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Speaker: A lot of the analysis techniques that were being employed now on the white clots. And about three years ago, there was one piece of analyses that I came across just by accident, which was a thing called an ICP analysis, which is an analysis that determines the elements present in white clots. It was done by a gentleman in The USA called Mike Adams, who presented the findings of his initial white clot analysis from samples that Richard had provided him some almost three years ago now. And it surprised me what the findings were, because I have used the ICP analysis. It’s called Inductively Coupled Plasma Mass Spectrometry. Don't worry about the name. It's just a very well known piece of analytical equipment that you can rely upon to tell you what elements are there in the white clot and in what abundance. And what surprised me was in the analysis that Mike presented was a very the highest element they found was phosphorus, followed by sodium, tin, and later on sulphur, and carbon. And we also found that there were no normal blood marker elements present in the white clots. So, we've actually employed two laboratories in Europe, and there's a long reason behind that because I'm associated with people in Europe who do this kind of work. Anyway, we sent clot samples, they all ran ICP analyses. They found exactly the same results that Mike Adams found. We found aberrantly high levels of phosphorus, we found aberrantly high levels of sulphur, tin, sodium, and carbon. And being an organic chemist, I know a little bit about tin chemistry, because I used to sell tin catalysts, believe it or not, for polymerisation reactions. So I had to study the chemistry of tin. And I wondered why tin would be present in a white clot. So, obviously, we could not believe this first series of results. It's set about replicating the results in two separate labs, and they came back and said, you're right, there's high levels of phosphorus, tin, sulfon, carbon. So the next thing we did was to do an analysis called HPLC, High Performance Liquid Chromatography. And what this does is actually pulls apart the white clots and tells you what the protein components are. And surprisingly, we found that the highest level of proteins we found was fibrinogen. But the HPLC analysis actually will determine what kinds of fibrinogen chains are there. In a normal red blood clot, there's normally one to one to one ratio of the alpha chain, beta chain and gamma chain. We found in our white clots that the beta chain far exceeded the alpha chain and the gamma chain. And in fact, it was beta gamma alpha. So the alpha was the lowest proportion. Now, we're not biochemists, and we're not medically qualified in any way. But we can do simple research to find out why the fibrinogen content was so high. Fibrinogen forms fibrils. This is exactly why when John O'Learny first described the white clots as being calamari like, he is exactly correct. That's exactly the texture that fibrin will bring into a white clot. Okay, so from then on, we then ran some amino analysis results. My colleagues ran an amino acid analysis, and they found a high level of Praline, Aspartic acid, Lysine, a whole range of about 18 amino acids, all that have a phosphorus affinity. And as we said in the white clot, the element that has the highest concentration that we could confirm is porpoise. If you take time then to research the aberrant pathways that what they call excess phosphorylation will cause, it causes a whole range of problems in the human body. So we have determined, we think, the pathway to the formation of these white clots. We have three separate pieces of analysis, all confirming our findings. So we've now pieced together the formation of the white clots, and I'll come back to the very beginning. When we administer the injections, there is a lipid nanoparticle carrier, it's called a phospholipid. We found by our analysis that when the phospholipid releases the mRNA core of the lipid nanoparticle, at the very moment that it releases the core, it actually exposes a phosphorus head of the phosphorus lipid. The phosphor lipid reacts within the bloodstream naturally formed fibrinogen, and that's what's nucleating the white clot formation. Now we can prove all this. We've actually got over 200 peer reviewed papers confirming the pathway that I'm describing. And more importantly, once that initiation of the DSPC now the actual phospholipid that is encapsulating the lipid nanoparticle is a phospholipid called DSPC. I won't go into the name of it, but what it means is that that particular phospholipid we found, again through research, that it will liberate 80 to 90% of raw phosphorus heads as it releases the mRNA core. Those phosphorus heads all react with the fibrinogen in the bloodstream and they cause sandy blood. The reason you guys are seeing sandy blood and coffee grounds is that's the nucleation pathway to the final white clot formation. The other factor we found and proved the spike protein bonds to the phosphorylated fibrinogen. The body is generating methylcetiopridine generated spike, that spike in the bloodstream then starts to coagulate with the phosphorylated fibrinogen, and that feeds what we call a monomeric reaction that continues to grow. Those particles are free flowing in the blood and they find an anchor point. The anchor points they find are in fact the damaged endothelial layers. When an endothelial layer of the vascular system is damaged via inflammation and the cytokine storm that the spike protein generates, That opens up the natural phospholipid layer of the endothelial layer, and that forms anchor points for these nuclei. From these anchor points, that's when the clots begin to grow. So, I'm trying to encapsulate this in a very simple term. It's quite a complex number. Very

For all you science lovers, check this out. You'll notice a change happening here as these white nanoparticles rush in, drastically altering the cells. They're no longer round and normal. In the final picture, just above the fourth image, observe how the blood cells have lost their smooth, symmetrical shape. They're now covered in lumps and protrusions. According to Dr. Sherri Timpenny and Luke Montagner, this is an intentional attack on human blood.

I mixed dental anesthetic with blood and saw tiny machines that are not platelets. These robots are building something, which is not normal. They are dead erythrocytes, not platelets. The robots are jumping around, not platelets. It's surprising to see these little robots working.

We invited the staff of the Giovannini studio to discuss their study on the blood of over a thousand people, including those who were inoculated and those who were not. They found that the blood of inoculated individuals showed alterations in shape and arrangement of red blood cells when observed under dark field microscopy. In a normal blood sample, the red blood cells are spherical and well-oxygenated, with no foreign bodies present. However, in the blood of inoculated individuals, they observed the presence of nanotube-shaped inclusions that attracted red blood cells, leading to potential circulatory issues. This phenomenon, known as impelamento, can have thrombogenic effects. This occurs specifically in the blood of inoculated individuals.

The clots being formed, we've run mass spectrometry on these clots. They don't look like normal clots. They don't have the same composition. They don't have fibrin. They don't have thrombin, which are normal things you would see in a normal coagulation cascade. They have, instead, all the fibrinogen chains—alpha, beta, and gamma. They have them in a strange differential where it's not one-to-one-to-one; it's about 36 to 16 to 4 by ratios. They're aberrantly cross-linked by sulfide bonds. There's a ton of tin for some reason. I don't know why there's tin in there, but there's a ton of tin in there and a ton of phosphorus. And the spike protein is actually coated in GLIKNAK, which is a phosphate donor. So that might explain all the phosphorus if it's providing the energetics or in some way by cleaving or creating phosphate bonds. So I think that that's a big problem because that's a slow progression of coagulopathy that's, I think, narrowing the lumens of the vascular system, which is contributing to some of the organ failure that we're seeing, some of the neurological symptomatology that we're seeing, some of the fatigue, and things of that nature. And then finally, the spike protein is shown to produce—it’s shown to induce misfolding of proteins and actually

I've examined blood samples and have observed anomalies in everyone I've tested, even myself. I'm now seeing things I never saw in un*vaxxed individuals before. I'm noticing unusual formations, like chains potentially building, which typically occurs when the blood is breaking down. These formations seem more noticeable during decomposition. What's interesting is that while the body decomposes, these anomalies don't. They change and morph into something else. The red blood cells are visible, but when these formations mass together, that's when they evolve or morph. It could happen at any moment.

The coronavirus spike protein's shape before interacting with our cells is key to triggering an antibody response. To study this, we create the spike protein in the lab, maintaining its precise shape. This is achieved using a "clamp"—a small fragment of HIV protein—that holds the spike protein in its natural, pre-interaction conformation. This ensures the lab-made protein accurately reflects the virus's structure, allowing for effective antibody response studies.

I washed my slides and found similar anomalies in everyone, including myself, which I hadn't seen in the unvaccinated before. I observe my blood regularly, and I've noticed some unusual formations that resemble liver congestion. These changes seem to become more apparent as the body decomposes, yet the anomalies themselves do not decompose; they morph into different forms. The red blood cells are present, but when they gather into masses, they begin to evolve. This process hasn't started yet, but it could happen at any moment.

Under a darkfield microscope, the speaker examined the Pfizer vaccine liquid and observed circular and square particles floating around, which they did not see in older vaccines like measles and flu shots. After the liquid sat overnight, long wires appeared to form. Later, the speaker observed what looked like the image of a circuit board. The speaker was freaked out by this and worried about potential repercussions, given prior death threats. They questioned what these microscopic observations might imply and what potential problems could arise.

We purchased spike protein subunit MFC tag from Sino Biological and prepared it using doubly distilled water. A stock solution (0.25 mg/ml) was created by adding 400 µl of diluent to 100 µg of spike protein. This was diluted to working solutions. We assessed different spike protein concentrations in platelet-poor plasma using fluorescence microscopy. A healthy blood sample was divided into four tubes with varying spike protein concentrations (1000, 100, 50, and 1 ng/ml). Samples were incubated for 30 minutes at room temperature. We're replicating this experiment. I'll extract blood, add PBS buffer, and the spike protein. Then we'll look at the fluorescence.

Clumping of red blood cells in the vascular system is considered a major problem because it blocks blood flow in capillaries, impeding oxygen and nutrient transfer to tissues and waste removal. A microscope slide shows red blood cells clumping into long chains in human blood after five minutes of exposure to a video display terminal, model Ikigami EM125A. After five minutes of exposing the blood to a shielded, full spectrum OttLite fluorescent fixture, model 2,020, the long chains of red cells break up, using darkfield microscopy.

Here's a shorter version of the transcript: We're examining fluorescent micrographs of plasma from healthy individuals. We're looking at a PPP smear, a smear with added spike protein, and plasma exposed to spike protein. The goal is to see if adding spike protein creates larger microclots than in healthy blood. We'll be conducting an experiment to investigate this. A question was raised about whether blood type matters, specifically if O positive individuals have fewer reactions to COVID. While I'm not certain, it's something to consider.

Our study investigates the impact of the SARS-CoV-2 spike protein on blood hypercoagulation. We used microscopy and mass spectrometry to examine the spike protein's interaction with platelets and fibrinogen. Results using platelet-poor plasma showed the spike protein may disrupt blood flow. Mass spectrometry revealed structural changes in beta and gamma fibrinogen, complement, and prothrombin after adding spike protein S1. These changes, similar to those observed in COVID-19 patient blood clots, showed resistance to trypsinization. We propose that the spike protein's presence in the bloodstream contributes to hypercoagulation in COVID-19 patients, potentially causing impaired fibrinolysis and persistent microclots. This finding has significant clinical implications. Our goal was to determine the spike protein's effects, as its interaction with these blood components warrants further investigation.

We tested different vaccines on blood slides to observe their effects. Pfizer caused immediate cell clearing, J&J led to cell clumping, and the cells became nonfunctional. The changes were rapid and significant, raising questions about the vaccines' impact on blood cells. More research is needed to understand these findings.

Speaker 0 introduces 'science for injection' and describes a change visible in cells: 'you begin to see a change here. Okay? And then you have this drastic chain of these white nanoparticles coming in.' This is followed by 'the obvious change in the cells, and they are no longer round and normal.' The final image is said to show a person's blood cells 'no longer being smooth and symmetrical. They are now covered with lumps and protrude protrusions.' The speaker labels this as an 'intentional world war on human blood' attributed to 'doctor Sherri Timpenny and Luke Montagner.' The segment ends with, 'Now this is the Nobel'

Clumping of red blood cells in the vascular system is considered a major problem because it blocks blood flow in capillaries, impeding oxygen and nutrient transfer to tissues and waste removal. A microscope slide shows red blood cells clumping into long chains in human blood after five minutes of exposure to a video display terminal, model Ikigami EM125A. After five minutes of exposing the blood to a shielded, full spectrum OttLite fluorescent fixture, model 2,020, the long chains of red cells break up.

I washed my slides and examined blood samples from everyone, including myself. I noticed some anomalies that I hadn't seen in unvaccinated individuals before. These anomalies appear when the blood starts breaking down, particularly during decomposition. Interestingly, while the blood changes, the anomalies do not decompose; instead, they morph into different forms. The red blood cells are present, but when they cluster together, they begin to evolve into something else. This transformation could happen at any moment.

The speaker has been sequencing clots and found real human DNA, showing a signature of neutrophil extracellular traps, a reaction occurring in sepsis when the immune system clots around foreign entities. This process takes free-circulating DNA into clotting structures, leaving a specific signature. Sequencing reveals patient genome sequences that might predispose them to clotting. While billions received shots, not everyone clotted, suggesting a subset has a bad reaction. Genetic predispositions in the clotting cascade may increase risk. Initial analysis of two clots shows high-impact variants in genes involved in fibrin formation and clotting. Kevin McCarran's work demonstrates some clots bind thiophlavin, a marker for amyloid, suggesting a potential amyloidosis issue. Pathology needs careful examination as it may underlie clotting problems. This information is being suppressed, but citations are provided for reference.

I am a live blood analyst and I have noticed some abnormal things in people's blood recently. I don't have all the answers, so I'm reaching out for help. The blood samples I've examined have unusual illuminated objects that glow green and self-assemble into different organisms like crabs or squids. There are also metallic-looking pieces that eventually turn into unknown living things. These objects become dormant when the microscope is off, but come alive when it's turned back on. This is not normal because blood is supposed to die when it dies.

The speaker discusses the formation of clots induced by the spike protein in the blood. These clots can be white and fibrous, and they can vary in size. The speaker mentions that these clots can lead to heart attacks or strokes if they block the flow of oxygen in the body. They also mention an enzyme called Nattokinase, derived from fermented soy, which can break down fibrin and dissolve clots. The speaker suggests that using enzymatic mechanisms to break down clots early can prevent the accumulation of amyloid proteins and the worsening of clots.

I washed my slides and found anomalies in everyone's blood, including my own. These anomalies, resembling liver congestion, can potentially form chains as blood breaks down. Despite the body decomposing, the anomalies do not break down but instead morph into something else, possibly evolving further. While observing red blood cells, I noticed these anomalies forming masses that could transform into something new. The transformation process may occur soon or not at all.

We take a blood sample from the patient and prepare a native blood spread. The sample is heated and dried, rendering the blood cells dead. After cooling, we mix a solution and apply it to the sample. Looking through a microscope, we observe the destroyed blood cells without movement. However, after a short time, the parasites inside the dead blood cells come back to life, swimming actively. These parasites are self-sufficient life forms, not apoptotic bodies. All human cell material in this sample is completely dead.

The speaker claims to present scientific evidence of changes in blood cells. The speaker points to a change, followed by white nanoparticles entering the cells. The speaker states the cells are no longer round and normal. The speaker notes an image showing blood cells no longer smooth and symmetrical, but covered in lumps and protrusions. The speaker claims this is an intentional world war on human blood, citing Doctors Sherri Timpenny and Luke Montagner.

For those who appreciate science, here’s some insight. This image shows a normal cell, but after an injection, noticeable changes occur. White nanoparticles invade, altering the cells' appearance; they lose their round, normal shape. The final image illustrates blood cells that are no longer smooth and symmetrical, now covered with lumps and protrusions. This suggests a deliberate attack on human blood, as noted by experts like Dr. Sherry, Tim Penny, and Nobel laureate Luc Montagnier.