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What was once considered an "old timer's disease" actually begins in your 20s and 30s, though diagnosis often occurs much later. Epidemiological data indicates that Alzheimer's diagnoses are increasing in the 40-50 age range, not just in the 60-70 range. Just as prediabetes and insulin resistance can be identified early, it's now possible to see what's happening in the brain much earlier. This allows for intervention and prevention of progression to full-blown dementia.

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Sugar consumption has increased 400-fold since 1964, and blood sugar is the root of much illness. Alzheimer's disease is type three diabetes, or insulin resistance in the brain. The brain is crack-addicted to sugar and even secretes its own insulin. The brain will take what it needs, leaching calcium from bones or stripping amino acids from muscle. If the brain wants sugar, it activates the r f one a two receptor on the back of the tongue, which provides a dopamine release when sugar is consumed. The trick is you have to swallow.

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Ten percent of 65-year-olds, 25% of 75-year-olds, and 50% of 85-year-olds will develop dementia or Alzheimer's disease. Despite spending over $2 billion on drug treatments, efforts have failed. Scientists at Brown discovered insulin resistance in the brain, likening Alzheimer's to type 3 diabetes. Poor metabolic health, often due to insulin resistance caused by excessive insulin and sugar, drives the cascade leading to Alzheimer's. Evidence of Alzheimer's can appear 30 to 40 years before symptoms arise. The typical diet, high in sugar and starch, contributes to diabetes of the brain. Understanding this allows individuals to heal their brains.

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The brain is the most metabolically demanding organ, relying heavily on glucose from carbohydrates, though it can also use ketones. Blood flow to the brain is crucial for cognitive function; improving it enhances cognition, while restricting it impairs it. Age-related cognitive decline and dementia, sometimes referred to as type 3 diabetes, can benefit from dietary changes. Some individuals with Alzheimer's experience relief on ketogenic diets, as this approach can improve blood flow and reduce brain inflammation. While it's not a cure for Alzheimer's, reducing inflammation is linked to enhanced cognitive function.

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Inflammation releases pro-inflammatory proteins, causing insulin resistance. Insulin resistance in various tissues spreads chronic disease. Hypertension, the most common cardiovascular problem, is mainly caused by insulin resistance. Alzheimer's disease is referred to as type three diabetes, or insulin resistance of the brain. Erectile dysfunction, a common form of male infertility, stems from insulin resistance in blood vessels. Polycystic ovary syndrome (PCOS), a common cause of female infertility, results from insulin resistance affecting the ovaries and sex hormone production.

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Alzheimer's and dementia are actually type 3 diabetes, caused by insulin resistance in the brain. Elevated blood sugar levels for 10 years prior to diagnosis are common in early onset patients. The misconception that these diseases cause memory loss is incorrect; patients lose access to their memories, but this access can be restored. The brain consumes 45% of the carbohydrates we consume, and it will take what it needs from other sources, such as calcium from bones or amino acids from muscle tissue. The brain also activates a receptor that rewards us with dopamine for consuming sugar, leading to addiction for many.

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Alzheimer's and dementia should be called type three diabetes, as they are caused by insulin resistance in the brain. The brain makes its own insulin, despite what endocrinologists say about the pancreas being the sole producer. The brain becomes addicted to sugar and will do what it takes to get it, including stripping calcium from bones or activating the r f one a two receptor for a dopamine reward. Excess sugar in the brain is stored as amyloid plaque in the neurosynaptic junctions. People with Alzheimer's are not losing their memory, but rather losing access to it due to this buildup.

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Insulin resistance occurs when cells resist insulin's attempts to deliver glucose. After eating, glucose is created, and insulin transports it to cells. Overeating causes cells to reject the glucose, but the body continues producing insulin. To avoid diabetes, insulin stores the excess glucose as fat, especially around the belly and organs, elevates triglycerides, and creates a fatty liver. Diabetes occurs when insulin can no longer store the glucose and it ends up in the blood. A standard A1C diabetes test may not detect insulin resistance, as it often appears normal until the condition has progressed for years. A specific insulin resistance test exists. However, if you have poor nutrition, excess belly fat, and elevated cholesterol, you are likely insulin resistant, regardless of a normal A1C result. It is important to take action before it's too late.

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We are one of the few nations still calling Alzheimer's and dementia Type 3 diabetes. Insulin resistance in the brain, known as Type 3 diabetes, is the main cause of Alzheimer's. The myth that Alzheimer's is memory loss is false; it's actually loss of memory access, which can be restored. Look into the link between COMT gene mutation and diabetes. Insulin's main role is not to lower blood sugar, but to block other energy use in the body.

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To overcome Alzheimer's, it is crucial to avoid a specific food that worsens symptoms. Insulin plays a protective role in the brain, preventing the development of amyloid plaques and tangled fibers. However, consuming carbs increases insulin, leading to insulin resistance. This resistance creates a deficiency of insulin inside cells, including the brain. Carbs, especially sugars and refined grains, also cause inflammation and mitochondrial damage in the brain. To address insulin resistance, it is recommended to follow a ketogenic diet, lower carb intake, and practice intermittent fasting, such as one meal a day. Additionally, reducing stress is important as it can increase cortisol levels and glucose, contributing to insulin resistance. A nutrient-dense and healthy meal is essential.

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The development of Alzheimer's goes through four phases. In phase one, individuals are asymptomatic, but it can be detected via PET scans, spinal fluid, and the new P tau217 test, which can identify brain signaling tendencies toward Alzheimer's before it manifests. Phase two is subjective cognitive impairment (SCI), lasting about ten years, where individuals sense something is wrong, but cognitive tests appear normal. Phase three is mild cognitive impairment (MCI), where individuals struggle with cognitive tests but can still care for themselves. The final phase is dementia, where individuals struggle with activities of daily living. Early intervention is key, as virtually everyone with SCI can be turned around. Reversals of cognitive decline have been observed, but the later one waits, the harder it is to achieve a complete return.

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The brain is the most metabolically demanding organ, relying heavily on blood flow for cognitive function. Improved blood flow enhances cognition, while restricted blood flow impairs it. Age-related cognitive decline and dementia are sometimes called type three diabetes or diabetes of the brain. Some Alzheimer's patients find relief on ketogenic diets, which isn't a cure, but can improve brain function by improving blood flow and reducing inflammation. Inflammation depletes cognitive function, while reducing inflammation enhances it.

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Insulin resistance occurs when cells resist insulin's efforts to move glucose, leading to excess glucose in the blood. This can result in fat storage, elevated cholesterol, and a fatty liver. The usual diabetes test may not detect insulin resistance, so symptoms like belly fat and high cholesterol should not be ignored. By addressing nutrition and lifestyle factors early, you can prevent diabetes.

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Speaker 0 raises the question of whether there is a link between cognitive degeneration diseases like Alzheimer's and the ketogenic diet, noting ongoing research to establish causality. Speaker 1 responds with clinical observations and experience. They describe patients expressing regrets late in life, noting that deterioration has been ongoing for years before recognition. They say there is no research proving reversal of Alzheimer's before onset, but clinically these patients often show noticeable improvement on a ketogenic diet, even after years of decline. They recount a specific case: a woman with Down syndrome diagnosed with Alzheimer's-like symptoms at around age 40. The mother began a ketogenic diet for her daughter after previously losing about 100 pounds, and both mother and daughter rapidly produced high ketone levels, with ketones around three, compared to an average of one. Within a week, the daughter showed improved functional abilities, performing tasks around the house. A week later, the mother reported the daughter had begun asking to participate in church activities, and for the first time in the daughter's life, she could understand and respond with a three-syllable word (previously only two-syllable words were possible). The mother observed that the daughter, who had been Alzheimer’s-diagnosed, appeared to have improved brain function on the ketogenic diet, and the daughter also lost additional weight (about 15 to 20 pounds). Speaker 0 references supporting studies: in dementia and Alzheimer's disease, the brain struggles to use glucose efficiently, and ketones provide an alternative, cleaner fuel source. Ketogenic diets can boost mitochondrial function and energy availability in brain cells, and keto lowers systemic inflammation, which is linked to slower cognitive decline. Ketones may protect neurons from damage and promote the growth of new neural connections. Alzheimer's is described as “type three diabetes,” with keto improving insulin sensitivity and potentially reducing risk. Small studies show temporary improvements in memory and cognition in people with mild cognitive impairment or early Alzheimer's, but the evidence is early-stage. Long-term adherence can be difficult, and the diet isn’t suitable for everyone, such as underweight individuals or people with certain medical conditions.

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Alzheimer's is a vascular disease characterized by reduced blood flow to the brain (focal ischemia) and insulin resistance, sometimes called diabetes type three, which impairs glucose uptake by brain cells. This leads to oxidative stress, immune dysfunction, and misfolded proteins, manifesting as tau tangles and amyloid plaques. Nitric oxide can potentially correct all known aspects of Alzheimer's. It improves blood flow and glucose uptake in the brain, addresses the metabolic component, reduces inflammation, inhibits oxidative stress, and prevents immune dysfunction.

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Alzheimer's is now being referred to as diabetes type 3 due to the impact of a high carbohydrate diet. Some presenters argue that fat contributes to diabetes, but the only fat that can do so is margarine because it can block receptor sites. The brain is 70% fat and loves fat as fuel. Coconut oil and olive oil are concentrated foods that can be consumed in small amounts. For example, I have about two teaspoons of olive oil daily. However, if I had Alzheimer's, I would take three tablespoons of coconut oil per day. The book "Stop Alzheimer's Now" shares stories of people who have reversed Alzheimer's by using coconut oil. Additionally, coconut oil is also beneficial for autism as it acts as a neuro healer.

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Insulin resistance can lead to prediabetes, diabetes, and related complications. For 10-15 years, blood sugar levels can remain normal, but more insulin is required to maintain them. With insulin resistance, each time you eat, blood sugar spikes, and the body produces more insulin to compensate. Eventually, the body can't keep blood sugar at normal levels, leading to prediabetes. Blood sugar tests may reveal higher-than-normal levels, such as over 100 for a fasting test. As the condition progresses, the body's insulin production declines, resulting in uncontrolled diabetes, where blood sugar rises despite high or decreasing insulin levels. Early detection is crucial, but blood glucose tests are lagging indicators. Testing fasting insulin levels can help identify climbing insulin levels, indicating potential problems earlier.

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What was once considered an "old timer's disease" actually begins in your 20s and 30s, though diagnosis often occurs much later. Epidemiological data indicates that Alzheimer's diagnoses are increasing in the 40-50 age range, not the 60-70 range. Just as prediabetes and insulin resistance can be identified early, it's now possible to detect early signs of Alzheimer's. This allows for intervention and prevention of progression to full-blown dementia.

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Alzheimer's is referred to as type three diabetes, meaning insulin resistance in the brain. Our brains become highly dependent on sugar, and the brain manufactures its own insulin. The problem is that when insulin resistance occurs in the brain, unlike the body, there isn’t a stored-sugar exchange through glycogen. The body stores sugar as glycogen in the liver and muscles and can release glucose back into the bloodstream, but the brain lacks this same storage-and-release mechanism. Within the brain, there are neurosynaptic junctions—little spaces where nerve endings don’t touch, and signals jump across the gap. When these gaps fill with amyloid plaques and neurofibrillary tangles, people say that’s the genesis of Alzheimer's. However, the transcript states that the truth is the genesis of it was insulin resistance.

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Ten percent of 65-year-olds, 25% of 75-year-olds, and 50% of 85-year-olds will develop dementia or Alzheimer's disease. Despite spending over $2 billion on drug treatments, there has been no success. Scientists at Brown discovered insulin resistance in the brain, likening Alzheimer's to type 3 diabetes. Poor metabolic health, often due to insulin resistance caused by excessive insulin and sugar, is a significant factor in Alzheimer's. Evidence of Alzheimer's can appear 30 to 40 years before symptoms arise. The prevalence of sugar and starch in diets contributes to diabetes of the brain. Understanding this connection can lead to brain healing.

The Dhru Purohit Show

Worst Thing In The World Feeding Alzheimer's & Dementia! - Fix This ASAP In 2025 | Ben Bikman

Guests: Ben Bikman

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Alzheimer's disease has traditionally been viewed as a result of amyloid beta plaques and neurofibrillary tangles in the brain, which can only be confirmed postmortem. This has led to a need for a paradigm shift in understanding the disease. Emerging research suggests a metabolic theory of cognitive decline, indicating that markers of metabolic health, such as fasting insulin and glucose levels, may be more predictive of Alzheimer's risk than age. A Finnish study found that fasting insulin was a stronger predictor of Alzheimer's than age. The hippocampus, crucial for memory and learning, relies on glucose and ketones for energy. Insulin regulates glucose transport in the hippocampus, and as insulin resistance develops, the brain struggles to access glucose, leading to an energy deficit. This has led some researchers to label Alzheimer's as "type 3 diabetes," highlighting the connection between insulin resistance and cognitive decline. Insulin's role extends beyond glucose control; it affects every cell in the body. Chronic high carbohydrate intake leads to elevated insulin levels, causing insulin resistance and metabolic dysfunction. This dysfunction can manifest in various health issues, including cognitive decline, PCOS, and erectile dysfunction, all linked to insulin resistance. Research indicates that even in early cognitive decline, glucose metabolism in the brain is impaired while ketone metabolism remains intact. Studies show that dietary changes, particularly reducing carbohydrates and increasing healthy fats, can improve cognitive function. The food we consume plays a critical role in either exacerbating or alleviating chronic diseases, including Alzheimer's. To combat cognitive decline, individuals should focus on improving metabolic health through dietary changes, such as reducing refined carbohydrates and increasing healthy fats. This approach not only addresses Alzheimer's risk but also enhances overall well-being. The message is one of empowerment: individuals can take control of their health through informed dietary choices.

The Dhru Purohit Show

The ROOT CAUSE Of Alzheimer's & How To REVERSE IT! | Dr. Dale Bredesen

Guests: Dale Bredesen

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Alzheimer's disease typically takes about 20 years from the onset of pathophysiology to diagnosis. Cognitive improvement is possible at any age by addressing underlying causes, such as unrecognized toxins or pathogens. Notably, nocturnal oxygen desaturation is critical; studies show that low oxygen levels during sleep correlate with brain volume loss. Many people are unaware of their declining oxygen saturation due to conditions like sleep apnea, which can starve the brain. As the aging population grows, cognitive decline has become a primary concern, surpassing cancer. Currently, around 5.6 million Americans have an Alzheimer's diagnosis, but projections suggest that 45 million could eventually die from the disease. Factors like obesity, poor immunity, and low vitamin D levels increase risks for both Alzheimer's and COVID-19. The complexity of Alzheimer's means it cannot be attributed to a single cause. Early lifestyle factors in younger individuals can increase future risk. The disease is often linked to inflammation, toxins, and hormonal deficiencies. For instance, insulin resistance contributes to cognitive decline through inflammation and reduced trophic support. Addressing these factors early can prevent or reverse symptoms. Dale Bredesen emphasizes the importance of personalized approaches to treatment, identifying root causes, and creating targeted interventions. His research indicates that multiple contributors, including toxins from mold and environmental factors, must be addressed for effective treatment. The Bredesen protocol encourages individuals over 45 to undergo a "cognoscopy" to assess cognitive health proactively. Success stories illustrate that individuals can improve their cognitive function by following a comprehensive protocol that includes lifestyle changes, dietary adjustments, and addressing environmental toxins. The narrative of patients like Deborah, who recognized early signs of cognitive decline and sought intervention, highlights the importance of early detection and personalized care. The overarching message is that Alzheimer's can be an optional disease with appropriate preventive measures and lifestyle adjustments.

The Dhru Purohit Show

DOCTORS REVEAL How to PREVENT Alzheimer's in 6 SIMPLE STEPS!

Guests: Ronesh Sinha, Lisa Mosconi, Dale Bredesen

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Alzheimer's disease is projected to affect 45 million Americans, with lifestyle factors like diet and insulin resistance playing critical roles in brain health. The Western diet accelerates brain aging, while fasting can help reverse these conditions. Insulin resistance occurs when the body struggles to manage glucose, leading to various metabolic issues. This condition can manifest years before glucose levels rise, making it essential to monitor waist circumference and lipid panels as early indicators. The link between insulin resistance and Alzheimer's is significant; elevated glucose and insulin levels can hinder the brain's ability to clear amyloid plaques, which are associated with Alzheimer's. Chronic inflammation and insulin resistance create a perfect storm for neurodegenerative diseases. To reduce Alzheimer's risk, individuals should evaluate their lifestyle and health metrics, focusing on triglyceride levels, HDL cholesterol, liver function tests, and inflammation markers. Dietary habits, particularly carbohydrate intake, are crucial in managing insulin resistance. Many people, especially those from South Asian backgrounds, may consume healthy foods that still contribute to insulin resistance due to high carbohydrate content. The concept of "skinny fat" highlights that individuals can appear thin yet have high visceral fat, which is more harmful than subcutaneous fat. This phenomenon is particularly prevalent in certain ethnic groups, where even small amounts of excess weight can lead to significant metabolic issues. The discussion emphasizes the importance of personalized nutrition and exercise, as well as the need for awareness about the impact of modern diets and sedentary lifestyles on health. Fasting and meal sequencing can help manage blood sugar levels, and incorporating more vegetables and healthy fats into meals can mitigate the effects of carbohydrates. Women are disproportionately affected by Alzheimer's, with hormonal changes during menopause contributing to cognitive decline. Estradiol, a key hormone, supports brain health, and its decline can lead to increased risk for Alzheimer's. Understanding these changes is vital for prevention, and early intervention can significantly impact long-term health outcomes. The conversation also touches on the importance of addressing environmental toxins, such as heavy metals and mold, which can contribute to cognitive decline. Regular monitoring and proactive health measures, including brain scans and lifestyle modifications, are essential for reducing the risk of Alzheimer's and other neurodegenerative diseases. Overall, the key takeaways include the importance of diet, exercise, hormonal health, and environmental factors in maintaining cognitive function and preventing Alzheimer's disease. Individuals are encouraged to take proactive steps in their health journey, focusing on personalized strategies that align with their unique needs and backgrounds.

The Dhru Purohit Show

Avoid These RISK FACTORS To Prevent BRIAN INFLAMMATION! | Dr. Datis Kharrazian

Guests: Datis Kharrazian

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In this discussion, Dr. Datis Kharrazian and Dhru Purohit explore the critical link between brain inflammation, insulin surges, and neurodegenerative diseases like Alzheimer's. Dr. Kharrazian introduces the concept of "type 3 diabetes," emphasizing that insulin resistance and surges can lead to brain fatigue and inflammation, which are precursors to cognitive decline. He highlights that feeling excessively tired after meals is a significant indicator of insulin surges, which can activate neuroglial cells in the brain, leading to inflammation. The conversation also touches on the role of body fat in neuroinflammation, with fat cells acting as endocrine glands that release inflammatory cytokines. Dr. Kharrazian explains that neuroinflammation can be triggered by various factors, including air pollution, diet, and lifestyle choices, which can impair the brain's ability to clear debris and maintain function. They discuss the importance of sleep and exercise in promoting neuroglial health, as well as the impact of diet, particularly the consumption of high-flavonoid foods, on brain function. Dr. Kharrazian stresses the need for stable blood sugar levels to prevent neuroinflammation and improve cognitive endurance, suggesting that individuals should avoid energy fluctuations after meals. The discussion concludes with practical advice for maintaining brain health, including improving indoor air quality, managing blood sugar, and incorporating anti-inflammatory foods and supplements. Dr. Kharrazian emphasizes that these lifestyle changes can significantly enhance brain function and reduce the risk of neurodegenerative diseases.

The Dhru Purohit Show

How Insulin Resistance DESTROYS Your Brain & Causes ALZHEIMER'S / DEMENTIA! | Ben Bikman

Guests: Benjamin Bikman

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In this podcast episode, Dr. Benjamin Bikman discusses strategies to improve insulin sensitivity and reduce the risk of Alzheimer's disease and cognitive decline. He emphasizes the need to shift the current paradigm in understanding Alzheimer's, which has traditionally focused on amyloid plaques and neurofibrillary tangles, to a metabolic perspective. Research indicates that fasting insulin levels are more predictive of Alzheimer's risk than age, suggesting that metabolic health is crucial in understanding cognitive decline. Dr. Bikman explains that the hippocampus, essential for memory and learning, relies on glucose and ketones for energy. Insulin resistance in the hippocampus can lead to an energy deficit, contributing to cognitive decline. He introduces the concept of "type 3 diabetes," referring to insulin resistance in the brain, which parallels type 2 diabetes. This highlights the metabolic origins of Alzheimer's, suggesting that poor metabolic health is a significant factor. The discussion also covers the impact of diet on insulin sensitivity. Dr. Bikman advocates for reducing processed carbohydrates and increasing healthy fats, particularly from animal sources and fruits like avocados and olives. He warns against industrial seed oils, which contribute to inflammation and insulin resistance. The conversation touches on the importance of maintaining a balanced omega-6 to omega-3 ratio to mitigate inflammation, which is linked to cognitive decline. Dr. Bikman encourages listeners to recognize early signs of cognitive impairment and take proactive steps to improve metabolic health through dietary changes, stress management, and inflammation control. He asserts that food can be a powerful tool in preventing and potentially reversing cognitive decline, empowering individuals to take charge of their health.