaccines suppress immunity, further raising cancer risk. The presence of DNA in vaccines can lead to long-lasting spike production, causing health issues. Vaccines inducing IgG4 antibodies are considered defective. DNA impurities in vaccines can intensify side effects. DNA should not be introduced into the body's cells.

There are concerns that the COVID-19 vaccines may have a potential link to cancer. The spike protein in the vaccines could inhibit tumor suppressor systems in the body. Additionally, the vaccines may impair natural DNA repair mechanisms, increasing the risk of DNA damage. Contamination has also been found in Pfizer vials, which could lead to a direct DNA injection and activation of cancer-related genes. This multi-hit hypothesis suggests that repeated vaccination could promote cancer development. There have been clinical observations of rapid cancer progression and reactivation of cancers in remission after vaccination. However, no agency has confirmed a direct link between vaccines and cancer.

This discussion reviews a series of replicated experiments that distinguish between cellular growth patterns in normal versus tumor cells and investigate the underlying cause of dysregulated growth. The core observations begin with the distinction between green cells and red cells: green cells produce green progeny with healthy nuclei, mitochondria that are number-stable, structurally sound, and function effectively, enabling regulated growth during normal turnover. In contrast, red cells are tumor cells that beget tumor cells, and these cells commonly harbor genetic defects and abnormalities in the number, structure, and function of mitochondria. The central question addressed is what drives the dysregulated growth observed in tumor cells: nucleus mutations or cytoplasmic mitochondrial abnormalities. In one key set of experiments by Israel and Schafer, the nucleus from a red tumor cell placed into the green cytoplasm produced regulated growth, both in vitro and in vivo, which was unexpected given the tumor nucleus. Conversely, transferring the nucleus from a normal cell into the cytoplasm of a tumor cell resulted in dysregulated growth. These outcomes challenge the notion that driver genes within the nucleus solely control dysregulated cell growth, as the results show that the cytoplasmic context can override nuclear origin. Further experiments emphasize the role of mitochondria. When green mitochondria were purified and introduced into red cytoplasm, regulated growth was observed. In another finding, introducing abnormal mitochondria into indolent cells caused those cells to become explosive, displaying rapid and unregulated growth. These results collectively indicate that the observed disorder is driven by mitochondrial dysfunction rather than nuclear genetic mutations. In summary, the evidence presented supports the view that mitochondrial dysfunction, rather than nuclear mutations, drives the dysregulated growth characteristic of tumor cells. The experiments demonstrate that altering cytoplasmic mitochondrial content can shift growth from dysregulated to regulated, and that introducing abnormal mitochondria into otherwise normal or indolent cells can induce aggressive, dysregulated growth. The overarching conclusion drawn is that the disorder is fundamentally mitochondrial in origin.

The discussion reports a comparative analysis of gene expression profiles among four groups: a healthy control group pre-COVID, individuals who were injured by mRNA, and subgroups including three individuals with cancer and a few with neurological and cardiovascular adverse events. The study found that in the mRNA injured group, thousands of gene expressions become dysfunctional, affecting mitochondrial function, immune function, and protein production, leading to the creation of abnormal proteins. It also notes that cancer surveillance genes are literally turned off, specifically mentioning p53 and KRAS, as well as BRCA. The overall claim is that flooding the body with synthetic messenger RNA unleashes biochemical havoc, with severe consequences.

Cancer is cell division out of control caused by damage to oxidative phosphorylation, leading to dysregulated cell growth. The organelle controlling the cell cycle is corrupted and not producing energy correctly. The environment damages energy metabolism in cells, causing dysregulated cell growth. While there are many secondary causes of cancer, the primary cause is damage to the cell's ability to generate energy through oxygen. Radiation, chemical carcinogens, intermittent hypoxia, chronic inflammation, oncogenic viruses, and rare inherited mutations are secondary risk factors. The primary risk factor is damage to oxidative phosphorylation with compensatory fermentation, as Warburg stated. Today's environment makes this possible, and when coupled with a diet high in processed carbohydrates, minimal exercise, and secondary risk factors, it results in an epidemic of deaths from cancer.

Cancer cannot occur if mitochondria in cells remain healthy, and healthy people are in metabolic homeostasis. Our bodies are falling out of this homeostasis due to environmental, dietary, and lifestyle factors. Mitochondria maintain metabolic homeostasis within cells and the body. When mitochondria become dysfunctional, it can manifest as cardiovascular disease, type two diabetes, cancer, or Alzheimer's, depending on the individual's cells and tissues. Every major cancer studied has defects in the number, structure, and function of mitochondria. This causes cells to rely on fermentation, leading to dysregulated cell growth. The speaker claims to have a clear idea of the origin of cancer and how to manage it.

Inherited mutations like BRCA1 and p53 damage mitochondria and disrupt oxidative phosphorylation. BRCA1 isn't 100% penetrant, meaning not every woman with the mutation develops breast cancer. Since these mutations aren't fully penetrant, risk factors exist, similar to how not everyone exposed to chemical carcinogens develops cancer. Lifestyle can influence whether these genes are activated or deactivated. Diet and lifestyle can either provoke or diminish the prominence of chemical carcinogens or genetic risk factors. Prophylactic organ removal may not be the best approach for BRCA1. Because BRCA1 isn't 100% penetrant, diet and lifestyle could reduce the risk associated with it.