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@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

Learn more about cancer (CA) in this thread I'm going to go over how the liver & toxic bile are completely connected to CA See tweet below This is sort of correct, read on to FULLY understand it You're going to want to RT this first tweet to spread it... 🧵

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

WHAT IS REALLY GOING ON IN THE BODY WITH THE C-VIRUS Look at the pics first please Nursing mother, either diagnosed C+ OR exposed to C+ people You will not hear this explanation anywhere else I'm not going to argue germ theory here, it's irrelevant /// THREAD ///

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

THE SIMPLE REALITY OF COFFEE / CAFFEINE Before starting a coffee/caffeine habit, you weren't more tired if you *didn't* have it You also didn't have withdrawal sides like headaches if you missed your dose Accept coffee/caffeine is a drug first /// THREAD ///

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

"The U.S. Preventive Services Task Force (USPSTF) has issued recommendations against the use of 2 nutritional supplements to prevent cardiovascular disease & cancer. They are beta-carotene & vitamin E. Specifically, beta-carotene even has the risk of causing cancer." ///Thread

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

Having trouble raising your Vit D levels? Find you feel WORSE taking Vit D pills? Check this thread FB comment: "Yes, when I was at my sickest my vit d level was really low. My dr couldn’t believe it because I was taking cod liver oil and lots of high vit d foods. 🤢" [cont]

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

Want to optimize your hormones? PAY ATTENTION TO THIS THREAD "Animal Vit A" damages testosterone-producing Sertoli cells: Retinol supplementation induces oxidative stress & modulates antioxidant enzyme activities in rat sertoli cells https://pubmed.ncbi.nlm.nih.gov/11328675/ There's more! 🧵

Retinol supplementation induces oxidative stress and modulates antioxidant enzyme activities in rat sertoli cells - PubMed Recent intervention studies revealed that supplementation with retinoids resulted in a higher incidence of lung cancer. Recently the causal mechanism has begun to be clarified. We report here that retinol caused cellular oxidative stress and modulated superoxide dismutase, catalase and glutathione p … pubmed.ncbi.nlm.nih.gov

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

HOW BLUE LIGHT DAMAGES THE EYE Blue light is a hot topic these days Vitamin A toxicity is becoming a hot topic too, with at least 1/3 of Americans legit victims of it Did you know that the two are intricately connected? 🧵THREAD🧵

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

Many claim that natural & "synthetic" forms of vitamin A (vA) are different...they say: - Natural forms are not toxic & synthetic is - Somehow, even though they are CHEMICALLY IDENTICAL, they function vastly differently They are quite wrong, I'll show you in research here 🧵

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

If you want to help others, please RT the first tweet of this thread! Video of what is covered here: https://www.youtube.com/watch?v=sa9Gzp-alYU This thread is NOT debating the fact that HUMANS need SUNLIGHT (and/or UV-B light at minimum) to be healthy

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

LOWERING TOXIC COPPER IN DIABETIC MICE IMPROVED THEIR INSULIN RESISTANCE Straightforward...using a molybdenum compound to lower toxic copper & CORRECT DIABETES. Whodathunkit??? Role of copper ion in the pathogenesis of type 2 diabetes https://pubmed.ncbi.nlm.nih.gov/19461160/ / / / THREAD / / /

Role of copper ion in the pathogenesis of type 2 diabetes - PubMed Reactive oxygen species (ROS) are induced under diabetic conditions and are likely associated with the development of type 2 diabetes. It is also known that ROS production is facilitated in the presence of copper ion through the Fenton reaction. The aim of this study was to examine the involvement o … pubmed.ncbi.nlm.nih.gov

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

Why you should stop poisoning yourself & your kids with toxic cod liver oil aka CLO (& fish oils, for that matter) Chances are, you've never seen the research I'm going to drop on you Once you realize the mistake, don't be too hard on yourself, move forward & throw it AWAY 🧵

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

Ladies (men too!) who have issues w FERTILITY, listen up I get people SO HEALTHY that I warn them to be diligent if they DON'T want more kids Friday on my livestream I'll cover a testimonial from 40-y/o woman who slipped 1X in her measures & is now pregnant I fix fertility 🧵

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

Men, pay attention. 46 years old, not on ANY hormones, 2 kids, full time business owner My total testosterone just clocked in at 893.5 ng/dL (normal range given: 264 - 916) I think it is safe to infer that I know what the heck I'm talking about in regards to health 🧵

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

Humans do not need retinol, carotenoids, or "Vit A" for anything I regularly fix chronic disease by getting people OFF as much food-based Vit A in their diets as possible (supplements only add more to the problem) Want to see evidence Vit A is absolutely toxic to your skin? 🧵

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

A LOW "BLOOD VITAMIN A" TEST MEANS NEXT TO NOTHING, & FATTY LIVER IS A VITAMIN A TOXICITY CONDITION If you think getting a serum retinol ("blood Vitamin A") test is going to show if you are vitamin A toxic or not, you are sadly mistaken...and I've said this over and over. 🧵

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

Infertililty is absolutely the goal of these people, see image below I fix infertility ALL THE TIME Let me help everyone see how they have induced infertility in plain sight They tell you in the research and in their chosen "drugs" how they do it Follow me here... 🧵

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

Want to know what causes "age spots" aka lipofuscin, and how to get rid of them? The cause is Vit A (retinal, aka retinaldehyde) The solution is to minimize Vit A (all of it) Need to see the proof? 🧵

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

The Basics Of A Low Poison Diet Once one accepts Vit A is a poison, here are the foods to reduce / minimize / avoid: Animal foods: - Liver & organ meats - Egg yolks - Dairy - Pork (contains retinoic acid, doesn't show up on Vit A testing) Plant foods next 🧵

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

New study on the poison called "Vitamin A"! Higher levels of serum retinol (blood Vit A) in Chinese children linked to: - Metabolic syndrome - General obesity - High LDL - High total cholesterol - High uric acid (related to gout) I'm here to help you stop poisoning yourself 🧵

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

PART 2: Are you STILL eating the filter + sewage treatment plant of the body, aka LIVER? PART 1: Continuing where we left off, covering case studies of human poisonings from consuming animal livers of all types (fish, chicken, beef) 🧵

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

Warning! Are you still eating TOXIC organ meats? In this thread, I'll cover the evidence that ALL organ meats, including wild, grassfed, and conventional animals are filled w toxins This thread may save your life RT to help people stop poisoning themselves!

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

MEN...you DON'T have to take Testosterone Replacement Therapy (TRT) to fix your depression & low T levels! What you have is a combo of TOXICITIES & DEFICIENCIES TRT is a band-aid cover-up It does *nothing* to fix the problems I'll cover in this /thread/ RT this & HELP MEN

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

How To Correct PCOS (Polycystic Ovarian Syndrome) Do you have this issue, or know someone who does? It can be fixed It is a combo of COMMON toxicities and mineral deficiencies I test for & address daily Let me walk you through it RT this to help the women in your life

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

There is a myth that taking K2 will protect you from vit D3 calcifying you into a statue...this is NOT true: "progression in aortic & coronary artery calcification score was NOT significantly different bw patients treated w MK-7 + vit D & patients receiving placebo. 🧵

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

Are you *still* eating carrots? We were all LIED TO about the supposed "benefits" of carrots, and have been for a very long time I'm going to take you on a ride through the deceptive health history of the carrot / thread /

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

The Worldwide "vitamin" A Toxicity Epidemic: What I Learned By Looking At Gold-Standard *Liver Biopsy* Studies Bold statement, right? Yes, it's happening. Yes, I'll show research No, it does NOT show up in blood tests It shows up in the liver itself SHARE THIS / thread / 👇

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

Why Tattoos Make People Sick & Send Them To Early Graves Most people haven't looked into the research on tattoos I have. It's ugly. Real ugly Do what you choose to do, I just provide information Hopefully you'll reconsider getting any (more) done after this / thread / 👇

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

Bill Gates, Population Control, & "vitamin" A: What We Can Learn By Following His MONEY$$$ Many are familiar w this "man" 😉 & his goals for humanity Did you know "vit" A is a CRUCIAL part of his approaches? What does HE know about it that YOU don't? SHARE THIS / thread /

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

Why Too Many EGGS May Be Shortening Your Life (HINT: It's NOT The Cholesterol!) Eggs are a fave food in Twitter-land, w ppl claiming they're 1 of the healthiest foods If you're open-minded, you need to see this research Then ask why you've never seen it before / thread / 👇

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

"vitamin" A Deficiency Doesn't Exist: What I Learned By Examining Human Studies It is claimed that "vit" A is *essential* & required thru diet That said, if even *1* person can be healthy w/o any of it, then it's NOT essential Let us explore this topic IN DETAIL / thread /

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

LADIES (especially), are you interested in NOT ruining your health with toxic cosmetics? Then you want to read this / thread / & SHARE IT Questions: Is topical "vitamin" A a contributor to "vit" A toxicity? Is topical "vit" A connected to CANCER & DEATH? 😬🤭😱 👇

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

How "vitamin" A is a MAJOR cause of the OBESITY epidemic I guarantee you've NEVER heard of this connection before We'll go into the science, as we always do Buckle up, buttercup SHARE THIS to help others / thread / 👇

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

WARNING! Is eating "healthy" putting you on the fast track to diabetes? Did you know there is TONS of research linking "vitamin" A to diabetes? Why weren't you ever told this by the health goo-roos? I'm here to correct this oversight now / thread / 🧵👇

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

How vitamin A TOXICITY is connected to Alzheimers & dementia These are terrible diseases of our modern era "mOdErN mEdIcAl ScIeNcE" ➡️ ZERO progress on figuring it out What if it's connected to toxicity of a so-called "vitamin"? Don't forget 😉 to RT this / thread / 👇

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

Live Somewhere With Little Sun? Pay Attention. Supplemental Vitamin D3 Pills are Literal Rat Poison (Seriously!) You want to raise your blood vitamin D levels, but… taking D3 pills is a BAD idea. Here’s what you can do instead and get the LIGHT you need…

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

There are Multiple Things You Should Do to Boost Your Vit D Levels Without Damaging Your Health But Did You Know that These Things Can Improve Your Health in Other Areas? Here’s How You Can Boost Your Vitamin D Without Taking RAT POISON (Vit D3 Supplements)

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

The Nutrition Detective "How To Fix Vit D WITHOUT Taking Rat Poison Pills" MEGA Thread I cover: - Why you don't want to take D3 or D2 supps - How to use LIGHT to ⬆️ D levels - Taint-sunning science - How TOXICITIES & DEFICIENCIES affect D levels Let us begin 👇

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

"vitamin" A Causes Cataracts: Connecting the Real-World Evidence ~6 yrs ago the CBC (Canadian Broadcasting Company) came out with this ~5 min video segment on people in China w cataracts getting free surgeries, ORIGINAL version here (this becomes important later): https://www.youtube.com/watch?v=dRFbUHcCRCo Quoted from 0:26 in: "For an aging rural population w the highest number of cataract sufferers in the world. No one knows exactly how many in China, though it's well in the tens of millions." You'll note the patients have many liver/age spots, bad skin in general, & most seem to have an orangish tint to their skin. Liver/age spots are lipofuscin accumulation, which is caused by toxic "vit" A accumulation, see my threads: The orange-ish skin is from excessive carotenoids aka "plant vitamin A" accumulating in their skin and subcutaneous fat Lipofuscin accumulates in the eye, and remember that retinyl is "vit" A: "Lipofuscin is a fluorescent material with significant phototoxic potential that accumulates with age in the retinal pigment epithelium (RPE) of the eye. It is thought to be a factor in retinal degeneration diseases. The most extensively characterized lipofuscin component, N-retinylidene-N-retinylethanolamine (A2E)" https://pubmed.ncbi.nlm.nih.gov/22417141/ Where might all this "vit" A come about in their diets? 🤔 Key quote from 4:07 in: "His mother dries sweet potatoes by the fire for dinner, it's the only vegetable they can grow here." Wouldn't it be strange then if someone in the Canadian media edited out the connection between very high-"vit" A food consumption a la sweet potatoes and the population with the world's highest rates of cataracts? Watch the same video on the CBC website, jump to 4:00 mark and watch the same part, watch for the change: https://www.cbc.ca/player/play/1131719747560 It has been edited to say: "...potatoes by the fire for dinner, it's the only vegetable they can grow here." Someone found it important enough to go back and edit out "His mother dries SWEET..." Those who know would call this lying by omission For those of you who are familiar w excessive blue light damaging the eyes, make sure to see my thread here about how blue light ONLY causes eye DAMAGE when "vit" A is PRESENT: Such a strange bunch of coincidences, yes? Share this important information with those you know who have eye issues!

Lipofuscin and A2E accumulate with age in the retinal pigment epithelium of Nrl-/- mice - PubMed Lipofuscin is a fluorescent material with significant phototoxic potential that accumulates with age in the retinal pigment epithelium (RPE) of the eye. It is thought to be a factor in retinal degeneration diseases. The most extensively characterized lipofuscin component, N-retinylidene-N-retinyleth … pubmed.ncbi.nlm.nih.gov

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

My opinion on the pneumonia that is the new scary white lung syndrome It is simply another variation of toxic bile and its common toxic constituents leaking into the bloodstream and causing problems systemically (lungs definitely included). I'll hit on the major points Let us start with #toxicbiletheory Are bile markers higher in the blood in pneumonia? Yes "The present study indicates that elevation of serum bilirubin and of blood urea are both common accompaniments of pneumonia [...] In Port Moresby, pneumonia remains the commonest cause of jaundice among patients admitted to the medical wards" https://sci-hub.st/10.1111/j.1445-5994.1974.tb03202.x Remember, bilirubin is BILE-irubin, and high bilirubin is one of many potential markers of excess bile in the blood. Bile in the blood is BAD Babies born to mothers who have excess bile in their blood (intrahepatic cholestasis of pregnancy) can get pneumonia from the bile in their own blood: "Bile acids are detectable in the bronchoalveolar lavage fluid of newborns from intrahepatic cholestasis of pregnancy affected by respiratory distress syndrome. Elevated serum bile acid levels in these infants allow us to hypothesize that bile acid reaches the lung after an uptake from the circulation. These findings strongly support a role for bile acid in causing bile acid pneumonia." https://publications.aap.org/pediatrics/article-abstract/121/1/e146/71033/Bile-Acid-Induced-Lung-Injury-in-Newborn-Infants-A?redirectedFrom=fulltext More connecting bile and pneumonia: "TBAs [total bile acids] on admission were significantly higher in ICU patients compared to controls. TBAs were 3x higher in septic than cardiogenic shock patients and 6x higher than in surgical patients and controls as shown in Table 2. [...] Hyperbilirubinemia (serum bilirubin ≥ 2 mg within 48 h of admission) was observed more often in patients with septic shock (33%) compared to cardiogenic shock (21%) and patients after surgery (5%)." We connected bile acids in the blood to sepsis, now we connect sepsis to pneumonia: "Most common causes of sepsis were pneumonia (46%)..." https://annalsofintensivecare.springeropen.com/articles/10.1186/s13613-017-0272-7 They're so close, they just have their cause-and-effects in the wrong order. It's the BILE causing the sepsis AND pneumonia, because you AREN'T SUPPOSED TO HAVE TOXIC BILE IN YOUR BLOOD. Sepsis and pneumonia are SYMPTOMS caused by the toxic bile, see? It so happens that a side effect of the oral "medication" UDCA, a secondary bile acid (the most toxic kinds) that is directly related to the supplement TUDCA, is PNEUMONIA: ""Unanticipated" UDCA toxicity includes hepatitis, pruritus, cholangitis, ascites, vanishing bile duct syndrome, liver cell failure, death, severe watery diarrhea, pneumonia, dysuria, immune-suppression, mutagenic effects and withdrawal syndrome upon sudden halt." Toxic bile will contain toxic copper and toxic "vitamin" A, and both these toxins will deplete zinc. Can we find this copper & zinc pattern in the blood in pneumonia? "In middle-aged and older Finnish men, increased serum Cu/Zn-ratio and Cu concentrations are each linearly associated with an increased risk of incident pneumonia." https://pubmed.ncbi.nlm.nih.gov/35781862/ Did you know that one of the COMMON side effects of giving people "active vitamin A" aka retinoic acid, as CHEMOTHERAPY, is pneumonia? Remember, the body dumps toxic "vitamin" A into the bile: "Formerly referred to as retinoic acid syndrome, differentiation syndrome is a common and potentially life-threatening complication seen in patients with APML treated with ATRA [all-trans retinoic acid aka "active vitamin A"] and/or ATO [arsenic trioxide, another poison] therapy. The clinical features of differentiation syndrome are also observed with other complications (i.e., sepsis, fluid overload, pulmonary hemorrhage, pneumonia, renal failure, and congestive heart failure)." https://www.sciencedirect.com/topics/nursing-and-health-professions/retinoic-acid-syndrome Also make sure to read this amazing paper by Anthony Mawson, titled: Liver Damage and Exposure to Toxic Concentrations of Endogenous Retinoids in the Pathogenesis of COVID-19 Disease: Hypothesis "We propose that SARS-CoV-2 virus-induced liver damage causes retinoic acid and stored retinyl esters to be released into the circulation in toxic concentrations, unbound to protein, with resulting damage to organs including the lungs, heart, blood vessels, and skin. Several lines of evidence support this model of disease causation." https://pubmed.ncbi.nlm.nih.gov/33983857/ The only question left in my mind is, what is triggering the massive toxic bile dump? I still absolutely think that EMF is a huge suspect in this mess, and you can see my video on it here with the references: https://youtube.com/live/Y2fNwCaW-Bs?feature=share So there you go

Circulating bile acids predict outcome in critically ill patients - Annals of Intensive Care Jaundice and cholestatic hepatic dysfunction are frequent findings in critically ill patients associated with increased mortality. Cholestasis in critically ill patients is closely associated with stimulation of pro-inflammatory cytokines resulting in impaired bile secretion and subsequent accumulation of bile acids. Aim of this study was to evaluate the clinical role of circulating bile acids in critically ill patients. Total and individual serum bile acids were assessed via high-performance liquid chromatography in 320 critically ill patients and 19 controls. Total serum bile acids were threefold higher in septic than cardiogenic shock patients and sixfold higher than in post-surgical patients or controls (p < 0.001). Elevated bile acid levels correlated with severity of illness, renal dysfunction and inflammation (p < 0.05). Total bile acids predicted 28-day mortality independently of sex, age, serum bilirubin and severity of illness (HR 1.041, 95% CI 1.013–1.071, p < 0.005). Best prediction of mortality of total bile acids was seen in patients suffering from septic shock. Individual and total BAs are elevated by various degrees in different shock conditions. BAs represent an early predictor of short-term survival in a mixed cohort of ICU patients and may serve as marker for early risk stratification in critically ill patients. Future studies should elucidate whether modulation of BA metabolism and signalling influences the clinical course and outcome in critically ill patients. annalsofintensivecare.springeropen.com

Serum copper-to-zinc ratio and risk of incident pneumonia in caucasian men: a prospective cohort study - PubMed Serum copper (Cu) and zinc (Zn), essential micronutrients that have important immunomodulatory and antimicrobial properties, are biomarkers of ageing. Serum Cu/Zn-ratio may be a more reliable marker for age-related degenerative conditions compared with serum Cu or Zn alone. We aimed to assess the as … pubmed.ncbi.nlm.nih.gov

Retinoic Acid Syndrome - an overview | ScienceDirect TopicsScienceDirect sciencedirect.com

Liver Damage and Exposure to Toxic Concentrations of Endogenous Retinoids in the Pathogenesis of COVID-19 Disease: Hypothesis - PubMed Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) has a marked tropism for the biliary tract; it damages the bile ducts and hepatocytes and can lead to liver decompensation, cirrhosis, and sepsis. The pathogenesis of liver damage and its association with damage to the lung, heart, and bra … pubmed.ncbi.nlm.nih.gov

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

Fevers are not what you've been told Fevers are massive dumps of toxic bile leaking into the bloodstream and causing havoc in the system, see below See "bilious fever", a concept that has been around since the 18th century: "The term...was used by medical practitioners in the 18th and 19th centuries for any fever that exhibited the symptom of nausea or vomiting in addition to an increase in internal body temperature and strong diarrhea, which were thought to arise from disorders of bile, the two types of which were two of the four humours of traditional Galenic medicine." [nausea and vomiting are caused by bile getting into the stomach] https://en.wikipedia.org/wiki/Bilious_fever What is known to happen when bile leaks into the bloodstream? "The clinical presentation of a patient with a postoperative biliary leak may include right upper quadrant pain, nausea, vomiting, anorexia, and fever." [there's the nausea & vomiting again!] https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5088090/ Boy, the above sure sounds like "the flu aka Coofid" to me! NO VIRUSES NECESSARY, only toxic bile going in large amounts into places it ISN'T supposed to be! Galen put forth that disease was related to bile disorders around 200 AD Studies on the toxicity of bile in the blood were done BY THE ROCKEFELLER INSTITUTE way back in 1905 & 1906, why have you never heard of this? (you should know why 😉): https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2124573/pdf/280.pdf https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2124605/pdf/127.pdf Your health issues have always been related to your liver and #toxicbile, and we are the only ones focusing on fixing it See my Love Your Liver livestreams #53 & #71 to get caught up on this new paradigm of chronic disease: https://www.youtube.com/@NutritionDetective/streams

Bilious fever - Wikipedia en.wikipedia.org

Diagnosis and Management of Postoperative Biliary Leaks Postoperative biliary leaks have become more common in the past three decades since the development of laparoscopic biliary surgery. The role of the radiologist and interventional radiologist is important in the diagnosis and treatment of such ... pmc.ncbi.nlm.nih.gov

Nutrition Detective This is the YouTube channel of Dr. Garrett Smith, the Nutrition Detective. Dr. Smith is the foremost expert on the worldwide epidemics of Vitamin A toxicity... youtube.com

@NutriDetect - Nutrition Detective 🔍 - Dr. Garrett Smith

Let me explain how Morley Robbins and his "protocol" are wrong on ceruloplasmin production supposedly requiring the toxin called "vitamin" A Morley and his minions constantly say that natural and “synthetic” forms of “vitamin” A are completely different in function. This is entirely unsupported in science and is a classic Appeal to Nature fallacy If science uses the same chemical processes to identify natural and synthetic forms of "vitamin" A, then they have the EXACT SAME STRUCTURE and would therefore express the EXACT SAME FUNCTION. This should seem obvious, or else one has a lot of proving to do to show they are different in function even while they are exactly the same in structure. Let's see them do that I thoroughly demonstrate that there is ZERO DIFFERENCE in the toxic effects of natural and synthetic “vitamin” A in this thread: His group will deny any study as garbage that doesn’t use “natural vitamin A", but when it serves their narrative, they'll quote the study and just ignore the hypocrisy First, you should know that ceruloplasmin, a COPPER-BINDING PROTEIN (it protects us from toxic free copper) is an ACUTE PHASE REACTANT, which means it goes UP in INFLAMMATORY STATES. I say that copper and "vitamin" A are both toxic. This will be a coincidence you don't want to forget What type of "vitamin" A is commonly used in the studies he quotes to show “vitamin” A is necessary for ceruloplasmin production? Hint: not "natural"! Let me go over some of the research in short: Giving rats *synthetic* ACCUTANE aka 13-cis retinoic acid raised ceruloplasmin https://pubmed.ncbi.nlm.nih.gov/3655940/ Second study, giving rats *synthetic* ACCUTANE aka 13-cis-retinoic acid raised ceruloplasmin https://pubmed.ncbi.nlm.nih.gov/9202972/ So giving *synthetic* ACCUTANE aka "active vitamin A" to rats increases the ACUTE PHASE REACTANT called CERULOPLASMIN that would typically go up in INFLAMMATORY STATES. Interesting, no? Before we go into the next part, you may want to read my thread here on how "vitamin" A deficiency (natural or synthetic) DOESN'T EXIST: Now, if their stance on "vitamin" A and ceruloplasmin is true, shouldn't "vitamin A deficiency" cause ceruloplasmin to GO DOWN? Is that what actually happens? Rat liver cells: "The intracellular concentrations of albumin and ceruloplasmin were not significantly altered by vitamin A deficiency." https://pubmed.ncbi.nlm.nih.gov/6337857/ No changes in ceruloplasmin with "vit" A deficiency? "Concentrations of ceruloplasmin...are increased in the serum of vitamin-A-deficient rats compared to normal rats." https://pubmed.ncbi.nlm.nih.gov/6859810/ Wait, "vit" A deficiency caused ceruloplasmin to go UP? (if you see my thread above and my comment below, I don't think the rat diets were actually low in "vit" A at all) "The ceruloplasmin level of control animals and vitamin A-deficient rats was determined. An average increase between 22 and 33% was observed in the animals with vitamin A deficiency, the highest levels being observed in the females. These results are in agreement with Peterson's previous work." https://pubmed.ncbi.nlm.nih.gov/533078/ Another study saying "vit" A deficiency caused ceruloplasmin to go up? (these studies are not hard to find) Finally, a study on chickens fed a "vitamin A deficient" diet for 20 days showed they had the HIGHEST CERULOPLASMIN at the end of the 20-day study! https://pubmed.ncbi.nlm.nih.gov/3034814/ (see picture from full study below) So, based on this small group of studies, it would seem the "science" isn't very straightforward on this, and any studies showing "vit" A raised ceruloplasmin very specifically used SYNTHETIC RETINOIC ACID aka ACCUTANE...so Mr. Robbins by his own standards SHOULD NOT be able to use any of it to justify his position So why such disparate results? First, they gave the rats and liver cells RETINOIC ACID, and the ACUTE PHASE REACTANT called CERULOPLASMIN which goes UP in INFLAMMATION reacted accordingly to the toxic input. This makes sense to me Next, when one understands that animal studies commonly use casein protein that very likely still have "vit" A in the casein as oxidized retinoic acid (which is not found when they analyze for retinol, so they say there is "no vitamin A" but they are quite wrong, the heating and alcohol they used to supposedly "remove" the vA just changed its form so they couldn't see it any longer), then nearly all the studies above make sense in my framework I put people on low "vitamin" A diets for YEARS, and they are not developing so-called "copper deficiency" or ceruloplasmin deficiency...shouldn't this problem have shown up by now if I'm wrong? I would love to see the pseudo-intellectual tap-dancing done to try to make all of these studies align to somehow say that "vitamin" A is necessary to make ceruloplasmin, because if one takes them at face value, they most certainly DON'T The chicken study and my client results also don't agree with their assertions Remember, any study that uses synthetic forms of "vitamin" A must be disregarded by their own statements...and the "vitamin A deficient" diets raised ceruloplasmin in both rats and chickens 😂 Do I think he is wrong? Yeah, that's an understatement

Induction of ceruloplasmin synthesis by retinoic acid in rats: influence of dietary copper and vitamin A status - PubMed Ceruloplasmin, a copper-containing acute phase plasma protein, has been shown to be regulated by 13-cis retinoic acid in rats. Ceruloplasmin activity was significantly increased within 24 h and remained elevated for at least 72 h after a single injection of 13-cis retinoic acid. With daily injection … pubmed.ncbi.nlm.nih.gov

Regulation of ceruloplasmin by retinoic acid in the developing rat - PubMed Ceruloplasmin (Cp), the major copper-binding protein in the plasma, is an acute phase protein with ferrioxidase activity. Both its oxidase activity and hepatic mRNA abundance increase during the developmental period. To test the possible role of retinoic acid, a derivative of vitamin A known to part … pubmed.ncbi.nlm.nih.gov

Subcellular localization in normal and vitamin A-deficient rat liver of vitamin A serum transport proteins, albumin, ceruloplasmin and class I major histocompatibility antigens - PubMed The subcellular localization in rat liver cells of retinol-binding protein (RBP), prealbumin, ceruloplasmin, albumin, and class I transplantation antigen chains was investigated by radioimmunoassay determinations. The concentration of RBP was high in the rough and smooth endoplasmic reticulum (SER). … pubmed.ncbi.nlm.nih.gov

[Changes in blood levels of haptoglobin and ceruloplasmin in vitamin A deficiency in Sprague-Dawley rats] - PubMed The aim of the present study was to find out whether the elevation of the serum ceruloplasmin level, previously described in vitamin-A-deficient rats, is a specific phenomenon. Quantitative variations of serum ceruloplasmin, albumin and haptoglobin (whose concentration increased during inflammation) … pubmed.ncbi.nlm.nih.gov

[Relation between ceruloplasmin and vitamin A in Sprague-Dawley rats] - PubMed The Moore's hypothesis concerning a relationship between the metabolism of copper and that of vitamin A led us to consider a possible relationship between this vitamin and ceruloplasmin, the carrier protein for copper. Experiments were carried out on Sprague-Dawley rats. The ceruloplasmin level of c … pubmed.ncbi.nlm.nih.gov

The effect of different dietary levels of vitamin A on metabolism of copper iron and zinc in the chick - PubMed Chicks were fed on diets containing either no added vitamin A or 3300 micrograms/kg or 330,000 micrograms/kg retinol equivalents for 30 d. Concentrations of copper, iron and zinc were higher in liver and lower in plasma at low and high intakes of vitamin A. Haemoglobin, packed cell volume and erythr … pubmed.ncbi.nlm.nih.gov

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