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@SBakerMD - Dr Shawn Baker 🥩
How can a physician be so darn clueless? https://t.co/ZYZwLDIkoW
Video Transcript AI Summary
A lipidologist comments on the dynamic variability of cholesterol levels, criticizing extreme fasting and exercise as abnormal and dangerous. The speaker argues that such practices are consistent with human history and physiology, challenging conventional medical beliefs. The lipid energy model is discussed, highlighting the body's ability to adapt to energy demands. The speaker criticizes modern medicine's reliance on pharmaceuticals and lack of understanding of human biology. The absurdity of the lipidologist's comments is emphasized, with the speaker questioning the necessity of constant eating and sedentary behavior. The speaker concludes by inviting feedback on the topic.
Full Transcript
Speaker 0: Check this out. This is a, physician, a lipidologist, by the way, who commented on the fact that, you know, I'd mentioned the other day that I had checked my cholesterol over 2 subsequent, about 15 hour periods. And and the first time, it was a 156 milligrams per deciliter, which would be considered completely fine by most, you know, conventional physicians. And the second time was 346. It went up a 190 points in about 14 hours, which is shows the extreme dynamic variability of cholesterol, particularly given, you know, certain situations, low carbohydrate diets, exercise, fasting, things like that all have these significant impacts.
And we know that a lot of people just assume that cholesterol is relatively static and making decisions, sometimes lifelong decisions based on a single laboratory. That is how a lot of, you know, medicine has practiced. Right? And so faced with that, this lipidologist says something which I think is completely it shows a complete lack of insight into human biology, human history, human evolution. He says, who would ever resort to doing that extreme of fasting and exercise.
It's abnormal, and it's dangerous. Right? And so I I did eat for 37 hours, and I did an hour of cardio. Now can you imagine a scenario ever in the history history of humanity where something like that could have existed? Could you imagine, humans as hunters, you know, tracking animals for a day or 2 before they killed them, before they ate them, having to do some exercise to acquire their food?
I mean, this would have been the day to day to reality for significant portions of humanity for significant periods of time. Even even in recent thousands of years, that would have not been unusual. And then certainly if you go back into an evolutionary model where hunter gatherers were known to exist. I mean, this is, this is completely consistent with normal human physiology. And to think that we have a dynamic energy system that can respond to that in different ways.
Remember the whole thought behind the lipid energy model is that, things like fat is transported to meet demands. The liver liver transports fat to meet demands. And if you're fasted, you don't have a, you know, a glycogen filled liver, then those demands will be met with likely fat. And does he think that humans always had access to, you know, a grocery store? I mean, was he thinking that Uber Eats existed, you know, 20000, 50000 years ago perhaps?
And this is completely one of the problems, you know, with with medicine today, they have no insight into how the human body actually works. Believe it or not, as physicians, they don't understand. They all they understand is pharmacodynamics. Farming this, pharma that. This is where their education comes from.
This is where their thought comes from. They've lost the ability to think critically about these things. They have no curiosity. The only curiosity they have is what's the next drug gonna do. You know, it's very sad.
It's a very sad commentary. And so Pablo, perhaps if you, fasted and maybe exercised, you get rid of that double chin you're sporting there, buddy. So anyway, think about this. Think about the absurdity of that comment to say that it's dangerous. It's dangerous not to eat every 3 hours or something like that.
I mean, this is where we this is where we've gone as a physician saying that. You know, this is just insanity. Anyway, guys, let me know what you think. Do you think it's dangerous to go a whole 37 hours without eating? Do you think it's dangerous to do any kind of exercise without eating?
Do you think you'd have to just shove Twinkies in your mouth every 15 minutes and and just move around like a sloth? Perhaps that's a perhaps that's a way we're we're designed to live. Do you think anybody think that's realistic? Anyway, it's comical. Makes me laugh.
You know? Anyway, I'm off to LA for a couple days. We'll talk to you guys soon. Bye bye.
@SBakerMD - Dr Shawn Baker 🥩
BIG NEWS about Cholesterol!! @joerogan @realDaveFeldman @nicknorwitz https://t.co/vTPNZr7hbf
Video Transcript AI Summary
The speaker discusses a study conducted by Dr. Matt Budoff from the Alonquista Institute at UCLA. The study looked at individuals on low-carb ketogenic diets with extremely high LDL cholesterol levels. They compared these individuals to a control group with normal LDL cholesterol levels. The study found no statistically significant difference in plaque buildup in the arteries between the two groups. While the speaker acknowledges that LDL cholesterol is still considered a risk factor for cardiovascular disease, the findings suggest that in this specific population of healthy individuals on a low-carb diet, high LDL cholesterol may not be a significant factor in the development of cardiovascular disease. Further research is needed to explore these findings.
Full Transcript
Speaker 0: Alright, folks. So the data that I've been talking about is finally in. This is super exciting stuff. Stick around. I'm gonna try to go through this, the best that I can to present, something I think is incredibly, incredibly powerful, incredibly, incredibly earth shaking in a world of of health, quite honestly, and and cardiovascular disease.
And so, last night, doctor, Professor Matt Budoff, at from the Alonquista Institute at UCLA presented a collection of data, which will soon be published in Metabolism. The journal, the abstract will be published there very shortly. And what they what he looked at were A collection of people on very low carb ketogenic diets that happen to have extremely, extremely high LDL cholesterol. We're talking as high as 600 milligrams per deciliter, just off the chart, super high. And what they did was they did something called a coronary CT angiogram.
So they looked at very high level, very precise, very sensitive study looking at the coronary vessels in the heart. And they looked to find out how much plaque was in the heart of these people with this super, super high cholesterol. And they compared it to age match controls that were identical essentially in every other way. They had the same level of Body mass index was almost identical. The ages were almost identical.
Average age was about 55. They all were healthy. None of them had diabetes. None of them had hypertension. So they are identical effectively in every single way, except the one difference being is 1 group had super high, ridiculously high LDL cholesterol versus one that had normal LDL cholesterol.
Okay? And the average person that had the high cholesterol had it at least that high for at least 5 years, right? And Matt Matthew Budoff, the principal investigator, is Arguably, the world's leading authority on how rapidly you can detect plaque accumulation in the coronary vessels over time. And 5 years is plenty of time to see this, right? So the prediction should be based on the current understanding or beliefs around Cholesterol and heart disease is the group that has the highest cholesterol over time should definitely have more plaques in their arteries.
That is what we would expect, right? What did they find? So what did they find? So this is A, slide from his presentation, it shows that the 2 groups here, the 1 on the left is a group that had the super high cholesterol. And the one on the right is the group that had the normal cholesterol.
And guess what? There was no difference statistically between the 2 groups. It did not matter if you had super high cholesterol as high as 600 mg per deciliter versus somebody that has a normal LDL cholesterol, right? In fact, in fact, even though there is no statistical difference, The trend was that the people with the high cholesterol had less plaque in their arteries. Right?
So what does this mean? Does it mean that you can ignore LDL cholesterol? It doesn't matter. No. It doesn't mean that.
Doctor. Butoff, in his 17 minute presentation, Says that, no, you cannot ignore that. He's still a believer in LDL cholesterol being causal to cardiovascular disease. But what he is saying is that It appears to be, in some instances, you know, this particular set of population, low carb, lean, otherwise metabolically healthy, But it appears that cardiovascular disease is not developing or not developing at any significant rate at all. So that may give some of you guys that are in this situation some level of relief.
His suggestion was if you have high cholesterol but all the other markers look good, get sort of an imaging study, maybe get a coronary calcium score. And if it is low or 0, then you're probably pretty good. I think that's that's probably where it is. So to me, as I've been saying all along, it means that LDL cholesterol, while potentially causal For cardiovascular disease, it becomes a dependent variable. It depends on what else is going on.
And so, This is big. This is huge. The study is going to continue on probably for several years. They're going to get 1 year data to see if any progression has occurred. And if they Still do not see progression, even though they've already had 5 years of a collection period, more or less, then it is likely that We can start to say that, hey, LDL cholesterol and by extension, ApoB, because ApoB is part of LDL.
LDL is part of OB subset basically that we have to rethink the entire nature of LDL is always, always, always, or APO B is always, always, always cause of cardiovascular disease because clearly in this population, which has never been studied before, particularly in this level of detail. It appears to not be so. All right, guys? Tomorrow At 10 a. M.
Pacific Time, I'm going to be hosting, both, Doctor. Nick Norwitz and David Feldman to discuss this in detail in a much longer format. But this is the overview. I hope you guys appreciate it. I I think there's more to come on this.
More research needs to be done. There's more questions that need to be asked. Can you expand this to everybody and say LDL cholesterol doesn't matter to anybody? No, you cannot say that. But we're starting to see some significant questions that need to be asked.
All right, guys. Thanks a bunch. Let me know what you think. Me know if this is if this is been your experience too. I see a lot of people saying, Hey, I'm carnivore.
I'm keto. My cholesterol is through the roof, but my CHC score is 0. I've got no evidence of heart disease. Let me know if that's been your case in the in the comments below. Alright.
We'll talk to you soon.
Speaker 1: Yeah. These are the baseline characteristics, and there were differences most notably, of course, in LDL cholesterol. Juice 72 Average LDL in the keto group, Miami Heart group had an LDL of a 123. All the other variables were well matched, as best we could match these these 2 different populations. So when we looked at this population, they were both 55 years old.
Again, the, lean mass hyperresponders had an LDL of 272 milligrams per deciliter on an average of four 0.7 years duration. So it wasn't like this was 1 week of being having LDL that this high. This was a almost a 5 year duration of having a ketogenic diet and an LDL above 190 milligrams per deciliter. When we compare them to patients with more normal, LDLs, LDL mean LDL of 123. There was no difference in coronary plaque burden.
So we did not see any, evidence that this LDL of 2 72 induced more atherosclerosis over this 5 year period.
@SBakerMD - Dr Shawn Baker 🥩
BRAND NEW STUDY!! Higher total Cholesterol (243 mg/dl) associated with greater chance of reaching 100!! Many patients had data from 35 years ago for the “iT MUst bE ReVERsE CaUsAtion” crowd!! https://link.springer.com/article/10.1007/s11357-023-00936-w
Blood biomarker profiles and exceptional longevity: comparison of centenarians and non-centenarians in a 35-year follow-up of the Swedish AMORIS cohort - GeroScience
Comparing biomarker profiles measured at similar ages, but earlier in life, among exceptionally long-lived individuals and their shorter-lived peers can im
link.springer.com
@SBakerMD - Dr Shawn Baker 🥩
How do you know you’ve been lied to about red meat? Spend a few months eating primarily red meat and see what happens to your health! Good to see so many people waking up!!! https://carnivore.diet/carnivore-diet-success-stories/
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